ECG Flashcards

0
Q

Depressed ST interval with inverted T wave

A
  • indicator of ischemia

- may see reciprocal changes

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1
Q

ECG interpretation process

A
  • rate
  • rhythm
  • P waves: I,II,aVL,aVF, V1-3: less than 2.5 boxes
  • T waves: Limb leads 3; Q wave ~1 box
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2
Q

ST elevation with significant Q wave and again, inverted T wave

A
  • indicator of infarction/injury

- clinically significant Q wave = at least 1 box (most reliable)

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3
Q

Prematurity of beats: 3 options

A
  • atrial (if p waves present atria is at fault)
  • junctional (no p waves normal QRS complex)
  • ventricular (abnormally wide QRS complexes)
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4
Q

Irregularly irregular

A
  • either Multifocal atrial tachycardia

Or - Afib (Afib atrial beats of 350+ BPM: looks like junk)

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5
Q

Epicardial vs endocaridal ischemia

A
  • epicardial ischemia -> ST elevation

- endocaridal -> ST depression (possible becuase it happens inside out so epi ischemia is more like infarct)

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6
Q

Pace and etiology

A
  • 150-250: SVT
  • 250-350: atrial flutter
  • 350+: Afib
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7
Q

Wide qrs complexes: 2 options

A
  • ventricular origin to rhythm OR

- bundle branch block

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8
Q

Tall QRS complexes

A
  • L vent hypertrophy (more muscle bigger impulse)
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9
Q

V1-V6 Precordal lead trend

A
  • V1 - deflection
  • V3/4 equal +/- deflection
  • V 6 + deflection
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10
Q

Multiple forms of p wave appearing on lead II @ rhythm > 100 BPM

A
  • MAT
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11
Q

Identifying which side of heart bundle block is on

A
  • ID the Precordal lead with biggest QRS interval, and that’s the side the block is on
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12
Q

Physiological effect of severe ischemia (injury)

A

La- altered Na/K -> decrease intracellular K -> membrane potential increases and ST segment increases

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13
Q

Classic triad of changes in infarction

A
  • ST elevation
  • q wave
  • inverted T wave
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14
Q

Left ventricular hypertrophy ECG changes

A
  • S wave in V1 + R wave in V 5-6 >= 35mm
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15
Q

Right ventricular hypertrophy ECG changes

A
  • R wave in V1 is >= S wave in V1
16
Q

ST elevation not related to infarction

A

Variant (prinzmetal) angina

17
Q

Abnormally large T wave

A
  • potentially hyperkalemia
18
Q

Etiology and morphology of MAT

A
  • typically due to COPD

- varying P wave shape

19
Q

L axis deviation in lead III of >= - 40

A
  • left anterior hemiblock
20
Q

QRS axis >= 120 in lead III

A
  • left posterior hemiblock
21
Q

Prolonged PR interval

A
  • 1st degree block
22
Q

P wave without following QRS complex upon progressive elongation of PR interval

A
  • 2nd AV block Mobitz wenckebach
23
Q

P wave without following QRS complex

A
  • 2nd degree Mobitz type 2
24
Q

P wave and QRS complex have no relationship to each other

A

3rd degree heart block

25
Q

Normal QRS complexes without p wave/ delayed qrs complex following normal rhythm without p wave stimulation

A
  • junctional rhythm/junctional escape rhythm
26
Q

Negative deflection of P wave in V1

A
  • right atrial enlargement
27
Q

Biphasic Pwave

A
  • left atrial enlargement
28
Q

Multiform P waves @ rhythm < 100 BPM on lead II

A

WAP

- wandering atrial pacemaker