D2 Flashcards

1
Q

Differences in pediatric skin

A
  • Children’s Epidermal Layer Less Bound To Dermal layer
  • Poor Adherence Results In Separation Of layers
  • Much Higher Risk Of Separation In Preterm infants
  • Increased Exposure To Iatrogenic Risk Factors
  • Increased exposure to body fluids
  • Limited/no ability to self care or self report
  • Higher risk of accidental injury
  • Higher Risk Of Inflammatory Conditions
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2
Q

factors that influence skin healing

promotes? Delays?

A

Promotes
- moist clean environment
- good nutrition

Delaying Healing:
- Immunocompromised
- Impaired Circulation
- Stress
- infection,antiseptics, medication
- foreign bodies, mechanical friction
- co-existing diseases/ morbidities

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3
Q

Skin condition assesment

A
  • Location/distribution
  • Extent
  • Colour
  • Type
  • Pruritus?
  • Pain/tenderness/ other Bleeding?
  • Swelling
  • Infected?

more
- Age of onset
- Allergies
- Exposure to irritants
- History in the family
- Asthma/Hay fever

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4
Q

Erythema, ecchymosis, petichea, primary and seconday lesions

A

Erythema
- A reddened area caused by increased amounts of oxygenated blood in the dermal vasculature

Ecchymoses (bruises)
- Localised red or purple discolorations caused by extravasation of blood into dermis and subcutaneous tissues

Petechiae
- Pinpoint, tiny, and sharp circumscribed spots in the superficial layers of the epidermis

Primary lesions
- Skin changes produced by a causative factor;
- primary lesions in paediatric: macules, papules, vesicles, patches, bullae, plaque, wheals, nodules, pustules, and cysts

Secondary lesions
- Changes that result from alteration in the primary lesions, such as those caused by rubbing, scratching, medication, or involution and healing

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5
Q

Managent of Iacotengic risks: Reduce and promote

A

Reduce:
- Pressure, especially over bony prominences
- Friction And Shear
- Epidermal stripping
- Contact with irritants (urine and faeces,ostomy fluids,IVfluids)

Promote:
- Optimal Oxygenation
- Hydration And Nutrition
- Circulation
- Movement

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6
Q

managment of ostomies

A
  • tracheostomy, gastrostomy, ileostomy, nephrostomy, urostomy, jejunostomy, colostomy

Care priorities
- Maintain position and patency of tubes * - Protect surrounding skin
- Prevent/reduce skin exposure to fluids
- Keep as clean and dry as possible
- Use barriers – creams, protective and/or absorbent wound products, ostomy wafers

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7
Q

diaper dermatitis

what? Diaper wetness produces?

A

prolonged and repetitive contact with irritant (e.g., urine, faeces, soaps, detergents, ointments, friction)

diaper wetness produces:
- higher friction
- greater abrasion damage
- Increase In pH from the breakdown of urea in the presence of faecal urease
- Possibly from detergents or soaps from inadequately rinsed cloth diapers or the chemicals in disposable wipes

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8
Q

Candida albicans infection and interventions

A

Candida albicans infection
- perineal inflammation and a maculopapular rash with satellite lesions that may cross the inguinal fold

Nursing Interventions
- Reduce contact with irritant
- Changing diapers often
- Don’t Use Perfumed Products/wipes
- Keep skin dry
- Use barrier creams– Re-apply each change (Zinc oxide – remove with mineral oil)
- Minimise Friction
- Avoid Frequent Washing/firm rubbing
- Monitor For Secondary Infection And Treat Accordingly

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9
Q

AD eczema

daignosis and managment

A

Descriptive diagnosis
- Intense pruritus- inflammatory chronic skin disease
- Associated with asthma and allergies (family hx)
- dermatological manifestations appear subsequent to scratching, lesions disappear if scratching is stopped
- Improvement in humid environments

interventions

Managment
- Bathe in tepid water with mild or no soap
- Apply hydrating moisturisers
- Apply topical medications as required
- Dress in warm moist dressings/clothes, with dry outer layer
- Cool wet compresses/dressings Evaluate for secondary infections
- Address Itch, scratching, pain, stress, fatigue, sleep disturbance

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10
Q

Burns

what? contributing factors to severity?

A

Young children at high risk of hot fluid burn injuries
- Scalds = 50% of all burn injuries
- Children under 5 highest risk
- Severity, therapy, prognosis related to amount of tissue destroyed: total body surface and depth
- Infant skin thin and more easily burned

Contributing factors:
- Location
- Causative agent
- Age of child
- Respiratory involvement
- General health

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11
Q

burns classification and common rule

A
  • Depth of injury (used to be classified as “degree”)
  • Superficial (first degree)
  • Partial-thickness (second degree)
  • Full-thickness (third degree)
  • Full-thickness + underlying tissue (fourth degree)

Commonly used rule
- child’s palm, including fingers = 1% of body surface area.
- For children, a burn of 10% of TBSA can be life threatening if not treated appropriately.

