Dental Development/Morphology/Occlusion Flashcards

1
Q
  1. An issue in HISTODIFFERENTIATION during tooth development would cause what type of anomaly?
  2. At what morphologic stages would HISTODIFFERENTIATION occur?
  3. What are some examples of anomalies due to issues during this stage?
A
  1. create anomalies in the structure of dentin and enamel
  2. Cap and Bell stages (both early and advanced)
  3. AI type 1 (hypoplastic); AI type 4 (hypoplastic and hypomaturation); DI
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2
Q
  1. An issue in PROLIFERATION during tooth development would cause what type of anomaly?
  2. At what morphologic stages would proliferation occur?
  3. What are some examples of anomalies due to issues during this stage?
A
  1. number of teeth and structure of teeth
  2. in all 3 (bud, cap, and both bell stages)
  3. hypodontia; hyperdontia, oligodontia, epithelial rests
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3
Q
  1. An issue in MORPHODIFFERENTIATION during tooth development would cause what type of anomaly?
  2. At what morphologic stages would MORPHODIFFERENTIATION occur?
  3. What are some examples of anomalies due to issues during this stage?
A
  1. size and shape of teeth
  2. in all 3 (bud, cap, and both bell stages)
  3. microdontia, pg lateral, mulberry molars, hutchinson incisors, absence of cusp/root; macrodontia, tuberculated cusps, cusp of Carabelli, taurodontism, dens in dente, dens evaginatus, dilaceration, gemination, fusion, concrescence
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4
Q

If a patient has AI type 2, at what point in the histophysiologic stage did an issue arise?
AI type 1?
AI type 4?
AI type 3?

A

AI type 2–> during apposition and during mineralization
AI type 4–> during apposition, during mineralization, and during histodifferentiation
AI type 1–>during histodifferentiation
AI type 3–> during maturation

AI type 1 is hypoplastic
AI type 2 is hypomaturation
AI type 3 is hypocalcification
AI type 4 is both hypoplastic and hypomaturation

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5
Q

What syndromes are associated with hyperdontia?

A
  • Down Syndrome
  • Sturge Weber Syndrome
  • Cleidocranial dysplasia
  • Crouzon Syndrome
  • Apert syndrome
  • CLP
  • Orofaciodigital syndrome type 1
  • Gardner syndrome
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6
Q

What syndromes are associated with missing teeth?

A
  • ectodermal dysplasia
  • Crouzon syndrome
  • Achondroplasia
  • Ellis-van Creveld (chondroeectodermal dysplasia)
  • Williams syndrome
  • incontinentia pigmenti
  • orofaciodigital type 1
  • Rieger syndrome
  • non-syndromic CLP
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7
Q

What syndromes are associated with microdontia?

A
  • Ectodermal dysplasia
  • Crouzon syndrome
  • Down syndrome
  • Hemifacial microsomia
  • Ellis-van Creveld (chondroectodermal dysplasia)
  • pituitary dwarfism
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8
Q

What syndromes are associated with macrodontia?

A
  • hemifacial hyperplasia/hypertrophy
  • Crouzon syndrome
  • Otodental syndrome
  • XYY syndrome
  • pituitary gigantism
  • pineal hyperplasia with hyperinsulinism
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9
Q

What is the most common type of hyperdontia? In which sex? In which area of the mouth?

A

MC in males in max anterior; with MC type being MESIODENS

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10
Q

Which teeth are most commonly missing? And which sex is it more common in?

A

More common for females to have missing teeth.

3rd molars>mand 2nd pre>max laterals>max 2nd pre

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11
Q

Which teeth are most commonly associated with microdontia?

A

max laterals, 2nd premolars, 3rd molars

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12
Q

what is dens invaginatus vs dens evaginatus?

A

dens invaginatus is Dens in dente – tooth within a tooth

dens evaginatus is talon cusps

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13
Q

Which teeth are most commonly associated with dens in dente and how does that occur?

A

MC dens invaginatus: max laterals
–IEE invageinates

tx: seal or composite resto
if deepr lesion, may need IPC/DPC, RCT

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14
Q

Which teeth are most commonly associated with dens evaginatus and how does that occur?

A

talon cusps are mostly associated with maxillary incisors>lateral incisors; 77% are permanent teeth

occurs with the evagination of the IEE

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15
Q

Characteristics of AI type 1

A
  • hypoplastic type
  • insufficient quantity of enamel with lack of complete IEE
  • BOTH dentitions affected
  • MC type vs types 2,3,4
  • 44% have long lower face causing anterior open bite
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16
Q

Prevalence of AI vs DI

A

AI is found in 1:14k

DI is found in 1:8k

17
Q

Which type of DI occurs with OI?

A

Dentinogenesis imperfecta Type 1 occurs with OI types Ib and IVb

18
Q

What are the different types of DI and which is the most severe? which is the least?

A

3 different types (Shields type 1,2,3)
Type 1 is most mild and type 3 is most severe

DI type 1:

  • occurs with OI type 1b and 4b
  • is a dental mani of an underlying collagen defect
  • primary teeth more severely affected and in permanent teeth only centrals and 1st molars affected
  • EXPECT TO SEE:
    1. amber translucence
    2. bulbous crowns
    3. short roots
    4. PARLs
    5. alveolar abscesses
    6. rapid attrition
    7. pulpal obliteration

DI type 2:

  • “hereditary opalescent dentin”
  • OCCURS ALONE–no assc with OI
  • AD type
  • SAME characteristics as type 1
  • irregular/tubular pattern

DI type 3:

  • MOST severe
  • EXPECT TO SEE:
    1. bell-shaped crowns
    2. opalescent hue
    3. shell teeth with short roots and enlarged pulp chambers
    4. ONLY mantle dentin formed
    5. permanent tooth pulps small or completely obliterated
    6. rapid wear of crowns
    7. enamel pitting
    8. REGULAR tubules
    9. multiple pulp exposures
19
Q

What conditions/syndromes are associated with DI?

A

OI, Ehlers-Danlos, Goldblatt syndrome, and Schimke immune-osseous dysplasia

20
Q

Characteristics of OI?

A

OI has 4 major types with type 1 being MC and type 2 being lethal in prenatal period.

DI is more common in type 3 and 4 but DI type 1 occurs with OI type 1b and 4b.

Expect to see:

  1. bowing of legs
  2. blue sclera
  3. fragile bones (fractures)
  4. bitemporal bossing
  5. loose ligaments due to defective collagen
  6. impaired hearing
  7. macrocephaly