cheo final Flashcards

1
Q

Airway assesment
patent vs obstructed

A

Patent
- active, alert
- Air entry heard equally, bilaterally
- good colour
- Normal, easy respirations

Obstructed
- panic to unresponsive
- Floppy to rigid muscle tone
- Silent Chest
- no air entry heard
- no adventitious sounds
- No chest movement
- pale to cyanotic
- No respiratory effort

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

partially obstructed airway

A

Partially
- Normal to agitated/drowsy
- pink, pale, cyanotic
- Tripod/sniffing positioning

Upper Airway
- Stridor (Inspiratory) on exertion (mild obstruction)
- at rest + respiratory distress (moderate obstruction)
- + exhaustion (severe obstruction)
- Drooling; gurgling

Lower Airway
- Wheezes (musical, high pitched)
- End expiratory(mild)
- Expiratory(moderate)
- Inspiratory and expiratory (severe)

Decreased air entry
Increased respiratory effort

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

breathing assessement

A

Effort
- rate and depth
- chest symmetry
- adventitious breath sounds (wheeze,
stridor, crackles)
- cough
- retractions, tracheal tug, nasal flaring
accessory muscle use/head bobbing
grunting
- positioning → tripod
- gasping

efficacy
How effective is gas exchange?
- LOC, behaviour, agitation to exhaustion
- muscle tone

Colour
- mucous membrane
- cyanosis, or history of?

hypoxia (low O2) or hypercapoena (high CO2)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

circulation assessement

A

Circulation
- Assessments includes inspection and hands on palpation
- Perfusion
- Heart rate & rhythm

rate
- Cardiac rhythms are usually regular in children
- common arrhythmias are subtle and require auscultation for at least 1 minute
are heart sounds S1,S2 normal?

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

perfusion

A

Central
- LOC, agitation/irritable
- Muscle tone
- Colour (pale;mottled)
- Urine out put
- Pulses; Apical (auscultate), Brachial,femoral,carotid(palpate)

Peripheral
- Capillary refill <3sec
- CWSM–compare bilaterally
- Pulses(palpate): Radial,dorsalis pedis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

disability

A

Pediatric Neuroassessment
- LOC AVPU
- Modified GCS
- to determine severity of injury/insult identify trends over time, subtle changes are most important indicators,
- Posture, muscle strength, tone
- Behaviour, activity, response to stimulation, agitation, weak cry
- Pupil size and reaction

A. Alert
V. responds to Voice
P. responds to Pain
U. Unresponsive

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

childrens differences with fluids

A

anatomical & physiological differences
- Ratio of body surface area to body mass
- Higher Metabolic Rate – H20, O2, glucose
- Renal tubular immaturity –> Concentrated urine

weak Immune system

Dependence on caregivers

Different composition of total body water
- Higher extracellular water: intracellular water
- This means we can lose it faster

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

intake and output

A

Intake and output
- Normal fluid requirements based on size, smaller have higher needs
- Dependency, breastfeeding
- Serum electrolytes

Output
- > 0.5-3 cc/kg/hr
- 1ml/kg/hr minimum for infants and small children or > 4 wet nappies/24hrs
- Stools, emesis, blood loss
- Frequency, type, amount, consistency, colour, odour

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

conditions to increase vs decrease fluids

A

Increase fluid
- Child with respiratory problems need to increase fluid (loosens)
- Fever

Decrease
- Respiratory overload
- congenital heart issues
- Renal impairment
- Head injuries (cerebral edema)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

glucose

A

Hypoglycemia (is more common)
- Trembling or ‘jitteriness’
- Pounding heart
- Cold sweating
- Pallor

Neurological signs/symptoms
- Difficulty concentrating/confusion
- Blurred/doublevision
- Difficulty hearing
- Slurred speech
- Dizziness and unsteady gait
- Loss of consciousness/seizure

Hyperglycemia Signs
- Increased urine output
- Excessive thirst
- Weight loss

** Note: If any alterations in LOC -always check BG!***

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

who pain ladder

A

Step 1 mild, pain score 1-3
- Tylenol
- NSAID (Ketorolac, Ibuprofen, Celebrex)

