Ears and nose Flashcards

1
Q

what type of hearing occurs in the middle ear

A
  • conductive hearing
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2
Q

what type of hearing occurs in the inner ear

A
  • sensorineural hearing
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3
Q

pathogenesis of AOM

A
  • eustachian tube dysfunction
  • allergies
  • viral infections
  • bacterial infections
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4
Q

what does AOM stand for?

A
  • acute otitis media
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5
Q

what does OME stand for?

A
  • otitis media with effusion (chronic OM)
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6
Q

what is the gold standard for dx of otitis media?

A
  • pneumatic otoscopy
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7
Q

AOM

A
  • rapid onset < 48 hours
  • si/sx of inflammation in middle ear
  • mild pain
  • bulging of TM
  • intense redness
  • +/- otorrhea
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8
Q

severe AOM

A
  • AOM
  • mod-severe pain OR fever > 102.2
  • bulging of TM
  • intense redness
  • +/- otorrhea
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9
Q

OME

A
  • inflammation of middle ear
  • liquid collection
  • no pain associated
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10
Q

middle ear effusion (MEE)

A
  • liquid in middle ear

- no reference to etiology, pathogenesis, pathology, or duration

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11
Q

otorrhea

A
  • discharge from ear

- can come from external auditory canal, middle ear, mastoid inner ear, intracranial cavity

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12
Q

otitis externa

A
  • infection of external auditory canal
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13
Q

tympanometry

A
  • measure acoustic immittance of ear

- function of ear canal pressure

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14
Q

causes of AOM

A
  • strep pneumoniae **
  • h. flu
  • m. catarrhalis
  • virus
  • ostiomeatal complex disfunction
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15
Q

causes of OME

A
  • osteomeatal complex/ eustachian tube dysfunction
  • sequelae of AOM
  • viral
  • unknown
  • bacteria
  • biofilm
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16
Q

when is watchful waiting appropriate tx for OME?

A
  • kids not at increased risk of speech, language, or learning problems
  • 3 mo from date of effusion onset/ dx
  • no suspected abnormalities in structure or TM
  • must reexamine kid at 3-6 mo intervals until effusion gone
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17
Q

tx options for OME

A
  • watch and wait
  • tubes
  • surgery
  • PO or topical prednisone (off guidelines)
  • antihistamines (off guidelines)
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18
Q

treatment guidelines for non-severe AOM

A
  • 6-23 mo. abx or watch and wait IF you have good f/u
  • 24 mo. bilat give abx
  • > 24 mo. abx or observe
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19
Q

abx options for treatment of AOM in kids

A
  • gold standard= amoxicillin 80-90 mg/kg BID to max dose of 1000 mg/dose
  • quinolone drops if perf present
  • Augmentin ES
  • bactrim if > 2 mo
  • 2nd or 3rd gen cephalosporin
  • IM ceftriaxone
  • azithromycin
  • clindamycin
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20
Q

treatment for AOM in adults

A
  • **worried about secondary causes like tumor*, sinusitis, allergies
  • f/u is mandatory
  • abx for kids plus tetracyclines and quinolones PO
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21
Q

general management of AOM (besides abx)

A
  • pain relief- APAP, NSAIDs, topical numbing drops
  • topical decongestants (avoid in peds)
  • no cold meds if kid < 2 yo
  • prob no cold meds in kid < 4 yo
  • maybe cold meds in kid > 4 yo
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22
Q

what should a normal TM look like

A
  • cone of light around 5 o’clock
  • pearly gray color
  • umbo present in middle
  • lateral process of malleus
  • TM intact
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23
Q

AOM follow up

A
  • should see improvement in 24-48 hours
  • reeval in 2 weeks
  • middle ear effusion can persist for a few months, does not mean AOM has not resolved
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24
Q

