Diagnostic Imaging of GI Flashcards

1
Q

On survey radiography, what should the gastric and intestinal lumen be seen to contain? Colon?

A

Gastric/SI: gas and fluid

Colon: faeces

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2
Q

What can be seen more clearly using contrast radiography?

A

Masses/mucosa/gastric filling

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3
Q

List 5 methods of contrast radiography of the GI tract. When would each be indicated?

A
  • pnumogastrogram (FB in stomach)
  • Barium meal (assess gastric emptying)
  • Double contrast gastrogram (gas+barium, to identify mucosal lesions and rugae of stomach)
  • Upper GI contrast study - barium series (ID stomach and intestine, GI motility, obstruction)
  • Barium enema (ID colon [only with ascites*?!], intussesception, mural lesions)
    > with use of US and CT these are rarely used any more
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4
Q

In what dimension should the stomach lie in a normal dog?

A

Perpendicular to spine

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5
Q

What contrast medium is usually used for GI studies? Why? What else may be used?

A

Barium sulphate - ^ atomic number, bland (-> therapeutic)
> I- solution may be used, has quick intestinal transit time but may be useful when rupture of GI tract suspected as will leak easily. Doesnt coat mucosa like barium sulphate.

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6
Q

How is “localised” or “focal” dilation defined? What is most likely to cause this?

A

1.6x L5 diameter or 2x rib diameter BUT not a hard and fast rule - take all clinical signs into account

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7
Q

What may confused for a SI dilation?

A

Dilatued uterus due to pregnancy - will only contain foetuses from d44. Follow caudally at level of colon and if it enters the pelvic inlet then is probably uterus!

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8
Q

Is lodgement of foreign material usually the cause or result of an obstruction?

A

Can be EITHER!

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9
Q

What types of foreign material may be visable radiographically?

A
  • metallic
  • mineralised eg. stones
  • rubber/plastic
  • non-opaque eg. friut seeds, corn cobs (may not stand out)
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10
Q

What is generalised dilation usually the result of?

A

Intestinal hypomotility eg. acute viral enteritis (parvo) electrolyte imbalance, drugs

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11
Q

Will FBs always be visable on radiograph?

A

NO! -> ultrasound

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12
Q

What may be seen radiographically with a partial obstruction?

A

Gravel sign - gut contents become compacted, fluid absorbed -> mineral opacity with a “corner” just before obstruction

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13
Q

What may cause an abnormal luminal shape on radiograph?

A
  • disordered motility eg. linear foreign body

- filling defect eg. intussecpetion, FB

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14
Q

What is the stacking of intestines often seen with linear FBs referred to as?

A

Plication

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15
Q

How can barium series be used to confirm suspicions of GI pathology?

A

If deformity is present at all time periods/views then most likely real rather than an artifact of the image

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16
Q

How can abnormal mucosal surface be identified? What does this suggest?

A

Contrast radiography only (on radiography feaces may stick to gut wall and appear as a pathology etc.)
> suggests infiltration or ulceration

17
Q

How would an intussusception be visible on contrast radiography?

A

Long filling defect with contrast around the edges

18
Q

What disease may affect the appearance of the duodenum secondarily?

A

Pancreatitis

19
Q

Other than a pathology, what may impact gastric emptying time?

A

Stress (sympathetic innervation)

20
Q

What time period is accepted as convincing evidence of delayed gastric emptying?

A

Food in stomach 24hours after ingestion

21
Q

What is the average normal gastric emptying time for barium sulphate suspension in the dog and cat?

A

Dog 1-3hrs, 1.5hrs in pups
Cat 0.25-2hrs
very variable!!

22
Q

What is the average normal gastric emptying time for barium sulphate meal in the dog and cat?

A

Dog 4-6hrs, 3.5-7.5 in pups
Cat 4-17hrs
very variable!!

23
Q

What 3 main areas can ultrasonography assist in the assessment of?

A

GI wall
Motility
Associated organs (pancreas and LNs)

24
Q

What is the normal thickness of the GI wall? How many layers are present?

A

normal thickness 3mm (<5mm in canine duodenum and stomach)

5 layers visable

25
Q

How many peristaltic waves/min are normal?

A

4-5 (hypomotility often -> dilation)

26
Q

What are the 5 layers of intestinal wall visable on ultrasound?

A

Lumen, mucosa, submucosa, muscularis, serosa

27
Q

Which layers of the intestinal wall are hyper echoic and which are hypo?

A
  • Lumen hypER
  • Mucosa hypO
  • Submucosa hypER
  • Muscularis hypER
  • Serosa mega-hypER [NB: U/S exaggerates thickness of serosa so measure from lumenal side to check thickness of gut wall]
28
Q

How may hyper-motility be seen on US?

A

Corrugation of GI tract

29
Q

What landmark is assocaited with visualising the pancreas on ultrasound?

A

Pancreaticoduodenal vein (anechoic line)

30
Q

What LN pathology is easily diagnosed by ultrasound? Can LNs be detected by any other means?

A

Lymphadenomegaly

LNs can only be visualised with ultrasound

31
Q

Is visualisation of the pancreas by US sensitive?

A

NO - changes in pancreas may not correlate to current pathology, and lack of differences on ultrasound cannot rule out pathology -> use in conjuction with other clinical signs