Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

Pathology 101 Seneca College > Disease Process & Inflammation > Flashcards

Disease Process & Inflammation Flashcards

You may prefer our related Brainscape-certified flashcards:
Physics 101 flashcards Psychology 101 flashcards Chemistry 101 flashcards Biology 101 flashcards Organic Chemistry 101 flashcards Sociology 101 flashcards
1
Q

What is Pathology ?

A

The study of DISEASE and the diagnosis of disease; pathos = suffering, feeling

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What is PATHOPHYSIOLOGY ?

A

The study of the functional CHANGES caused by disease;

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What is ETIOLOGY?

A

The study of causation; the CAUSE of disease/pathologies.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What is MANIFESTATIONS?

A

Indications or REPRESENTATIONS of disease.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What is COMPLICATIONS?

A

A SECONDARY disease, an accident, or a negativereaction occurring during the course of disease.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What is the SIGNS ?

A

An OBJECTIVE finding of disease (determined by healthcare professional).

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What is SYMPTOMS?

A

A SUBJECTIVE finding of disease (determined by the patient).

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

PATHOLOGY: Flu PATHOPHYSIOLOGY: (Changes) ETIOLOGY: Virus (Cause) MANIFESTATIONS: (Indications) COMPLICATIONS: (Secondary) SIGNS: Cough, ———–, Fever (Objective) SYMPTOMS: Headache, Sore ———–(Subjective)

A

PATHOLOGY: Flu PATHOPHYSIOLOGY: Upper Respiratory Infection (Changes) ETIOLOGY: Influenza Virus (Cause) MANIFESTATIONS: High Fever (Indications) COMPLICATIONS: Pneumonia (Secondary) SIGNS: Cough, Sweating, Fever (Objective) SYMPTOMS: Headache, Sore Throat (Subjective)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What is EPIDEMIOLOGY ?

A

The study of health-determinant PATTERNS in society.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What is INCIDENCE? Measurement of the risk of

A

Measurement of the risk of developing a new condition in a given period of time; # of NEW CASES per population in a given period of time. Ie. How progressive a disease is.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What is PREVALENCE ? Measurement of total number…….

A

PREVALENCE Measurement of total number of cases of a new condition in a population; # of TOTAL CASES in a population divided by # of individuals in population. Ie. How common a disease is.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

RISK The potential or ———— of developing a condition.

A

Chance

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

MORTALITY is —-

A

DEATH which measures of the number of deaths in a population during an interval of time.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

MORBIDITY is ————-state

A

Morbidity is a diseased state which measures the amount of those in a diseased state in a population (often measured as incidence rate or prevalence rate).

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What is Tissue Perfusion ?

A

* Delivery of arterial blood to the capillary bed of a tissue (capillary à tissue) * Measured in millilitres of blood per 100g of tissue * Allows oxygenation of tissue * Necessary for maintenance of cells/tissue

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Decreased blood supply to a tissue and decreased tissue perfusion is called ?

A

(Ischemia)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

A. Results in to tissue decreased B. Oxygenation and can lead to——-

A

A. (Hypoxia) B. Infraction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

When blood is not properly delivered to the cells of the tissue, damage can occur to both the ——- and the surrounding ——————-

A

Cells and the surrounding interstitial fluid

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Insufficient blood flow to affected region: renal, ————–, —————, or gast——-

A

cardiac, cerebral, gastrointestinal

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

malfunction of the Interstitial Fluid leads to

A

inflammatory response

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Cell ithat lacks O2 leaves to——-

A

Ischemia, infarction/necrosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Intravascular Fluid is——

A

The space contained within blood vessels. The main intravascular fluid is blood. That portion of the total body fluid contained within blood and lymphatic vessels.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Interstitial fluid is —–?

A

Interstitial fluid is a thin layer of fluid which surrounds the body’s cells. Interstitial fluid has become useful in the monitoring of glucose levels in people with diabetes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Interstitial fluid consists of———–?