AOM prophylaxis

A
  • vaccines*
  • breast feeding
  • smoke free environment
  • no bottles in bed
  • abx prophylaxis NOT recommended
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25
otitis externa (OE)
- painful external ear | - **tragal movement pain
26
causes of OE
- pseudomonas aeruginosa most common bacteria - fungal - furuncles - allergies - injury
27
predisposing factors for OE
- inadvertent injury - allergies - psoriasis or eczema - seborrheic dermatitis - decreased canal acidity - irritants
28
si/sx of OE
- dainage thay may be foul smelling - painful otoscopic exam - red swollen ear canal - purulent debris inside ear canal - tenderness/pain over tragus - hearing loss - crusting outside of ear
29
OE treatment
- debridement - sx management with APAP or NSAIDs - topical abx - topical steroids - acetic acid - PO meds if they have comorbidities - consider using a wick for topical treatment
30
prevention of OE
- 1:1 mix of rubbing alcohol and white vinegar if TM is intact after swimming - no q tips
31
common causes of TM perforation
- infection - trauma - atmospheric overpressure - excessive water pressure - improper attempts at wax removal or ear cleaning***
32
treatment for TM perforation
- most heal spontaneously - refer if doesnt heal in 2 months, have significant hearing loss, or ossicular trauma - systemic abx if also hav otorrhea - surgery - paper patch, gelfoam plug, fibrin glue in office
33
auricular hematoma
- from direct trauma | - shearing forces separate anterior auricular perichondrium from cartilage
34
treatment of auricular hematoma
- early ID - drain - splint - compression
35
mastoiditis
- infection of mastoid air cells | - very concerning dx
36
treatment for mastoiditis
- consult - medical tx same as AOM - surgery to remove infected bone
37
what are the most concerning FB in the nose?
- button batteries - cause serious damage d/t alkaline tissue necrosis - can result in septal perforation within 4 hours - most other FB are removed at initial presentation and dont require ENT f/u
38
common locations for nasal FB
- most on floor of nasal passage
39
paired disk magnets in nose
- if have one in each nostril they are difficult to manually remove - prolonged attachment -> perforation of septum
40
clinical presentation of nasal FB
- usually in kids or adults with intellectual disabilities - mucopurulent nasal discharge - foul oder - unilateral epistaxis - nasal obstruction and mouth breathing
41
dx of nasal FB
- usually just done with otoscope or headlight - most FB are radiolucent so XR is not helpful - XR can detect button batteries or magnets and most pass easily through GIT
42
removal of intranasal FB
- using positive pressure techniques - direct instrumentation - most dont require surgery
43
intranasal FB that require ENT referral
- posterior FB not readily visualized - chronic or impacted FB with inflammation - penetrating or hooked FB - any FB that cant be removed after initial attempt
44
positive pressure techniques
- have pt occlude opposite nose and blow out | - occlude unaffected nostril and have someone blow into mouth
45
intranasal FB removal via instrumentation
- can be painful- use topical anesthesia - no nasal drops or pts with lodged button battery - may need procedural sedation if uncooperative - avoid forceps - can use hooks or catheters
46
prognosis of intranasal FB
- depends on size, shape and contents of FB - duration of FB - result of removal attempts
47
intermittent allergic rhinitis
- sx occur in response to specific exposure | - i.e. cat allergy
48
seasonal allergic rhinitis
- aka hay fever
49
persistent/ perennial allergic rhinitis
- sx occur year round | - i.e. mold allergy
50
what key feature distinguishes allergic rhinitis from other forms of rhinitis
- nasal itching
51
physical exam findings for allergic rhinitis
- nasal mucosa is edematous and pale - allergic shiners - nasal crease/ "allergic salute" - clear mucous - polyps - septal issues
52
management of allergic rhinitis
- environmental control - topical intranasal steroids** - 2nd gen antihistamines - montelukast - mast cell stabilizers - immunotherapy
53
nonallergic rhinitis
- chronic presence of nasal congestion, rhinorrhea, post nasal drainage - sx are perennial - no nasal or ocular itching
54
vasomotor rhinitis
- nonallergic | - intermittent sx from nonspecific irritants
55
mixed rhinitis
- combo of allergic and nonallergic rhinitis
56
gustatory rhinitis
- nonallergic | - rhinorrhea triggered by hot or spicy foods
57
rhinitis of pregnancy
- nonallergic - usually occurs in last six weeks - no known allergic cause - disappears 2 weeks after delivery