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12
Q

patho thermal injury

A
  • A major burn (greater than 30% TBSA)
  • edema and severe capillary damage
  • an increase in capillary permeability, allowing plasma, proteins, fluids, and electrolytes to be lost
  • Anaemia –> caused by direct heat destruction of red blood cells(RBCs), injured RBCs, and trapping of RBCs in the microvascular thrombi of damaged cells
  • increased blood flow
  • Increase in metabolism to maintain body heat,providing for the body’s increased energy needs.
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13
Q

managment of burns

A
  • Stop Burning Process (run cool water over burn; wet clothes, only remove clothes if they come off easily – otherwise, soak the area)
  • Keep the child warm – do not cool large burns
  • Assess Child’s Condition
  • ABC: Always Give Oxygen For Moderate–severe burns
  • Cover burn to Prevent Contamination
  • Transport Child To Appropriate Level Of Care
  • Pain management
  • Fluid Replacement Therapy: critical in first 24 hours
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14
Q

BURNS correctionon of fluis electrolyte balance

A
  • High Risk Of Hypovolemia And Sodium Losses
    Maintain Tissue Perfusion:
  • Normal Saline or Ringer’s lactate
  • Depending on Total Body Surface Area (TBSA) (>15-20%) IV infusion immediately to maintain urine output at ~1-2 mL/kg (under 30 kg) or ~ 30-50 mL/hr (>30 kg).
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15
Q

managment of burns

complications, minor burns, and ideal wound dressing

A

Reduce Complications:
- Airway Compromise
- Infection (local,pneumonia,systemic)
- Contractures
- Scarring

Care of Minor Burns
- Wound cleansing
- Debridement – with or without removal of blisters
- promote healing + puritis mngmt

Ideal burn dressing:
- Reduce risk of infection (broad spectrum antimicrobial)
- Require infrequent changing with minimal discomfort
- Promote re-epithelialization
- Cover wounds with antimicrobial ointment OR use occlusive dressings OR both

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16
Q

manament of major burns + biological skin coverings

A

Care of Major Burns
- Primary Excision
- Debridement
- hydrotherapy
- Topical Antimicrobial Medications

Biological Skin Coverings
- Allograft (human cadaver skin)
- Xenograft (porcine skin)
- Synthetic Skin Coverings
- Split-thickness skin grafts (sheet or mesh graft)

17
Q

Acute Gastroenteritis

A
  • Caused by a viral, bacterial or parasitic infection
  • Vomiting and diarrhea may lead to dehydration
  • May have fever and intermittent crampy abdominal pain
  • Sick contact and/or travel.
  • Common culprits are rota- virus or norovirus
18
Q

Patho acute gastroenteritis

A

Pathophysiology
- Excess fluid in the small intestine means H20, Na, K, HCO3 are drawn from extracellular fluid into the stool causing dehydration, electrolyte depletion and metabolic acidosis.
- Irritation of the mucosa by toxins cause increase in excretion of water and electrolytes
- Destruction of the mucosal lining=decreased surface thus decreased absorption
- Inflammation = decreased absorption
- Increased motility = decreased absorption

19
Q

vomiting infants red flag

A

Vomiting-Infants Red flag
- Vomiting is not normal for young babies- often indicative of a more serious problem
- Spitting-up?
- Important to determine whether the infant is spitting-up or is vomiting?
- Surgical concern
- Vomiting in infants is usually associated with a surgical emergency
- Dehydration very important to asses

20
Q

Biliary atresia

A

Biliary Atresia
- Rare newborn condition; is serious
- Can be congenital or from environmental factors (inflammation)
- Bile cannot get from the liver to the stool, causing acholic (pale in colour) stool.
- This blockage causes buildup of bile in the liver, which can lead to liver failure and eventually death.
- Treatment is a surgical intervention to allow bile to move from the liver to the intestines.
- Now a part of newborn screening- parents are the screeners
- Newborns will often have jaundice and hepatomegaly

21
Q

Appendicitis

s/s, managment, complications

A
  • 1st symptom – peri-umbilical pain
  • As inflammation spreads – pain migrates to RLQ
  • Anorexia, nausea, vomiting, diarrhea, low grade ever, rebound tenderness, guarding

Managment
- IV/blood work – bolus
- Analgesic
- NG tube if peritonitis or obstruction – insert using xylocaine
- Antibiotics (Amp/Tobra/Flagyl)
- NPO
- Surgical consult

Complications
- Peritonitis (from appendix rupture)
- Rupture causes sudden relief of pain
- S&S: fever, abdominal rigidity/distention
Ischemic bowel, sepsis, shock can result
- Rupture predisposes to complications post-op- adhesions and obstruction
- IV fluids and antibiotics stat