Dysfunctional Labor Flashcards

1
Q

physiologic changes of the uterus in labor

A

upper segment contracts and retracts to expel the fetus

lower segment along the cervix becomes thinner and passive

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2
Q

physiologic changes of the cervix in labor

A

becomes soft, pliable and dilated structure

result of collagenolysis, increased hyaluronic acid, and decreased dermatan sulfate

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3
Q

abnormalities of active labor

A

protraction - slower than normal

arrest - complete cessation of progress

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4
Q

what does an arrest in the latent phase imply?

A

true labor has not begun

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5
Q

what is considered a prolonged latent phase?

A

> 20 hrs for primiparous

> 14 hrs for multiparous

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6
Q

causes of prolonged latent phase

A

lack of substantial cervical change
excessive use of sedatives or analgesics
fetal malposition

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7
Q

prolonged latent phase management

A

sleep - true vs false labor

morphine - 62-85% will progress to active phase

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8
Q

protraction disorder

A

protraction of dilation - dilation less than norm

protraction of descent - descent less than norm

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9
Q

if 2 or more hours elapse with no cervical dilation, what has occured?

A

arrest of dilation

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10
Q

if no change in descent has occured within 1 hour what is the dx?

A

arrest of descent/station

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11
Q

what is the risk associated with active phase abnormalities?

A

increased risk of perinatal mortality

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12
Q

etiology of active phase abnormalities

A

inadequate uterine activity
cephalopelvic disproportion
fetal malposition
anesthesia

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13
Q

dystocia

A

difficult labor

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14
Q

3 P’s of dystocia

A
power = uterine contractions or expulsive forces
passenger = position, size or presentation of fetus
passage = pelvic bone contractures
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15
Q

what needs to occur before dystocia is dx?

A

an adequate trial of labor has been attempted

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16
Q

when should augmentation be considered?

A

if contractions are less than 3 in 10 min period or the intensity is less than 25 mmHg

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17
Q

what needs to be assessed prior to giving oxytocin in protraction and arrest disorders?

A

maternal pelvis
fetal position
station
maternal and fetal status

18
Q

how can “power” be assessed?

A

intrauterine pressure catheter (IUPC)

requires membranes to be ruptured

19
Q

what are montevideo units (MVU)?

A

measure the peaks of contractions in mmHg in a 10 minute period

normal = > 200 MVU for at least 2 hrs

20
Q

what needs to be documented before proceeding to C-section?

A

adequate contractions for at least 4 hours

21
Q

MOA of pitocin

A

increased intracellular CA which results in increase in actin/myosin activity

22
Q

cephalopelvic disproportion (CPD)

A

disparity between the size of the maternal pelvis and the fetal head that precludes vaginal delivery

23
Q

what indicates an increased likelihood of CPD?

A

primiparous woman who presents in labor with an unengaged head

24
Q

what is considered an abnormal fetal presentation?

A

anything other than vertex occiput anterior presentation

25
abnormal fetal structures that can cause dystocia
macrosomia shoulder dystocia fetal anomalies
26
persistent occipitotransverse (OT) position
occurs when the head fails to rotate and flex into the OA position may be caused by CPD
27
transverse arrest of descent
persistent OT position with arrest of descent for ≥ 1 hr
28
management of persistent OT position
if pelvis is adequate, infant is not macrosomic, and contractions are inadequate = start Pitocin and engage rotation of fetus if pelvis is inadequate or fetus is macrosomic = C-section
29
what is the normal outcome of persistent occipitoposterior (OP) position?
labor usually proceeds without issue second stage may be prolonged associated with more back discomfort
30
management of persistent OP position
observation and delivery OR operative vaginal delivery with vacuum or forceps
31
macrosomia
fetus weighing 4500 g (10 lbs!!!)
32
large for gestational age
birth weight equal to or greater than the 90% for a given gestational age
33
fetal abnormalities that may cause dystocia
``` hydrocephalus fetal ascites immune hydrops conjoined twins locked twins ```
34
risk factors that cause macrosomia
``` maternal DM hx of macrosomia maternal obesity weight gain during pregnancy multiparity male fetus > 40 wks gestation ethnicity maternal birth weight maternal height maternal age < 17 y/o ```
35
risk factors associated with macrosomia
maternal morbidity due to post-partum hemorrhage fetal morbidity and mortality due to fracture of clavicle, shoulder dystocia, and brachial plexus injury
36
what is recommended course of action for macrosomia?
prophylactic C-section
37
antepartum risk factors for shoulder dystocia
``` fetal macrosomia maternal diabetes obesity post-term gestation short stature hx of macrosomic birth hx of shoulder dystocia ```
38
labor risk factors for shoulder dystocia
labor induction epidural prolonged labor operative vaginal deliveries
39
possible fetal outcomes of shoulder dystocia
brachial plexus injuries fractured clavicle or humerus hypoxic-ischemic encephalopathy death
40
management of shoulder dystocia
McRobert's maneuver - hyperflexion and abduction of maternal hips suprapubic pressure fetal rotational maneuvers proctoepisotomy Zavanelli maneuver - cephalic replacement *last resort
41
is shoulder dystocia an obstetric emergency?
YES! call for help - anesthesia and NICU