58
rhinitis medicamentosa
- caused by nasal decongestants - should only use decongestants for 3 days - can also be caused by antiHTN meds, ED meds, antidepressants, BZDs, NSAIDs, estrogen
59
what are the most common causes of rhinosinusitis
- rhinovirus - influenza virus - parainfluenza virus
60
acute rhinosinusitis duration
- < 4 weeks
61
chronic rhinosinusitis duration
- > 12 weeks | - usually seen in middle aged adults
62
viral rhinosinusitis
- sx usually dev after first day of inoculation - nose blowing propels fluid from nasal cavity to sinuses - decreased cilliary motility - mucosal edema, thick secretions, obstructed sinuses
63
clinical presentation of viral rhinosinusitis
- usually < 10 days - nasal congestion/ obstruction - purulent d/c- yellow or green - fever, fatigue, cough - hyposmia/ anosmia - ear pain, HA - facial pain/pressure worsens when bending forward - mucosal edema and narrowed middle meatus - inferior turbinate hypertrophy
64
diagnosis of rhinosinusitis (viral or bacteria)
- mainly clinical - bacterial cultures are unreliable- should be performed by ENT - xray not indicated for uncomplicated - CT is modality of choice if complicated
65
treatment for viral rhinosinusitis
- fluids - OTC decongestants - intranasal steroids - limit nasal spray to 3-5 days use - analgesics - PO antihistamines - mucolytics - saline irrigation (neti pot) - tx does NOT shorten duration
66
acute bacterial rhinosinusitis
- sx usually last > 10 days - purulent d/c - usually unilateral sinus pain and is exacerbated by bending forward - usually URI that begins to improve then worsens
67
predisposing factors for bacterial rhinosinusitis
- usually a complication of viral infection - nasal mechanical obstruction - tooth infection - impaired mucocilliary clearance - immunodeficiency - smoker
68
most common causes of acute bacterial rhinosinusitis
- s. pneumoniae - h flu - m catarrhalis
69
treatment for bacterial rhinosinusitis
- same supportive tx as viral - augmentin for 5-7 days - doxycycline for PCN allergy - macrolides NOT recommended - should respond in 3-5 days, if not switch to high dose augmentin
70
acute fungal rhinosinusitis
- occurs in immunocompromised pts - usually d/t apergillus - treat with amphotericin
71
acute fungal rhinosinusitis clinical manifestation
- facial pain and nasal congestion - facial numbness, diplopia if facial n involvement - epistaxis - necrotic tissue - gingival eschars, sloughing of septum
72
diagnosis of fungal rhinosinusitis
- urgent ENT referral | - biopsy/ debridement
73
chronic rhinosinusitis risk factors
- allergic rhinitis - asthma - perennial allergies - smoking - immunodeficiency - anatomic abnormalities - defects in mucocilliary clearance
74
cardinal si/sx of chronic rhinosinusitis
- anterior or posterior mucopurulent drainage - nasal obstruction - facial pain/ pressure/ fullness - hyposmia (adults), cough (kids) - need at least 2 cardinal sx to dx chronic - may also see evidence of mucosal thickening
75
subtypes of chronic rhinosinusitis
- CRS with nasal polyposis - CRS without nasal polyposis- most common - allergic fungal rhinosinusitis
76
CRS with nasal polyposis
- bilateral nasal polyps that are gray/white color - polyps are not painful - gradual worsening nasal congestion/ obstruction - sinus fullness and pressure - hyposmia or anosmia
77
what is the biggest way to tell the difference between nasal polyps and nasal turbinates
- polyps= painless | - turbinates= VERY sensitive
78
samter's triad
- combo of asthma, CRS with NP, and aspirin sensitivity
79
CRS without polyposis
- persistent sx with periodic exacerbations | - acute flares respond well to abx
80
allergic fungal RS
- chronic intense allergic reaction to colonizing fungi - usually in immunocompetent that show evidence of allergy to at least one fungi - presents subtly over years - gross facial asymmetry and/or visual changes
81
CRS diagnostics
- sinus CT - mucosal thickening bilaterally - obstruction of osteomeatal complex - sinus opacification without facial pain/ pressure/ HA
82
CRS with NP treatment
- PO steroids to shrink polyps - nasal steroids after - leukotriene inhibitor for maint
83
CRS without NP treatment
- PO steroids - abx for 6 weeks - follow with nasal steroids - 2nd gen antihistamines - leukotriene receptors
84
allergic fungal CRS treatment
- remove inspissated mucus | - prolonged PO steroids
85
anterior nosebleeds
- usually occur in kiesselbach's plexus | - most common type of nosebleed
86
posterior nosebleeds
- usually d/t sphenopalatine artery - not usually associated with trauma - can result in serious hemorrhage
