DSA Low Back Pain Flashcards
Acute low back pain
<12 weeks
high likelihood to resolve (no residual loss of fxn)
simple or no management required
Chronic low back pain
> 12 weeks
high risk for loss of fxn/disability
tx resistant (need many different types of management)
Risk factors for low back pain
lifting weight @ work smoking depression obesity inactivity
What risk factors increase perception of pain?
smoking & depression
Protective factors for low back pain
regular exercise (exercise is MOST important factor)
education + exercise (education is not effective alone)
What are the common etiologies of low back pain?
Mechanical (97% of all causes)
Visceral Disease
Non-mechanical
Describe visceral etiologies for low back pain
likely present as acute low back pain w/ other sxs
often is referred pain from abdominopelvic structures (GI organs, abdominal aorta, renal, GU organs, endometriosis)
Describe non-visceral etiologies for low back pain
neoplasms (wt loss, deep bone pain @ night)
infection (fever, chills)
inflammatory (ankylosing spondylitis, psoriatic arthritis)
What genotype is associated with low back pain due to inflammation?
HLA B27
What are the major mechanical MSK etiologies of low back pain?
sprain/strain/overuse syndrome
piriformis syndrome
psoas syndrome
short leg syndrome
Etiology of Sprain/Strain/Overuse syndrome
injury & stress on soft tissue structures (either injury to muscles or injury to ligaments)
Pain pattern & DX of sprain/strain/overuse syndrome
aching pain over injured structure
DX by PE & palpation, by exclusion
Etiology of Piriformis syndrome
hypertonicity of piriformic muscle, nerve entrapment of sciatic nerve (as exits btwn priformis & superior gemelllus muscles)
Pain pattern & DX of piriformis syndrome
worse w/ sitting & @ risk if have trauma or overuse
DX w/ PE & + FAIR test (reproduces pain w/ Lat recumb, flex, IR & AB)
Etiology of psoas syndrome
chronic hypertonicity of psoas muscle (from T12-L4 to greater trochanter)