ECG Flashcards

1
Q

ECG

A

measures dynamic changes in membrane potential

recording of small extracellular signals produced by movement of APs through cardiac myocytes

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2
Q

ECG measures movement of positive charge

A

propagating depolarization

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3
Q

upwards deflection

A

depolarization moving towards the electrode

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4
Q

downwards deflection

A

depolarization away from the electrode

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5
Q

repolarization

A

toward the electrode

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6
Q

lead

A

difference in voltage

each lead looks at the heart from a unique angle and plane

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7
Q

bipolar leads

A

3 bipolar limb leads

looks at heart in vertical plane

einthoven’s triangle

body is an electrical “volume conductor”

output: voltage difference provides a direct representation of heart’s electrical activity in that lead

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8
Q

unipolar leads

A

3 augmented voltage limb leads

6 precordial or chest leads

measures the electrical potential at one electrode relative to a reference point (often a central terminal)

augmented voltage

circle of axes and einthoven’s triangle

have no negative pole

output: each lead records the potential at one electrode compared to a calculated average of the other two limb electrodes

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9
Q

standard bipolar leads

A

lead 1: RA- to LA +
lead 2: RA- to LL+ (views conducting system)
lead 3: LA- to LL+

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10
Q

einthoven’s law

A

lead 1+lead 3= lead 2 (like vectors)

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11
Q

advantages of ECG

A

simple and cheap

info about:
- anatomical orientation of heart
- chamber sizes
- arrhythmias and conduction blocks (is heart conducting normally)
- myocardial ischaemia (is heart getting enough blood)
- myocardial infarction
- congenital defects (abnormalities)
- changes in cardiac function with time/therapy

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12
Q

standard unipolar leads

A

aVR: RA +ve, (LA+LL) -ve
aVL: LA +ve, (RA+LL) -ve
aVF: LL +ve, (LA+RA) -ve

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13
Q

depolarization away from lead

A

negative deflection

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14
Q

depolarization toward lead

A

positive deflection

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15
Q

repolarization away from lead

A

positive deflection

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16
Q

repolarization towards lead

A

negative deflection

17
Q

chest leads unipolar “precordial”

A

view heard from horizontal plane

provides a view of the hearts electrical activity from a horizontal plane

provides info about specific areas of heard - good for diagnosing cardiac conditions

takes average of the three limb electrodes

18
Q

standard chest leads

A

add
V1 and V2 = septal
V3 and V4 = anterior
V5 and V6 = lateral

subtract
take average mean (1,2,3) = center of heart

19
Q

frontal plane leads

A

lead 1,2,3
aVL,aVR,aVF

20
Q

transverse plane leads

21
Q

what does each type of leads show us

A

scalar (1D) projection in that plane of the 3-D vector

12 leads allow us to reconstruct the amplitude and direction of this vector

22
Q

right axis deviation

A

RV hypertrophy
infants
tall and thin
left posterior hemiblock

23
Q

left axis deviation

A

obesity
pregnancy
left anterior hemiblock
LV hypertrophy

24
Q

extreme axis

A

dextrocardia
cardiac pacemaker

25
sinus rhythm
normal heart rhythm HR driven by SA node depolarization along normal conduction path subtle changes in HR occurs with each respiratory cycle (inspiration accelerated HR, expiration slows) - deepining of respirations accelerates these changes loss of this rhythm may be seen in diabetes
26
types of altered sinus
mechanisms that vary the firing frequency of the cardiac pacemaker cells pp interval is altered bradycardia Tachycardia respiratory sinus arrythmia
27
tachycardia
increases HR
27
bradycardia
decreases HR
28
respiratory sinus arrythmia
breathing changes alters vagus nerve output
29
atrioventricular conduction blocks
block between AVN and perkinji fibres
30
premature depolarizations
extrasystoles - coupled to normal AP, re-entrant pathway, comes out of no where occurs occasionally in most normal individuals - common in certain abnormal conditions
31
ectopic tachycardias
sporadic ectopic pacemaker AP in a re-entry loop begins and ends abruptly, could be a few beats or for hours, days ( and they often reoccur) happens in ventricles, but more commonly in atria
32
fibrillation
real trouble atrial or ventricular - jiggles (not pumping) multiple re-entry loops uncoordinated contraction - insufficient pumping of blood irregular type of contractions is inefficient in propelling blood probably represents a re-entry phenomenon not pumping blood to the rest of the body
33
right bundle branch block
no organized ventricle repolarizing wide QS slurred S in lateral leads altered T in septal leads
34
acute anterior infarction
in front of heart tissue blocked increased ST in V1-6, 1 and aVL reciprocal ST depression in 2,3 and aVF standing different in resting potential