Deadly Arrhythmias Flashcards
What signs and symptoms are helpful in evaluating stability in a pt with dysrhythmia?
Hypotension Hypoperfusion Altered mental status Ischemic chest pain Respiratory distress Heart rate > 300
What life-threatening rhythm can develop from tachycardias with rates approaching 300 and why?
Faster rate leads to closer T and R waves that can produce R on T phenomenon that can lead to V-fib
80% of bradyarrhythmias are caused by what?
Factors external to the cardiac conduction system including: ACS, hypoxia, tox, hypoperfusion, electrolyte disturbance
What are the most common bradyarrhythmias with ACS?
Sinus bradycardia and AV block
What are the drugs used for general bradycardia management and their doses?
Atropine: 0.5–1mg, q3–5min, max 3mg (kids 0.02mg/kg)
Glucagon: 2–10mg IV bolus
Adrenergic agents can be used but can worsen ischemia and are only a bridge to more definitive therapy. These include: dopamine, epinephrine, isoproterenol
What is often the definitive management of bradyarrhythmias?
Pacing
What site is preferred for placement of trans-venous pacer and why?
Right IJ to leave the left side for a permanent pacer
Sinus bradycardia is a common presentation of what ACS region of cardiac ischemia?
Inferior wall MI
Is atropine indicated when treating bradycardia secondary to MI?
Yes, and should be considered early in patients with symptomatic bradycardia from MI
What bradyarrhythmias respond best to atropine?
Sinus bradycardia responds best, but AV blockade can also be treated with atropine
What are common causes of a junctional rhythm?
ACS, Tox, or normal in trained athletes
What is the first intervention for symptomatic junction bradycardia while attempting to find and treat the underlying cause?
Atropine
More likely to work if rhythm is from the AV node or from ACS
Less likely to work if lower than the AV node (Bundle of His) or from tox cause
What is the management for unstable idioventricular rhythms?
Atropine is less likely to work and pacing are more likely to be required
Additionally, O2, IVF, and sufficient perfusion are key in these patients
When should 1st degree heart block and 2nd degree type 1 heart block be taken a little more seriously?
Symptomatic, or:
Presence of ACS, metabolic issues, on cardiac meds that may be causing the blocks, and new onset in these scenarios as they may transition to complete heart block
Which heart blocks should always be considered pathologic?
Second degree type II and third degree