Diving Related Injuries and Treatment Flashcards

1
Q

3 Objective of Recompression Treatment

A

1) Compress gas bubbles
2) Allow for bubble re-absorption
3) Increase ppO2 (through tissue and blood oxygen)

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2
Q

TT5 Indications

A

Type I DCS relieved within 10 minutes
Asymptomatic omitted decompression
Follow-up treatments for residual symptoms
Treatment of resolved symptoms following in-water recompression
Gas gangrene
CO Toxicity

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3
Q

TT6 Indications

A

Type I DCS not relieved within 10 minutes (cutis marmorata)
Type II DCS
Asymptomatic Omitted Decompression
Recurrence of symptoms shallower than 60 fsw
AGE
Severe Carbon Monoxide poisoning
Cyanide poisoning
Smoke inhalation
Symptomatic following treatment of in-water DCS
Symptomatic during SI of SURD

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4
Q

TT6A Indications

A

Symptomatic blow up from >60 fsw

Worsening or unchanged AGE/Type II DCS symptoms at 20 minutes at 60 fsw

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5
Q

POIS Definition

A

Expansion of gas trapped in the lung during ascent with over expansion and rupture of alveolar air sacks

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6
Q

Types of POIS

A

Mediastinal emphysema, Subcutaneous emphysema, AGE, Pneumothorax

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7
Q

AGE Definition

A

Alveolar rupture with air injected into capillary then from pulmonary veins to left ventricle and is pumped into systemic arteries.
- Brain is most significant site of an embolus, like CVA

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8
Q

Time course of DCS

A
  • 60% presents < 3 hrs
  • 83% presents < 8 hrs
  • 98% presents < 24 hrs
  • CNS DCS may have shorter latency (90% symptomatic <3hrs)
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9
Q

NIggles

A

odd fleeting aches and pains
may herald typical limb pain DCS
treat as Type I DCS

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10
Q

Type 1 Pain treated as type II

A

Cutis Marmorata

Thoracic/abdominal pain

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11
Q

Spinal DCS

A
  • Associated with short, deep dives
  • Area of damage most commonly in anterior horn
  • White matter more affected
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12
Q

Manifestations of cerebral DCS

A
  • Motor Deficits
  • Sensory Changes
  • Personality Changes
  • Memory Loss
  • Acute Psychosis
  • Loss of coordination
  • Ataxia
  • Extreme Fatigue
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13
Q

Type II DCS Select Ocular Manifestations

A
  • Nystagmus
  • Diplopia
  • Visual Fields (VF) Defects
  • Scotomata
  • Homonymous Hemianopsia
  • Central Retinal artery occlusion
  • Optic Neuropathy
  • Ocular Muscle impairment
  • Eyelid muscle pain
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14
Q

Symptoms of Hypercapnia

A
  • Rapid pulse and respiration
  • Unconsciousness
  • Dizziness
  • Eyes burning
  • Progressive Mental Confusion
  • Headache
  • SOB
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15
Q

Mechanism of CO, Cyanide, and sulfide Toxicity

A

These toxicities decrease the likelihood of oxygen attaching to red blood cells via hemoglobin

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16
Q

Contraindications for HBOT

A
Chronic Sinusitis
Congenital Spherocytosis
Emphysema with CO2 retention
History of optic neuritis
History of reconstructive ear surgery
History of spontaneous pneumothorax
History of thorax surgery
Seizure disorders
Sickle Cell Anemia
URI