Diseases of Pulp and Periodontium Flashcards
Clinical features of Pulp Hyperaemia
pain lasts seconds
pain on hot/cold/sweet
pain resolves after stimulus
caries close to pulp but still treatable
Clinical features of Acute Pulpitis
constant severe pain
reacts to thermal stimuli
poorly localised
reffered pain
NO/min response to analgesics
Diagnosis of Acute Periodontitis
TTP
non-vital tooth
increase in mobility
loss of clarity of lamina dura
RG shadow (apical granuloma)
Widening of PDL
Traumatic Periodontitis
caused by bruxism
TTP
Normal vitality
generalised widening of PDL
Tx = occlusal adjustment/ therapy for bruxism
most common abscess
acute apical abscess
signs of inflammation
heat
redness
swelling
pain
loss of function
site of swelling depends on
position of tooth in arch
root length
muscle attachment
potential spaces in proximity to lesion
local factors for antibiotics
toxicity
airway compromisation
dysphagia
trismus
lymphadenitis
location
systemic factors for antibiotics
immunocompromised patients:
acquired (HIV)
drug induced (steroids, cytostatics)
blood disorder (leukaemias)
diabetes
elderly
reversible pulpitis
level of inflammation where possible to return to normal when stimuli removed
mild pain with stimuli
no pain w/o stimuli
<5s pain
no mobilty
no pain on percussion
irreversible pulpitis
higher level of inflammation where pulp is damaged beyond recovery
sharp throbbing
severe pain w/ stimuli
pain >5s
spontaneous pain
what is periapical granuloma?
mass of chronic inflammed granulation tissue at apex
not true granuloma (no epithelioid histiocytes)
eitiology of PA Cyst (Radicular cyst)
caries/truma/perio
pulp necrosis
apical bone inflammation
dental granuloma
stimulates epithelial rests of Malassez
epithelial proliferation
PA cyst forms
