Derm lect 4 Flashcards

1
Q

Acute dermatitis (Eczema) is what type of hypersensitivity reaction

A

Type 1: IgE mediated

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2
Q
  • “the itch that rashes”
  • characterized by pruritus and disruption of skin surface
  • begins early in life with chronic exacerbations and remissions
A

atopic dermatitis

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3
Q

what is the atopid triad

A
  • atopic dermatitis
  • allergic rhinitis (hay fever)
  • asthma
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4
Q

clinical presentation

  • ranges from ill-defined, erythematous, scaling pathces to edematous papules and vesicles
  • flexural surfaces, hand/foot in children and adults
A

dermatitis (eczema)

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5
Q

a form of eczema that is coin-shaped

A

nummular eczema

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6
Q

clinical presentation:

  • cheeks, scalp and extensor surfaces common in infants
  • clue: sparing of diaper area
A

atopic dermatitis

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7
Q

what are some common complications from dermatitis (ezcema)

A
  • excoriation
  • lichenification
  • fissures
  • secondary cellulitis
  • eczema herpeticum- disseminated viral infection (HSV 1 or 2)
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8
Q

treatment for eczema

A
  • avoid exposure to triggers (allergens, wool clothing); rubbing or scratching; excessive bathing; lotions
  • use Emollients (vaseline)
  • during exacerbation can briefly use topical steroid
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9
Q

this secondary skin condition is the result of excessive scratching or rubbing.

Clinical presentation:

  • exaggerated skin markings, dry, leathery appearance, pigmentation
  • common areas: back of neck, wrists, forearms, lower legs, genitals
A

lichen simplex chronicus

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10
Q

clinical presentation:

  • deep seated vesicles that coalesce and rupture
  • common areas: hands (80%), sides of fingers, palms and soles
  • intensely pruritic
  • episodes are usually weeks to months apart, spontaneous remission after 2-3 weeks
A

Dyshidrotic eczema

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11
Q

what precipitates dyshidrotic eczema

A
  • emotional stress
  • hot weather
  • found in those with nickel allergy
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12
Q

treatment of dyshidrotic eczema

A
  • wet dressing
  • topical steroid
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13
Q

clinical presentation

  • rough, raised papules (flesh, red, or brownish)
  • forms horny plugs in hair follicles
  • common area: outer upper arms, thighs, cheeks, upper back
  • usually worse in winter months
  • improves with age
A

keratosis pilaris: disorder of keratinization

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14
Q

What is allergic contact dermatitis? What are some examples of things that cause it?

A
  • delayed-type hypersensitivity reaction
  • poison ivy; nickel; latex
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15
Q

this type of rash is caused by repeated friction and mechanical irritation. Ex: household duties: hands in water, detergents, solvents

A

irritant contact dermatitis

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16
Q

treatment for contact dermatitis

A
  • discontinue exposure to allergen or irritant
  • wear gloves or protective clothing
  • apply emollient
  • apply topical steroid twice daily for several weeks prn
17
Q

clinical presentation

  • greasy scales and yellow-red coalescing macules, patches, and papules affecting scalp, face (eyebrows, eyelids), ears, presternal skin and upper back
A

Seborrheic dermatitis

18
Q

what is the cause of seborrheic dermatitis

A

overproduction of sebum in associated with yeast: malessezia furfur (formly known as pityrosporum ovale)

19
Q

clinical presentation:

  • yellow, greasy adherent scale on vertex of scalp
  • may affect diaper area and axillary skin
  • seen in infants
A

cradle cap: form of seborrheic dermatitis

20
Q

treatment of seborrheic dermatitis and cradle cap

A
  • cradle cap: olive oil to loosen crusts
  • selenium sulfide or anti-dandruff shampoo
  • mild topical steroids
21
Q

clinical presentation

  • possible prodrome: malaise, pharyngitis
  • exanthematous eruption with a primary plaque “Herald patch” usually on the trunk
  • followed by secondary rash 1-2 weeks later: fine scaled, pink, oval lesions and plaques that are arranged in a christmas tree pattern
  • may be very itchy, but in most cases no pruritus
A

pityriasis rosea

22
Q

cause of pityriasis rosea

A

benign, viral skin eruption of unknown etiology

23
Q

treatment for pityriasis rosea

A
  • self-limiting rash
  • oral antihistamines (e.g. claritin)
  • medium strength topical steroid
  • sun exposure helps
24
Q

clinical presentation

  • four “P’s”- purple, pruritic, polygonal, papules
  • middle ages adults
  • common areas: wrists, back, shins, scalp, penis and mouth (50%)
  • wickham’s striae = white lines
A

Lichen Planus

25
Q

treatment for lichen planus

A

topical/oral steriods, cyclosporine

26
Q

etiology of psoriasis: what age population is commonly affected? what conditions predispose a person to this condition?

A
  • 15-30 and then 50-60 yrs
  • HLA gene, infections (strep), medicinal drugs, stress or injury
27
Q

clinical presentation

  • thickened red plaques with silvery scale
  • can have associated nail pitting, onycholysis, and arthritis
A

psoriasis

28
Q

removal of scale results in punctate bleeding

A

auspitz sign

29
Q

plaques develop in areas of skin injury (rubbing, scratching, scrapes)

A

koebner phenomenon

30
Q

clinical presentation

  • red plaques with well-defined edges covered with thick, silvery scale
  • extensor surfaces (elbows, knees, back are most commonly affected)
    • auspitz sign and koebner phenomenon present
A

Psoriasis vulgaris

  • most common form
31
Q

this variant of psoriasis has lesions are small drop-like plaques

A

guttate psoriasis

32
Q

this variant of psoriasis is commonly found on hands and feet

A

palmar-planter psoriasis

33
Q

treatment for psoriasis

A
  • no one regimen will work for everyone
  • general measures: sunshine, baths, emoliants, occlusive dressing

localized (<5% TBA)

  1. topical corticosteriods, vit D analogs, Tazarotene

Severe (>5% TBA): refer to dermatologist

  • do not use oral steroids