A

Interstitial fluid consists of a water solvent containing sugars, salts, fatty acids, amino acids, coenzymes, hormones, neurotransmitters, white blood cells and cell waste-products.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

Intracellular fluid is the place———–

A

Intracellular fluid is the place where most of the fluid in the body is contained. This fluid is located within the cell membrane and contains water, electrolytes and proteins. Potassium, magnesium, and phosphate are the three most common electrolytes in the ICF. It is the Fluid of Cytosol/Cytoplasm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

What is Extracellular Fluid ?

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

TISSUE PERFUSION is Impaired due to the cause of ———————-?

A

1, Changes in blood circulation,

  1. Diabetes mellitus,
  2. Hypertension,
  3. Organ damage,
  4. Obesity/compression
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

The local effects of the TISSUE PERFUSION

edema (———————)

weakened pulse

——————— (especially in extremities)

damp/——————

changes in ———————

Eg. Chronic —————————-

A

Edema (water retention)

weakened pulse

numbness (especially in extremities)

damp/cold skin

changes in temperature

Eg. Chronic Venous Insufficiency

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

SYSTEMIC EFFECTS

of

Tissue Perfusion

—————-: mood swings/irritability, confusion,

lethargy, altered speech, slower pupil reaction to light

—————————: low urine output, high blood pressure, proteinuria

————————-: nausea, abdominal pain

——————-: chest pain, hypotension, changes in respiration

A

Cerebral: mood swings/irritability, confusion,

lethargy, altered speech, slower pupil

Reaction to light

Renal: low urine output, high blood pressure,

proteinuria

Gastrointestinal: nausea, abdominal pain

Cardiac: chest pain, hypotension, changes in

respiration

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
30
Q

Compensation of the Tissues Perfusion Impaired

Alteration in arterial blood pressure (reninangiotensionpathway ——> ———— bp)

Vasodilation/vasoconstriction of blood vessels

Microvascular remodeling

Capillary ——————–

Alteration in blood ——————- (increase fluidity via inflammatory mediators)

Cell ——————- (cells commit suicide)

A

Alteration in arterial blood pressure (reninangiotensionpathway ——> increased bp)

Vasodilation/vasoconstriction of blood vessels

Microvascular remodeling

Capillary recruitment

Alteration in blood viscosity (increase fluidity via inflammatory mediators)

Cell apoptosis (cells commit suicide)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
31
Q

ISCHEMIA:

——————— blood supply to a tissue

————————- tissue perfusion

Results in decreased oxygenation to tissue (——————-)

Can lead to——————–

A

ISCHEMIA:

Decreased blood supply to a tissue

Decreased tissue perfusion

Results in decreased oxygenation to tissue (Hypoxia)

Can lead to Infarction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
32
Q

Premature cell death (Necrosis) due to prolonged ischemia (lack of blood and oxygen to cell tissue) ?

A

INFARCTION

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
33
Q

Inadequate oxygen supply to the cells and tissue

Prolonged hypoxia can lead to Cyanosis

A

HYPOXIA

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
34
Q

“The blue disease”

Blue colouration due to large amounts of

Deoxygenated hemoglobin (blood vessels become

Darker red ——-> appear blue through skin)

Most recognized on the lips (central) and

Extremities/fingernails (peripheral)

A

CYANOSIS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
35
Q

———————– CARRIES OXYGEN

THROUGHOUT THE BODY

A

HEMOGLOBIN

Hemoglobin is a protein in your red blood cells that carries oxygen to your body’s organs and tissues and transports carbon dioxide from your organs and tissues back to your lungs. If a hemoglobin test reveals that your hemoglobin level is lower than normal, it means you have a low red blood cell count (anemia)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
36
Q

Casues Of Systemic Hypoxia ?

