DEVELOPMENTAL DYSPLASIA OF THE HIP Flashcards

1
Q

DDH

A

abnormal development of the hip resulting in dysplasia and possible displacement/subluxation of the coxofemoral joint due to capsular laxity and some mechanical issues.

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2
Q

Development abnormality

A
bony structures 
(proximal femur and acetabulum), 
labrum,
joint capsule and 
soft tissue
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3
Q

Crucial – an early diagnosis

A

Ultrasonography
Screening
Minimal treatment techniques to lower morbidity and fast recovery

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4
Q

ANATOMY

A

Normal development of the hip – depends on :

  1. Normal growth of the triradiate cartilage
  2. Bone ossification nuclei of the acetabulum (ilion, ischion and pubis)
  3. A spherical femoral head and congruency with the acetabulum (centered)
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5
Q

At birth– cartilaginous structures:

A

acetabulum
entire femoral head
femoral neck - partially
greater trochanter

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6
Q

FISIOPATHOLOGY

A

Presure ↑↑ on the cartilaginous FH ► ↓ blood perfusion► necrosis of the chondrocytes
Affects FH and growth cartilage
Doesn’t affect the greater trochanter (normal dimensions)

Muscular imbalance :
Deteriorates growth and morphology of the proximal femur
Contracture of the adductors
Improper function (weakening) of the abductors

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7
Q

ETIOLOGY

A

Multifactorial

  1. Laxity of the ligaments and capsules
  2. Race – caucasians – a smaller acetabulum
  3. In utero position (20%)
  4. Oligohydramnios
  5. Sex (80% - females)

Genetic susceptibility
History - x 10 incidence in children with DDH parents
- twins - 34% (monozygotic)

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8
Q

EPIDEMIOLOGY

A

Incidence
Most frequent pediatric orthopedic issue in newborns
dysplasia - 1:60-100
luxation (displacement) - 1:1000

Barlow – instabilitaty 1 out of 60 nb.
60% get stable during the first week and stabile 80% up to the age of 2 months (0,2% remain with residual hip dysplasia)

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9
Q

EPIDEMIOLOGY

localization and demographics

A

Localization

  • Most frequent in the left hip of girls
  • Bilateral in 20%

Demographics
Frequent in Native Indians, Laplanders, Balcans (Romania – unknown incidence)
Rare in africans

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10
Q

PROGNOSIS OF DDH

A

Diagnosed early and properly – very good prognosis

Treatament by closed reduction - prognostic bun
Failure of conservative treatment – open reduction – uncertain prognosis, at least good for a short term

According to R. Graf - DDH diagnosis during the first 14 days of life and a proper treatment (ultrasound) – healing up to 3 months of life

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11
Q

Ortolani (Le Damany,1912) sign

Out of 3 cases only one develops DDH

A

Hip abduction (45-60°), pushing the greater trochanter→ FH centers

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12
Q

Barlow’s sign

A

Adduction and pressure on the knee and inner thigh→ FH exits the acetabulum

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13
Q

CLINICAL EXAM

other signs

A
  1. Galeazzi
  2. Limited abduction
  3. Piston sign
  4. Lumbar Hyperlordosis
  5. Gluteal/inguinal fold assimetry
  6. Trendelenburg sign and walking (insufficiency of the pelvitrochanteric muscles)
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14
Q

IMAGISTICS

A

Hip ultrasound–Graf (most used), Harcke

Recommended – SCREENING (and clinical exam)

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15
Q

CLASSIFICATION AFTER GRAF
CENTERED HIP TYPE I
NORMAL

A

Type I: • α angle ≥ 60º, β < 77º
• stable hip
(Ortolani and Barlow negative)

High risk hip -> surveillance

Type I: • α ≥60º
• familial history positive

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16
Q

CENTERED HIP TYPE II – ß < 77º

A

Type II a : • acetabular dysplasia (0 – 3 months)
• α = 50º - 59º
medical surveillance

Type II b : • acetabular dysplasia (3 – 12 months)
• α = 50º - 59º
Pavlik harness

Type II c : • acetabular dysplasia (0 – 12 months)
• α = 43º - 49º
palster casting in human position

17
Q

DECENTERED HIPS TYPE D, III & IV

A

Type D • α = 43º - 49º
• ß > 77º
• eversed labrum

    Type III a and b  • α < 43º

    Type IV   • α < 43º
		   • inversed labrum

adductor tenotomy

open reduction Ludloff /Smith-Petersen and
plaster casting

18
Q

Other imagistic exams

A
  1. Xrays
  2. Artrography
  3. MRI
  4. CT/CT-3D – not useful
19
Q

NATURAL EVOLUTION OF DDH

A
Untreated cases of DDH may
↓
Instability
↓
Degenerative changes 
present in teenagers
↓
Severe degenerative changes
↓
Osteoarthritis
(early hip surgery)
20
Q

TREATAMENT OF DDH

A

Conservative (according to R. Graf)
Phase
1.Reduction -Decentered hips(D,III,IV) - Manually,by extension,Pavlik
2.Retention - Unstable hips ( IIc unstable) - Human position,plaster according to f..
3.Maturation - Stable ‘‘dysplastic’’ hips ( IIc stable,IIb,IIa-) - Harness according M.Graf/Pavlik

21
Q

Frejka pillow

A

a pillow splint used for abduction and flexion of the femurs in treatment of congenital hip dysplasia or dislocation in infants.

22
Q

Pavlik harness

A

a support device used to immobilize a body part or hold it in position.
Pavlik harness a device used correct hip dislocations in infants with developmental dysplasia of the hip, consisting of a set of straps that hold the hips in flexion and abduction.

23
Q

Surgery

A
  1. Adductor release (tenotomy)
  2. Open reduction
  3. Derotation osteotomies
  4. Acetabuloplasties
  5. Arthroplasties
24
Q

Open reduction and pelvic osteotomies

A

Salter, Pemberton, Dega, Hopf, Steel, Ganz, Chiari, Dial, Shelf, etc.)

25
Q

CONCLUSIONS

A

Early diagnosis, early treatment

US Screening + clinics
Healing up to 3 months of age

Untreated – LLD (limb length discrepancies), limping, hip pain and early osteoarthritis

Even if treated properly hip arthritis is not excluded (especially for late treatment)