87
cause of anterior nosebleeds
- nose picking - mucosal trauma/ irritation - low moisture content - allergic or viral rhinitis - FB - facial trauma - chronic excoriation - OTC nasal spray - nasal steroids - alcohol - anticoag pts
88
cause of posterior nosebleeds
- carotid artery aneurysm - nasal neoplasm - anticoag pts
89
treatment of anterior nosebleed
- compress nares for 5-10 min - pt lean foward - short acting topical nasal decongestants - topical cocaine or spray - electrocautery
90
treatment of posterior nosebleeds
- topical sympathomimetics - double ballon packs - ENT consult for packing - rhino rocket X 3 days with keflex
91
tinnitus
- perception of sound when no external noise present - ringing, buzzhing, hissing, whistling, swooshing - men > women - more common in smokers
92
subjective tinnitus
- noise only perceivable to patient - traceable to auditory or neurological rxn to hearing loss - most common type of tinnitus - usually occurs d/t RTI
93
objective tinnitus
- head or ear noises that are audible to others - produced by internal fn in body circulation - rare
94
causes of tinnitus
- ototoxic meds - hearing loss- sensorineural - otosclerosis - chiari malformations - often d/t ear wax plug
95
diagnosis of tinnitus
- requires good history - HEENT exam, eval TM, CN exam, auscultate neck - MRI if unilateral esp with hearing loss- r/o retrocochlear lesion
96
treatment of tinnitus
- avoid excessive noise and ototoxic drugs - correct comorbidities - hearing aids - sound and behavioral therapy - TMJ tx - cochlear implants
97
nystamgus
- repetitive uncontrolled movements of eyes - cause reduced vision, depth perception, and affect balance/ coord - usually sx of another eye or medical prob
98
infantile nystagmus
- usually horizontal swinging | - congenital cause
99
spasmus nutan nystagmus
- occurs in kids - usually improves on its own without tx - kids often nod or tilt their head - eyes can move in any direction
100
acquired nystagmus
- develops later in childhood or adulthood | - cause is usually unknown
101
causes of nystagmus
- neuro problems - lack of eye development - astigmatism - congenital cataracts - inflammation of inner ear - medications (anti-seizure meds) - CNS diseases
102
diagnosis of nystagmus
- history - visual acuity assessment - det lens power needed to correct error
103
treatment of nystagmus
- glasses/ contacts - usually improves with time - treat underlying cause
104
vertigo
- illusion of motion, rotary sensation | - associated n/v, sweating, pallor
105
causes of vertigo
- hypofunction- vestibular neuritis - hyperfunction- BPPV - central neurological disorder- migraines
106
central vertigo
- gradual onset - cerebellum and brainstem affected - disproportionate gate - visual field defects - hemisensory loss - limb weakness - diplopia - slurred speach - difficulty swallowing
107
peripheral vertigo
- sudden onset - semicircular canals, otolith organs affected - tinnitus - hearing loss - aural fullness
108
what is the most common cause of severe spontaneous vertigo
- vestibular neuritis or labrythitis
109
common cause of neuritis or labrythitis
- usually ear infection - disrupts transmission of sensory info - typically viral cause
110
neuritis
- inflammation of vestibular n - NO hearing impact - dizziness and vertigo sx
111
labrynthitis
- inflammation of labyrinth in inner ear - hearing changes - dizziness and vertigo sx
112
treatment for neuritis and labrythitis
- usually pretty self limited : 2-3 weeks - steroids - antivert meds- meclizine - antiemetics- zofran - antivirals and abx rare
113
benign paroxysmal positional vertigo
- crystal gets displaced into semicircular canal - excessive response to head movement - commonly in females > 60 - rapid onset dizziness or spinning - short duration
114
clinical manifestations of BPPV
- clockwise rotary nystagmus - rapid onset dizziness lasting sec- min - precipitated by sudden head movement - nystagmus - nausea - no impact on hearing
115
diagnosis of BPPV
- dix- hallpike maneuver to determine which canal is affected - may need MRI/CT to r/o other causes
116
epley maneuver
- tx for BPPV in posterior canal
117
deep head hanging maneuver
- tx for BPPV in superior canal
118
lamperet (BBQ) maneuver
- tx for BPPV in lateral canal
119
tx of BPPV
- epley, deep head hanging, or lampered maneuvers - symptomatic tx - antihistamines - antiemetics - bzds - neuro referral - PT - surgery (rare)
120
Meniere's disease
- inner ear disorder -> vertigo, tinnitus, hearing loss, feeling of fullness in ear - frequent "drop attacks" - usually in one ear - sudden onset of sx that are separated by periods of time - usually seen in adult females