—————- exercise

High ——————–

Deep-sea—————

Premature birth (lungs ———————)

———————– (due to respiratory arrest, opiates)

—————— (collapsed alveoli, lung damage)

Pulmonary ————-

Ane—-

————–monoxide poisoning, ———————– poisoning

A

Casue Of Systemic Hypoxia

Strenuous exercise

High altitudes

Deep-sea diving

Premature birth (lungs not fully developed)

Hypoventilation (due to respiratory arrest, opiates)

Heart shunts (collapsed alveoli, lung damage)

Pulmonary embolism

Anemia

Carbon monoxide poisoning, cyanide poisoning

Methemoglobinemia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
37
Q

HYPOXIA Effects – Mild S/S: Headache
Haadache

  • *Fever**
A

Shortness of breath (SOB)
Euphoria

Nausea

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
38
Q

Severe Effects of Hypoxia

Loss of ———————
Seizures
—————
—————- (blue)
——————- – if oxygen replaced by other toxin
Death

A

Loss of consciousness
Seizures
Coma
CYANOSIS (blue)
“cherry-red” – if oxygen replaced by other toxin
Death

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
39
Q

Systemetic Compensation HYPOXIA

Most tissues –> ——————–
allows increased tissue perfusion to that specific tissue

Lungs ————————–!!
HPV –————————————————- —–> redirect blood flow from poorly ventilated lung areas to well ventilated lung areas

—————————————, ————————— (increase RBC’s), Acclimatization, etc.

A

Vasodilation

Vasconstriction

Hypoxic Pulmonary Vasconstriction

Hyperventilation, Polycythemia (increase RBC’s), Acclimatization, etc.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
40
Q

A non-specific reaction of the immune

system to a foreign invader is called

———————————————

A

INFLAMMATION

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
41
Q

Typically a response of the vascular

tissue to a pathogen or harmful stimulus

(eg. vasodilation after laceration)

called ———————————-

A

INFLAMMATION

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
42
Q

A protective response of the immune

system to eliminate pathogen/stimulus

and initiate the healing process

called————————-

A

INFLAMMATION

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
43
Q

Inflammation the same as INFECTION

True or False

A

FALSE

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
44
Q

Acute Inflammation Period is —————-

and it is called

A

Rapid onset, Short-course;

0-72 hours

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
45
Q

SUBACUTE Inflammation – stage in between acute &

chronic phase; 72hr—————————–

A

72 hrs -3 months

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
46
Q

CHRONIC Inflammatiomn is called – —————————————

———————————————————————-

A

CHRONIC Inflammatiomn is called – gradual onset, long-lasting,

recurring; > 3 months (greater than months)

47
Q

ACUTE INFLAMMATION

The initial, rapid natural response of the body

towards a————, —————, or ——————.

A

ACUTE INFLAMMATION

The initial, rapid natural response of the body

towards a _pathogen, toxin, or irritan_t.

48
Q

Occurs in the first 48-72 hours of the body’s

interaction with pathogen (usually noticeable

within first few minutes or hours).

A

Acute inflammation

49
Q

Infiltration of plasma cells and white blood

cells from the blood towards the pathogensite and injured tissues —–> leads————————————————-

A

To an Acute Inflammatory Response (Acute-Phase Response)

50
Q

In Acute-Phase Response
Tissues contain cells known as———————–and ————————(derived from ———-)

A

MACROPHAGES and MAST CELLS (derived from WBC’s in the blood)

51
Q

At the onset of an infection, pathogen or injury, these

cells become ACTIVATED and release———————————–

A

INFLAMMATORY MEDIATORS (Cytokines,

Prostaglandins, Histamine, Bradykinin, etc).

52
Q

The mediators also cause—————————–of the blood vessels ——-> leakage of plasma cells and fluid into tissue ———> ———————————

In acute————

A

INCREASED PERMEABILITY

SWELLING (Edema)

In acute-Phase Response

53
Q

Some of the mediators ————————will also

INCREASE PAIN SENSITIVITY—————————

A

(Bradykinin)

(Hyperalgesia​)

54
Q

The mediators will also

cause movement (——————-) of WBC’s

(mainly ——————-) to the site of injury to

eliminate the——————————–

A

INFILTRATION

Neutrophils

pathogen (via Phagocytosis)

55
Q

This response requires constant stimulation -

inflammatory mediators——————.

Therefore, as soon as the stimulus is

removed, ————————————-.

