ECG Flashcards
Depressed ST interval with inverted T wave
- indicator of ischemia
- may see reciprocal changes
ECG interpretation process
- rate
- rhythm
- P waves: I,II,aVL,aVF, V1-3: less than 2.5 boxes
- T waves: Limb leads 3; Q wave ~1 box
ST elevation with significant Q wave and again, inverted T wave
- indicator of infarction/injury
- clinically significant Q wave = at least 1 box (most reliable)
Prematurity of beats: 3 options
- atrial (if p waves present atria is at fault)
- junctional (no p waves normal QRS complex)
- ventricular (abnormally wide QRS complexes)
Irregularly irregular
- either Multifocal atrial tachycardia
Or - Afib (Afib atrial beats of 350+ BPM: looks like junk)
Epicardial vs endocaridal ischemia
- epicardial ischemia -> ST elevation
- endocaridal -> ST depression (possible becuase it happens inside out so epi ischemia is more like infarct)
Pace and etiology
- 150-250: SVT
- 250-350: atrial flutter
- 350+: Afib
Wide qrs complexes: 2 options
- ventricular origin to rhythm OR
- bundle branch block
Tall QRS complexes
- L vent hypertrophy (more muscle bigger impulse)
V1-V6 Precordal lead trend
- V1 - deflection
- V3/4 equal +/- deflection
- V 6 + deflection
Multiple forms of p wave appearing on lead II @ rhythm > 100 BPM
- MAT
Identifying which side of heart bundle block is on
- ID the Precordal lead with biggest QRS interval, and that’s the side the block is on
Physiological effect of severe ischemia (injury)
La- altered Na/K -> decrease intracellular K -> membrane potential increases and ST segment increases
Classic triad of changes in infarction
- ST elevation
- q wave
- inverted T wave
Left ventricular hypertrophy ECG changes
- S wave in V1 + R wave in V 5-6 >= 35mm