diabetes

Question 1

type 1

Answer 1

no insulin

Question 2

type 2

Answer 2

decreased insulin production.
tissue may be resistant to insulin or both.

Question 3

when energy needs aren’t meet increased hungry, body starvation

Answer 3

polyphagia

Question 4

kidneys are unable to retain excessive glucose, resulting in??

Answer 4

glycosuria

Question 5

HgbA1C

Answer 5

glucose binds w/ hemoglobin. higher % of glycosylated
2-3month period

Question 6

somogyi effect

Answer 6

hypoglycemia around 2am w/ rebound hyperglycemia as body tries to compensate may require decreases in PM insulin.

Question 7

ketoacidosis DKA

> 300

Answer 7

ketones formed as by product of fat breakdown.
ketones are acidic, leading to metabolic acidosis.

Question 8

DKA

Answer 8

kidneys try to compensate for acidosis by buffering.
as cells die without energy potassium is released into the bloodstream. then excreted by kidneys.

Question 9

DKA symptoms

Answer 9

ketosis
metabolic acidosis
Kussmaul resp
ketone breath (fruity)
nausea abd pain
dehydration

Question 10

when does potassium replacement begin?

Answer 10

voiding at least 25ml/hr

Question 11

HHNS

Answer 11

severe dehydration due to polyuria. more gradual progression, later recognition due to partial insulin/glucose uptake by tissues.

Question 12

HHNS symptoms

Answer 12

altered CNS
severe dehydration.
glucose >600
negative ketone urine
ph >7.4
^ serum potassium

Question 13

Normal Saline for

Answer 13

shock or severe hypotension: 1/2 normal saline otherwise

Question 14

hypoglycemia symptoms

Answer 14

nervousness
pallor
tremors
palpitations
sweating
hunger
weakness
dizzy
HA
drowsiness
irritability
loss of coordination
seizures
coma