A

degrade quickly

acute inflammation stops

56
Q

Acute Inflammation Prcoess in Action

A

Acute Inflammation Prcoess in Action

57
Q

Acute inflammation Diagram

A

Acute inflammation Diagram

58
Q

5 Cardinal Signs/Symptoms of Acute Inflammation

SIGNS (Objective):

HEAT (Calor)

SYMPTOMS (Subjective):

PAIN (Dolor)

A

SIGNS (Objective):

  • REDNESS (Rubor)
  • HEAT (Calor)
  • SWELLING/EDEMA (Tumor)
  • IMMOBILITY/LOSS OF FUNCTIONa
  • *SYMPTOMS (Subjective):**
  • PAIN (Dolor)
59
Q

True or False

The signs of acute inflammation(Pain, Heat, Redness, Swelling and loss of function) may only occur together when inflammation is on the surface (skin).

A

True

The signs of acute inflammation(Pain, Heat, Redness, Swelling and loss of function) may only occur together when inflammation is on the surface (skin).

60
Q

True or False

Acute inflammation of an internal organ may result in all of these signs/symptoms.

(Pain, Heat, Redness, Swelling and loss of function)

A

False

Acute inflammation of an internal organ may not result in all of these signs/symptoms.

(Pain, Heat, Redness, Swelling and loss of function)

61
Q

Persistent, prolonged inflammation, typically

lasting longer than 3 months duration,

It is called ————————————

A

Chronic Inflammation

62
Q

It is Simultaneous destruction and healing of the

tissue from the inflammatory response , which refered to ——————————

A

It is Simultaneous destruction and healing of the

tissue from the inflammatory response , which refered to Chronic Inflammation

63
Q

Increase in the proportion of Inflammatory Mediators which promote the inflammatory response compared to those which eliminate the pathogen occured in the ———————

A

Increase in the proportion of Inflammatory Mediators which promote the inflammatory response compared to those which eliminate the pathogen occured in the Chronic Inflammation

64
Q

Chronic Inflammation can lead to the following :

  • Tissue —————
  • —————- (Cell Death – eg. muscle loss in aging)
  • —————- (Scar Formation)
  • —————- ( Atherosclerosis, Rheumatoid Arthritis, Hayfever, some forms of Cancer, Inflammatory Diseases, Autoimmune Disorders)
A

Chronic inflammation lead to

  • Tissue Damage
  • Necrosis (Cell Death – eg. muscle loss in aging)
  • Fibrosis (Scar Formation)
  • Disease (. Atherosclerosis, Rheumatoid Arthritis, Hayfever, some forms of Cancer, Inflammatory Diseases, Autoimmune Disorders)
65
Q

Chronic Cardiovascular Diseases Inflammation ​

please breifely define the following :

Atherosclerosis ?

Cerebrovascular dis?

Cardiomyopathy ?

Stroke?

A

Cardiovascular Diseases :

Chronic Inflammation ​

Atherosclerosis is buildup of fats, cholesterol and other substances in and on your artery walls (plaque), which can restrict blood flow.

Cerebrovascular diseaase is a group of conditions, diseases, and disorders that affect the blood vessels and blood supply to the brain. If a blockage, malformation, or hemorrhage prevents the brain cells from getting enough oxygen, brain damage can result.

Heart failure is known as congestive heart failure, occurs when your heartmuscle doesn’t pump blood as well as it should.

Cardiomyopathy is a disease of the heart muscle that makes it harder for your heart to pump blood to the rest of your body. Cardiomyopathy can lead to heart failure.

Stroke is occurs when the blood supply to part of your brain is interrupted or reduced, preventing brain tissue from getting oxygen and nutrients. Brain cells begin to die in minutes.

66
Q

Causative Agent for acute Inflammation is ——————————

A

Pathogens; Injury

67
Q

What is the casuing agent for Chronic Inflammation ?

