Diabetic foot Flashcards
5 Presentations of diabetic foot?
Ulcer
Cellulitis, necrotising fasciitis
Gangrene
Infection
Charcot joint disease
Wagner classification?
0 - no ulcer
1 - superficial ulcer involving only skin
2- superficial ulcer involving muscle/ligs/bone
3 - deep ulcer involving abscess/tendinitis/OM
4 - Forefoot or toe gangrene
5 - Midfoot or hindfoot gangrene
What is Charcot arthropathy
Chronic progressive degeneration of a weight bearing joint causing deformity of bone architecture and joint alignment.
Causes of Charcot’s joint
Diabetes
Neurological -
Rheumatological
Toxins - alcoholism
Infection - leprosy, poliomyelitis, syphilis, tabes dorsalis
How to differentiate Charcot’s arthropathy with cellulitis and OM?
In Charcot’s, redness typically resolves with elevation
Brodsky’s classification for Charcot Joint?
1 = Tarsometatarsal joint Midfoot
2 = Subtalar joint hindfoot
3a = Ankle joint
3b = Calcaneal tuberosity fracture
4 = Any combination
5 = Forefoot
Lifetime risk for DFU btw ___ and ___%.
High recurrence ___% within 1yr, ___ at 3 years, ___% 5 years after healing.
Lifetime risk for DFU btw 19 and 34%.
High recurrence 40% within 1yr, 60% at 3 yrs, 65% 5 yrs after healing.
Diabetics ___ ~ ___ times higher risk of amputation.
___% 5 yr mortality post LL amputation.
Diabetics 15 ~ 31x risk of LL amputation
50% 5yr mortality post LL amputation.
3 zones in debridement of diabetic foot?
Red zone of actual injury/ulcer
Orange zone of inflammation
Green zone of clean tissue
Why must turgish foot with normal looking skin be checked?
Epidermis of plantar skin in sole of foot seals up v fast. Hence pathology can hide below it.
OM changes show up after _____ weeks
4-6 weeks
What to remember XR for all diabetic foot or foot in general?
must do WEIGHT BEARING XR!
____ of pts with current foot ulcer develop SSI. Of these, ____% go into OM.
50% of pts with current foot ulcer develop soft tissue infection. 20% of these go onto OM.
Implication of low toe pressure in pharmacological treatment?
Abx administered vascularly cannot reach target tissue in foot. Hence must have good collaterals.
Some characteristics of Diabetic foot
- May not have early symptoms
- Somatic/autonomic neuropathy
- May not have typical features of PAD
- Immune paresis = downregulation of host defences with signs of broad inflamm