121
what is the cause of meniere's disease
- fluid build up in inner ear -> impaired balance and hearing signals to brain
122
associated diseases of meniere's
- allergies - stress - viral infections
123
sx associated to diagnose meniere's disease
- usually done by ENT - 2+ episodes of vertigo lasting 20 min each - tinnitus - temporary hearing loss - fullness in ear - can get MRI and labs to r/o more serious causes
124
treatment for meniere's disease
- meclizine - bzds - salt restriction and diuretics - dietary changes- no caffeine, alcohol, chocolate - cognitive tx - gentamicin injection (caution)- - steroids - pressure pulse tx
125
drugs that can cause vertigo
- aminoglycosides - antidepressants - anxiolytics - furosemide - amiodarone - ASA - alcohol - cocaine
126
central causes of vertigo
- migraines - tumors of CN VIII - chiari malformations - brain ischemia - MS - brain injuries
127
eustachian tube dysfunction
- occurs when tube is blocked or doesn't open - air pressure outside TM > air pressure in middle ear - TM gets pushed in - TM becomes tense, doesn't vibrate or move
128
causes of eustachian tube dysfunction
- cold or other sinus/throat infections** - sx can persist for 1 week after all other sx resolve - allergies - blockages- i.e. enlarged adenoids - air travel
129
clinical manifestations of eustachian tube dysfunction
- fullness in ear** - possible hearing loss - popping when yawning or swallowing - ear pain
130
dilatory eustachian tube dysfunction
- tube doesnt dilate - causes hearing loss - see effusions, scarring, and thickening of TM - treat underlying cuase
131
patulous eustachian tube dysfunction
- valve incompetency - pt hears own voice amplified - movement of TM on otoscopic exam - tx only if sx severe and last > 6 weeks
132
treatment of eustachian tube
- swallow, yawn, or chew to increase flow of air in and out of tube - valsalva maneuver - decongestants - antihistamines - steroid nasal spray - if sx are persistent refer to ENT
133
what type of hearing loss is associated with outer ear problems?
- conductive hearing loss
134
what type of hearing loss is associated with middle ear problems?
- conductive hearing loss
135
what type of hearing loss is associated with inner ear problems
- sensorineural hearing loss
136
exostosis
- multiple bony growths
137
osteoma
- solitary bony growth
138
what is the most common cause of middle ear hearing problems?
- eustachian tube dysfunction
139
presbycusis
- age related hearing loss
140
what are endocrine issues associated with hearing loss?
- DM - hyperthyroidism - anemia
141
what are autoimmune diseases associated with hearing loss?
- RA - SLE - polyarteritis nodosa
142
what drugs are commonly associated with hearing loss?
- gentamycin, tobramycin, tetracycline - cisplatin, 5-FU - high dose ASA - PDE-5 inhibitors - quinine, chloroquine - cocaine - lead, mercury, cadmium, arsenic
143
vestibular schwannomas
- overproduction of schwann cells on vestibular nerve - slow growing - typically unilateral
144
neurofibromatosis type 2 (NF2)
- mutation in merlin gene - causes bilateral vestibular schwannomas - more likely to dev other NS tumors
145
clinical presentation of vestibular schwannomas
- disequilibrium - hearing loss- asymmetric sensorineural loss - tinnitus - if tumor is large enough can compress other CN
146
risk factors for vestibular schwannomas
- NF2 mutation - childhood low dose radiation - hx of parathyroid adenoma - ? cellphones - exposure to loud noise
147
diagnosis of vestibular schwannomas
- audiometry- best initial screening - CT - MRi
148
management of vestibular schwannomas
- surgery- good longterm control - radiation - observation
149
indications for conservative management of vestibular schwannomas
- > 60 years old - significant comorbidities - small tumor size - lack of sx - risk further hearing loss - pt preference
150
complications of vestibular schwannomas
- hearing loss - facial weakness - vestibular disturbances - persistent HA - CSF leakage - hemorrhage - infections
151
causes of ear barotrauma
- flying- most common - driving/hiking up mountains - diving - blast injuries
152
what cause of barotrauma is associated with inward rupture of TM
- diving
153
clinical manifestations of ear barotrauma
- ear pressure- most common - pain - hearing loss - tinnitus - vertigo
154
prevention for barotrauma
- plan for pressure changes - avoid flying/ diving - decongestants and antihistamines - ear plugs - ventilation tubes - surgical tympanoplasty if required