A

Persistent, non-degradeable pathogens; Autoimmune Reactions

68
Q

The Onset for Acute Inflamamtion is—————————————–

A

Immediate: minutes to hours

69
Q

The Onset for Chronic Inflamamtion is ——————————-

A

Delayed: weeks to month

70
Q

The Duration for Acute Inflammation is ——————

VS

The Duration for Chronic Inflammation is——————

A

The Duration for Acute Inflammation is Few days

The Duration for Chronic Inflammation is Months . years

71
Q

What are the Inflammatory Mediators for Acute Inflammation ?

A

Neutrophils, Macrophages, Mast cells

72
Q

What are the Inflammatory Mediators for Chronic Inflammation ?

A

Cytokines, Fibroblasts

73
Q

Inflammation can release extra ———–from the inflammatory site which circulates in the

—————

A

protein

bloodstream

74
Q

What are the three blood tests that can detect increased levels of protein, and thus, act as markers for monitoring inflammation ?

A

ESR (Erythrocyte Sedimentation Rate)

CRP (C-Reactive Protein)

PV (Plasma Viscosity)

75
Q

An Invasion and multiplication of pathogens in a bodily tissue is called —————?

A

Infection

76
Q

What are the S/S of systemic infection ?

A

Fatigue

Weight loss

Fever

Chills/Night Sweats

Generalized Pain/Body Aches

77
Q

Diagnostic Tests for Infection are the folloowing

Culture/swab (from moist areas: throat, cervix, etc)
—————————
————————————————————–

Biopsy

A

Urinanalysis
Stool sample
Spinal tap (lumbar puncture - CSF)

CT

78
Q

Bacterial Infection is consiedered to be ——————-?

A

LOCAL!!!

79
Q

What are the Signs/Symptoms of Local Infection ?

A

Redness (local)
Heat
Swelling/Edema
Pain (local)
Cured by Antibiotics
Can also lead to systemic s/s

80
Q

Local Infection can be cured by ——————-, can lead to ——————-S/S.

A

Cured by Antibiotics
Can also lead to systemic S/S

Eg. Swollen tonsil, pain on one,

pain on one side of throat

81
Q

Viral Infectiion is called —————————-

A

Systemic Infection

82
Q

The following Signs and Symptoms of

rarely painful or local (eg. herpes)

Itchy, Burning

Can also cause local s/s

are indication of ————————————- ?

A

Viral Infection

83
Q

NOT cured by Antibiotics—————–

A

Viral Infection

Eg. Generalized throat pain, chest pain, runny nose,

cough, body aches, sinus congestion, etc.

84
Q

Can lead to tissue injury and progress to disease

and NOT the same as inflammation!

is called ————————-

A

INFECTION

85
Q

Infection is categorized by ——————type or location

—————-, —————, ———————

e.g Skin, Eye, Vaginal, etc.

A

pathogen

Bacterial, Viral, Fungal

86
Q

Same s/s by different agents:

eg. Sore throat –

—————– (Streptococcus Pyogenes),

—————— (Adenovirus),

——————(Candida Albicans) —> ——————–

A

Bacterial

Viral

Fungal (Candida Albicans) —> Pharyngitis

87
Q

Under Infection Susceptibility,

What are the 4 factors that can decrease a host’s resistance to infection?

A

Route of Entry of Pathogen (Transmission)

Virulence of Pathogen (toxins, destructive enzymes, etc)

Quantity/Load of Initial Inoculant

Decreased Immune function of host (eg. Immuno-compromised)

88
Q

Under Infection Transmission

Transmitted ————— to person in same generation

It can be————or indirect

A

Person

Direct

89
Q

Under Infection Transmission

——————— to ————(trans-placental transmission)

A

Mother to Child

e.g. AIDS, hepatitis, cytomegalovirus, herpes, rubella

90
Q

Inhalation of aerosols (sneezing, coughing), Sexual transmission (oral, vaginal, anal)

What kind of Tansmission ? Is it direct or indirect ?

A

Horizontal Transmission

Direct

91
Q

Direct Horizontal Tramsission can be

——————or ———————-

A

Touching or Inhalation

92
Q

Indirect Horizontal Transmission pathogen can withstand harsh environment over time and remain infective. True or False

A

True

93
Q

Contact with contaminated inanimate objects

(furniture, door knobs, kleenex, food products)

A

Indirect Horizontal Transmission

94
Q

Fecal-oral route (sewage water, under-developed, food poisoning)

Eg. rotavirus, E.coli, Giardia, cholera

Is part of the ——————————————————-?

