Dysfunctional Labor Flashcards

1
Q

physiologic changes of the uterus in labor

A

upper segment contracts and retracts to expel the fetus

lower segment along the cervix becomes thinner and passive

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2
Q

physiologic changes of the cervix in labor

A

becomes soft, pliable and dilated structure

result of collagenolysis, increased hyaluronic acid, and decreased dermatan sulfate

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3
Q

abnormalities of active labor

A

protraction - slower than normal

arrest - complete cessation of progress

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4
Q

what does an arrest in the latent phase imply?

A

true labor has not begun

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5
Q

what is considered a prolonged latent phase?

A

> 20 hrs for primiparous

> 14 hrs for multiparous

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6
Q

causes of prolonged latent phase

A

lack of substantial cervical change
excessive use of sedatives or analgesics
fetal malposition

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7
Q

prolonged latent phase management

A

sleep - true vs false labor

morphine - 62-85% will progress to active phase

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8
Q

protraction disorder

A

protraction of dilation - dilation less than norm

protraction of descent - descent less than norm

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9
Q

if 2 or more hours elapse with no cervical dilation, what has occured?

A

arrest of dilation

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10
Q

if no change in descent has occured within 1 hour what is the dx?

A

arrest of descent/station

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11
Q

what is the risk associated with active phase abnormalities?

A

increased risk of perinatal mortality

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12
Q

etiology of active phase abnormalities

A

inadequate uterine activity
cephalopelvic disproportion
fetal malposition
anesthesia

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13
Q

dystocia

A

difficult labor

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14
Q

3 P’s of dystocia

A
power = uterine contractions or expulsive forces
passenger = position, size or presentation of fetus
passage = pelvic bone contractures
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15
Q

what needs to occur before dystocia is dx?

A

an adequate trial of labor has been attempted

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16
Q

when should augmentation be considered?

A

if contractions are less than 3 in 10 min period or the intensity is less than 25 mmHg

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17
Q

what needs to be assessed prior to giving oxytocin in protraction and arrest disorders?

A

maternal pelvis
fetal position
station
maternal and fetal status

18
Q

how can “power” be assessed?

A

intrauterine pressure catheter (IUPC)

requires membranes to be ruptured

19
Q

what are montevideo units (MVU)?

A

measure the peaks of contractions in mmHg in a 10 minute period

normal = > 200 MVU for at least 2 hrs

20
Q

what needs to be documented before proceeding to C-section?

A

adequate contractions for at least 4 hours

21
Q

MOA of pitocin

A

increased intracellular CA which results in increase in actin/myosin activity

22
Q

cephalopelvic disproportion (CPD)

A

disparity between the size of the maternal pelvis and the fetal head that precludes vaginal delivery

23
Q

what indicates an increased likelihood of CPD?

A

primiparous woman who presents in labor with an unengaged head

24
Q

what is considered an abnormal fetal presentation?

A

anything other than vertex occiput anterior presentation

25
Q

abnormal fetal structures that can cause dystocia

A

macrosomia
shoulder dystocia
fetal anomalies

26
Q

persistent occipitotransverse (OT) position

A

occurs when the head fails to rotate and flex into the OA position

may be caused by CPD

27
Q

transverse arrest of descent

A

persistent OT position with arrest of descent for ≥ 1 hr

28
Q

management of persistent OT position

A

if pelvis is adequate, infant is not macrosomic, and contractions are inadequate = start Pitocin and engage rotation of fetus

if pelvis is inadequate or fetus is macrosomic = C-section

29
Q

what is the normal outcome of persistent occipitoposterior (OP) position?

A

labor usually proceeds without issue
second stage may be prolonged
associated with more back discomfort

30
Q

management of persistent OP position

A

observation and delivery
OR
operative vaginal delivery with vacuum or forceps

31
Q

macrosomia

A

fetus weighing 4500 g (10 lbs!!!)

32
Q

large for gestational age

A

birth weight equal to or greater than the 90% for a given gestational age

33
Q

fetal abnormalities that may cause dystocia

A
hydrocephalus
fetal ascites
immune hydrops
conjoined twins
locked twins
34
Q

risk factors that cause macrosomia

A
maternal DM
hx of macrosomia
maternal obesity
weight gain during pregnancy
multiparity
male fetus 
> 40 wks gestation
ethnicity
maternal birth weight
maternal height
maternal age < 17 y/o
35
Q

risk factors associated with macrosomia

A

maternal morbidity due to post-partum hemorrhage

fetal morbidity and mortality due to fracture of clavicle, shoulder dystocia, and brachial plexus injury

36
Q

what is recommended course of action for macrosomia?

A

prophylactic C-section

37
Q

antepartum risk factors for shoulder dystocia

A
fetal macrosomia
maternal diabetes
obesity
post-term gestation
short stature
hx of macrosomic birth
hx of shoulder dystocia
38
Q

labor risk factors for shoulder dystocia

A

labor induction
epidural
prolonged labor
operative vaginal deliveries

39
Q

possible fetal outcomes of shoulder dystocia

A

brachial plexus injuries
fractured clavicle or humerus
hypoxic-ischemic encephalopathy
death

40
Q

management of shoulder dystocia

A

McRobert’s maneuver - hyperflexion and abduction of maternal hips

suprapubic pressure
fetal rotational maneuvers

proctoepisotomy

Zavanelli maneuver - cephalic replacement *last resort

41
Q

is shoulder dystocia an obstetric emergency?

A

YES! call for help - anesthesia and NICU