Dental Anomalies Part II Flashcards

1
Q

what is amelogenesis imperfecta

A
  • inherited genetic mutation
  • isolate or syndromicc
  • enamel is affected in all or nearly all teeth
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2
Q

what dentition is affected in amelogenesis imperfecta

A

primary and permanent

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3
Q

what is type 1 alemogenesis imperfecta

A

hypoplastic type: thinner but hard enamel

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4
Q

what is type 2 amelogenesis imperfecta

A

hypomaturation type: relatively hard but colored (not translucent)

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5
Q

what is type III amelogenesis imperfecta

A

hypomineralized or hypocalcified: softer rough colored undermineralized enamel

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6
Q

what is type IV amelogenesis imperfecta

A

hypoplastic- hypomature with taurodontism

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7
Q

describe the type 1 hypoplastic type amelogenesis imperfecta

A
  • thin bur mineralized enamel. looks radiopaque
  • quantitative enamel defect: failure during enamel matrix secretory stage (enamel agenesia is rare)
  • generalized or localized, pits, striae, groove defects
  • diffuse smooth or rough
  • teeth appear white to yellow-brown, small, and square shaped
  • open contacts “picket fence” appearance. flat occlusal surface, low cusps
  • delayed eruption and increased impaction
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8
Q

describe type II amelogenesis imperfecta

A
  • normal thickness of enamel. a qualitative defect
  • soft enamel - as radiodense as dentin
  • less translucid enamel: cloudy white, yellow or brown color
  • hard and protective
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9
Q

describe type III amelogenesis imperfecta

A
  • enamel of normal thickness but soft, porous and shedding easily from the dentin. normal shape at eruption
  • these teeth are very sensitive even to physical contact with a toothbrush
  • soft enamel that is less dense than dentin in radiographs
  • grossly worn teeth to gingival level
  • the color of enamel can range from white to creamy yellow-brown
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10
Q

describe dentinogenesis imperfecta

A
  • primary dentition more severely affected
  • amberlike translucency (yellow to blue-gray)
  • enamel separates easily from dentin (faulty DEJ)
  • accelerated attrition -> anterior open bite
  • cervical constriction -> bulbous crown
  • short and slender roots
  • partial or complete obliteration of pulp chambers and canals
  • types I, II and III
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11
Q

describe type I dentinogenesis imperfecta

A
  • associated with osteogenesis imperfecta
  • mutation in collagen synthesis genes
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12
Q

describe type II dentinogenesis imperfecta

A
  • no skeletal defects
  • enlarged pulp chambers in primary teeth (occasional)d
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13
Q

describe type III dentinogenesis imperfecta

A
  • brandywine isolate
  • found in people originating from southern maryland
  • enlarged pulp chambers
  • pulp exposure
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14
Q

what are the acquired abnormalities

A
  • attrition
  • abrasion
  • erosion
  • third molar impaction
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15
Q

what is attrition

A

physiologic wear of teeth

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16
Q

what is abrasion

A

mechanical wear of teeth

17
Q

what is erosion

A

chemical wear of teeth

18
Q

what are the ways third molars can be impacted

A

full impaction and partial impaction

19
Q

what are the orientations for third molar impactions

A
  • vertical
  • inverted
  • mesioangular
  • distoangular
  • horizontal
  • inverted mesioangular
  • inverted distoangular