Dental Anomalies II Flashcards

1
Q

How can eruption be disturbed?

A

Can be premature or delayed?

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2
Q

When are more likely to see premature eruptions of teeth?

A

High birth weight

Hormonal abnormalities - excess GH/ thyroid

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3
Q

Whats a neonatal tooth?

A

Teeth erupt in first 30 days due to superficial or ectopic position tooth germ

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4
Q

Issue w/ neonatal teeth?

A

No root is formed - mobile - risk of inhalating and will interfere w/ feeding

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5
Q

Management of neonatal teeth?

A

XLA due risks

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6
Q

When is it common to see delayed eruption?

A

Low birth weight
Pre-term syndromes
Endocrinopathies impaction

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7
Q

What are reasons for premature exfoliation?

A

Trauma or extraction
Immune - cyclic neutropenia
Cementum deficiencies

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8
Q

What are reasons for delayed eruption?

A

Infraocclusion
Ectopic successors
Hypodonita

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9
Q

What defects can see in structure of tooth?

A

All tissue - arrested development
Enamel defects
Dentine defects
Cementum defects

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10
Q

Example of enamel defects?

A

Hypomineralisation
Hypoplasia
Discolouration

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11
Q

What is hypomin?

A

Poor quality of enamel - porous leading to opacities

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12
Q

What is hypoplasia?

A

Poor quantitiy of enamel = pits, grooves

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13
Q

What are some systemic effects that can cause enamel defects?

A

Maternal and foetal conditions e.g rubella
Premature/ low birthwieght
F- - excessive dose
Severe/chronic child hood ilness

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14
Q

What child hood illness could cause enamel defect?

A

Feveres
Measles
Chicken pox
Use of tetracylinices

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15
Q

What systemic features can give localised enamel defects?

A

Infection/ trauma

Cleft lip/palate

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16
Q

Example localised enamel defect?

A

MIH

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17
Q

Why get opacities in hypo mineralisation?

A

Disruption to mineralisation at maturation stage causing porosity

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18
Q

How does hypomin appear?

A

White/cream/yellow/brown opaque patch - have altered translucency due to altered texture

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19
Q

Is hypomin localised or generalised?

A

Can be both

20
Q

How to differentiate between hypo min and fluoride induced opacities?

A

F- induced

  • Brown
  • See in primary and permannet teeth
  • Dose dependent appearance
  • Symmetrical distribution
21
Q

What defect is hypoplasia?

A

Quantitative - deficienct matrix production = less enamel

22
Q

How does hypoplasia appear?

A

Pits, grooves

Chronologically ‘matching’ teeth affected w/ systemic upset

23
Q

What can localised hypoplasia be referred to?

A

Turner tooth

24
Q

What is turner tooth?

A

Hypoplasia due trauma/ infection or primary tooth = damage underlying permanent tooth germ

25
What teeth are commonly turner teeth?
4s/5s
26
When can see change in colour of teeth?
Incorporating circulating substance - metabolic disorders (bilirubin)/ tetracycline Incoportation pulp products - loss vitality Exogenous agents - dietar/ bacterial
27
What is AI?
Hereditary condition causing enamel defect
28
What conditions are likely to have increased associated w/ AI?
AOB Hearing impairment Renal calcification Cone-rod dystrophy
29
How does AI present?
Vairable - diverse phenotpyes | Can be hypomin or hypoplastic
30
Classic appearance of AI?
Soft enamel wears away Bilateral and symmetrical - not always Snowcapped teeth
31
Different classifications of AI?
Hypoplastic Hypomature Hypocalcified
32
What dentition does AI affect?1
Both | Secondary more affected
33
What is type of gene mutation seen in AI only seen females?
Lyonisation
34
What is lyonisation?
Random inactivation one of X chromosomes
35
What see in lyonisation AI?
Bands of normal and abnormal enamel - alternate ameloblast formation
36
What are defects of dentine?
DI Radicular dentine dysplasia Fibrous dysplasia
37
What can dentine defects be associated w/?
Osteogenesis imperfecta Elhers-danlos Rcikets Hypophosphatasia
38
Most common dentine defect?
Dentinogenesis imperfecta
39
What gene responsible for DI?
DSPP - defect in non-collagenous dentine matrix protein
40
How does DI present?
Opalesent teeth w/ brown/ blue colour Bullbous crowns w/ short/thin roots Pulp chamber oblieration
41
What dentition does DI affect?
Both - primary is more severe
42
How is dentine abnormal DI?
Irregular and reduced tubules | Cellular inclusions from pulp = gradual obliteration
43
What is type I DI associated w/
Osteogenesis imperfecta | Mutation collagen type I genes
44
Clinical features of osteogenesis imperfecta?
Bone fragile/ deform Lax joins Blue scerla
45
What is dentine dysplasia?
Root dentine affects = normal crowns morphology and dentine but short blunt roots
46
What see in dentine dysplasia?
Mobile teeth