A

Indirect Horizontal Transmission

95
Q

Infections that usually do not cause disease in a healthy host

(ie. healthy immune system)

True / False

A

True

96
Q

Immunosuppression caused by:

——–,

skin damage,

malnutrition,

——————–,

—————-fections,

immunosuppressant agents (organtransplants),

——————————-

Eg. Pneumocystis carinii, Candida Albicans

A

HIV,

chemotherapy,

recurrent infections,

pregnancy

Eg. Pneumocystis carinii, Candida Albicans

97
Q

Time between exposure to pathogen/irritant

and moment when Signs/Symptoms first appear

is called the ————————————-

A

INCUBATION PERIOD

98
Q

Target Immuno-compromised/immunodeficient

is an OPPORTUNISTIC INFECTIONS

True or False

A

True

99
Q

These are Infections that leads to Clinical Onset

May be a carrier of a disease without exhibiting

May or may not be Infectious/Contagious at this time

s/s yet (eg. Chickenpox)

It is known as the ————————–

A

INCUBATION PERIOD

100
Q

Time between Infection (exposure) and Infectiousness (contagion)

that leads typically shorter than incubation period

(sometimes samelength, sometimes longer)

It is called ——————————————

A

Latency Period

101
Q

The pathogen is called Cellulitis

the Incubation Period is ——————

A

Cellulitis < 1 day

102
Q

The pathogens name are the following :

Influenza

SARS

COVID 19

the Incubation Period for each is ——————

A

Influenza 1-3 days

SARS Up to 10 days

COVID 19 1-14 days (median 5-6)

103
Q

The pathogens name are the following :

Polio

Chickenpox

the Incubation Period for each is ——————

A

Polio 7-14 days

Chickenpox 14-16 days

104
Q

The pathogens Mononucleosis 28-42 days, its Incubation Period for each is ——————

A

Mononucleosis 28-42 days

105
Q

The pathogen name is Kuru (prion disease),

Its incubation period is —————-

A

Kuru (prion disease) ——10-13 years

<strong>Prion diseases are a group of neurodegenerative disorders that can affect both humans and animals. </strong>

<strong>They’re caused by the the deposition of abnormally folded proteins in the brain, </strong><strong>which can cause changes in memory, behavior and movement</strong>

106
Q

Time between exposure to pathogen (Infection) and

the earliest time when thepathogen/causative agent can be recovered

from the patient (usually for diagnostic purposes)

is called ——————-

A

Prepatent Period

107
Q

Usually SHORTER than Incubation Period

Ie. Possible to diagnose/detect a pathogen before it manifests into s/s

It is called ———————-

Ex……………………..

A

Prepatent Period

Eg. In a parasitic Infection, time when eggs or larvae

can be recovered from feces, urine, or blood

108
Q

The dose or amount of a pathogen required to create an Infection in the body

is called—————————-

A

Inoculation Dose Period

109
Q

Used primarily for vaccinations when inoculating the body with antigenic

species of a virus in order to build the body’s immunity and create antibodies

(Immunization )

A

Inoculation Dose Period

110
Q

An infection that is Asymptomatic, Occurs during the Incubation Period of an Infection

is called ——————————–

Its Classification before the Infection manifests into s/s

A

Subclinical Infection

111
Q

Can only be identified by culture studies, It is one of the greatest risk of spreading infection at this time (silent killer!)

It is known as the —————————-

Eg. AIDS,

genital warts,

chlamydia,

TB

A

Subclinical Infection

112
Q

Time when s/s have ceased but Infection is still present, the tiime of recovery and healing (feeling back to“normal”)

A

Convalescent Period

113
Q

Later stage of Infection which may still be infectious/contagious is know as ———————

A

Convalescent Period

Pathology 101 Seneca College (2 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions