Diagnosis of OLBP - clinical reasoning framework Flashcards

1
Q

central pain mechanism - neurobiological processes underpinning

A
  1. neuromodulatory issue
  2. maladaptive psychology
  3. social issues
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2
Q

triage - fracture risk

A
trauma
sudden
sharp, deep pain
usually high severity
multidirectional provocation
highly painful on palpation
OP risk
BMI low
age > 50, more so if >70
non traumatic fracture before
metabolic disease
long term steroid use
acute spinal deformity
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3
Q

Infection risk

A
open wound
post surgery or had invasive operation
indwelling catheter/indwelling device used
red, swollen, hot locally
immunosuppressed
IV drug use
fever
night sweats
rigors
unwell feeling
UTI or other recent infection
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4
Q

Cancer risk

A
constant unremitting pain
non mechanical
insidious onset
no relief with rest
unexplained weight loss
associated symptoms i.e. coughing blood up
night pain
family hx of cancer
pt hx of cancer
age > 50 with first episode of LBP
risky activities i.e. smoking
generally feeling unwell
treatment unsuccessful
failure to ease pain
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5
Q

Neurological risk

A
bilateral distribution
bladder dysfunction
bowel dysfunction
glove & sock anaesthesia
saddle aneasthesia
S2 distribution
S2 toe flexion test +ve
unsteadiness
clumsiness
hyperreflexia
spasticity
widespread signs (over a few levels)
severe weakness (even if only 1 level)
progressive signs (even if only 1 level)
dermatomal rash
diabetic
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6
Q

Inflammatory risk

A
slow insiduous onset
persistant episodic pain
morning worst time 
morning stiffness lasts >1/2 hour
wakes 2nd half of night
eases with activity
worse with rest
multi jnt involvement
hot/red/swollen jnts
family hx
past hx
eye redness
psoarisis
dry eyes/mouth
stomach problems related to jnt pain
enthesopathies (tendon pain)
alternating SIJ pain
NSAIDS help +++
hot bath/shower helps +++
dactylitis ( swollen finger )
persistent limitation all directions
hip/shoulder/neck stiffness in AM
fatigue/fever/malaise
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7
Q

Non spinal pathology

A

Renal
gynaecological
other organ
psychiatric
Vascular i.e. AAA: - PVD/CAD
- HTN
- >60
- smoker
- pulsatile mass
- abdominal pain
- syncope (drop in HR/BP giving fainting)
- Bruit (blood gushing sound)

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8
Q

Peripherally dominant signs

A
well defined area
anatomical pattern makes sense
mechanical behaviour
appropriate level of irritability
turns off and on
clear stimulus response pattern
progression in keeping with physiology
standard history
predictable and consistent response to physical loading
loading response makes biomechanical sense
reasonably stable
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9
Q

Centrally dominant signs

A
widespread pain
not within usual anatomical boundaries
extensive spreading of pain
pain shifts around
sustained high levels of pain
spontaneous pain
non noxious triggers
non standard clinical course
other sensitivites (noise, smell, chemical etc)
distorted stimulus response relationship
non restorative sleep
extreme fatigue
concentration problems
unpredicatable and varied response to treatment
widespread hyperalgesia
widespread allodynia
pain affected by emotions
illness, anxiety and depression related to pain
negative perceptions and expectations
unhelpful behaviour (withdrawal, excessive rest)
contradictions in behaviour
inconsistencies in behaviour
extreme behaviours
waddells symptoms
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10
Q

Waddells symptoms

A
coccyx pain
whole leg pain
whole leg numbness
whole leg giving way
never free from pain
intolerance/reaction to many treatments
hospital admission with NSLBP
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11
Q

Peripheral neurogenic pain - nerve trunk pain

A
deep, aching pain
pulling pain
dragging
heaviness
along line of nerve 
tension point of pain
linking of pains form line of enrve
mild non-dermatomal paraesthesia
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12
Q

Peripheral neurogenic pain - dysaesthetic pain

A
sharp/shooting/stabbing/lancinating
burning/gripping
paroxysmal pain
pain felt in sensory distribution of pain
paraesthesia
anaesthesia
weakness
hyperalgesia
allodynia
latent pain
after pain
feel awful, wearing
autonomic changes
peripheral tenderness
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13
Q

Peripheral somatic pain

A

back pain usually main problem
deep, aching pain
back pain can be well localised
back pain can be sharp or catching

referred pain qualities:
deep, aching
dull but may become severe
static in location
expand slowly if stimulus increases
aware of centre but hard to localise boundaries
less often below knee
spreads slowly
non tender in referred area
after pain uncommon
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14
Q

Mobility dysfunction

A
older age
less episodic
less variation in symptom intensity
slower to resolve when aggravated
not increasing in frequency
less intense
less likely to be immobilised or stuck
associated with stiffness
morning pain
pain with position change
relief with stretching
less when warmed up
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15
Q

loading dysfunction

A
less episodic
slower development
mid range pain
pain with static loading
may be more fearful and anxious
relief when distracted
relief with relaxation
describe tightness
describe tiredness
muscular distribution to some pains
burning LBP (ischaemic) quality
maladaptive stability beliefs
worse with stability training
rigid and cautious to move
anticipate pain
breath holding
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16
Q

control dysfunction

A
younger age
more commonly episodic
tends to be more intense
quicker resolution
aggravated by minimal perturbations
agg by sudden or unguarded movment
described as being immobilised or stuck or locked
feeling of giving way
describe back going into spasm
catch or painful arc
frequent manipulation with short term benefit only
self manipulator
post trauma
post pregnancy
relief with brace or corset