Step 2 moderate, pain score 4-6
- Tylenol and NSAID
- Opioid (i.e. Tramadol, Morphine/ Dilaudid at the low end of range)

Step 3 severe, pain score >7
Tylenol and NSAID
- Opioid (i.e. Morphine / Hydromorphone Dilaudid® / Fentanyl) orals at higher end of range or infusion
- May require supplemental Ketamine or Lidocaine
- PCA, NCA or Epidural
- Gabapentinoids / Antidepressants

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

what is asthma? S/S

A

What is asthma
- Hypersensitive lungs (obstructive)
- Chronic inflamation of the airway
- Excess in mucus production
- Tightening of the airway muscles (around the bronchioles → broncocontrsitcion)

What does asthma look like on the outside
- Wheezing → too narrow space so it forcefully tries to pass through → noisie
- Flared nostrils
- Pale skin
- Rapid “belly breathing”
- Tracheal tug (skin pulling on neck and chest)
- Accessory muscle use
- Rapid shallow breathing

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

PRAM scores

A

P. pediatric
R. respiratory
A. assessment
M. measure

mild 0-3
moderate 4-7
severe 8-12

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

asthma control medications

A

Controller medications (inhaled corticosteroid → reducing inflammation)

RED PUFFERS
- Flovent ® (fluticasone)
- Alvesco ® (cicloconide)
- Pulmicort ® (budesonide)
- Qvar ® (beclomethasone)
- Asthmanex ® (mometasone)

Oral vs inhaled corticosteroid
- Inhaled steroids are localised
- Inhaled must be used everyday
- Oral only for emergency

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

asthma reliver medications

A

Reliever medications BLUE PUFFERS
- Ventolin® (salbutamol)
- Aeromir® (salbutamol)
- Atrovent® (ipratropium)
- Bricanyl® (terbutaline)

How its used
- Noticing symptoms of an asthma trigger
- Last in our body for 4 hours
- 2 puffs for it to be effects as needed → PRN

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

asthma combination medications

A

Combination medications
- Controller + reliever (long acting→ lasts abt 12 hours)
- Still want to brush teeth and stuff to avoid thrush

Types
- Advair® (fluticasone and salmeterol)
- Symbicort® (budesonide and formoterol )
- Zenhale ® (mometasone + formoterol ) - zenhale is also blue so check if its ventolin first

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

nursing care resp infections

A

Nursing Care: Plan & Management
- goal: Decrease respiratory effort, prevent exhaustion and/or Increase efficacy

Interventions:
- Frequent assessment, close observation depending on presentation position
- clear airway (suction) offer O2
- humidified high flow NP
- Bronchodilators (if bronchiospams)
- Corticosteroids? Antibiotics? Epi?
- Promote rest and comfort– limit disruptions → limit what is taking their energy
- Promote hydration and nutrition (anemia → hemoglobin and O2)
- Manage temperature → hyperthermic OR hypothermic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

resp diagnostics

A

Blood Gases
- Arterial/Capillary/Venous
- PaO2, pH, PaCO2,HCO3

Pulse Oximetry

XRay

PFT’s
- Expiratory flow rate
- Vital capacity

CT Scan/MRI Scopes

C&S

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Croup patho and s/s

A

Pathophysiology
- Mucosal inflammation/edema in the : Larynx, trachea, epiglottis
- Airway obstruction, hypoxia

Symptoms
- Respiratory distress mild-severe
- Sudden onset of harsh, barky cough
- inspiratory stridor → sounds like they are barking
- Accessory muscle use
- Retractions
- cyanosis
- Worse at night!!

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

acute LTB croup characteristics

A

Acute Laryngotracheobronchitis: croup
- Most common croup syndrome
- Mainly affects children < 5 years
- Mainly viral
- Usually follows an URTI

Characterized by:
- Gradual onset of low grade fever
- Barky brassy seal-like cough worse at night
- Inspiratory stridor
- Hoarseness

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

nursing plan for croup

A
  • Goal: Decrease respiratory effort, prevent exhaustion and/ or Increase efficacy

Interventions:
- Frequent assessment, close observation
- Environment calm &quiet; avoid unnecessary impingements (airway can be obstructed by stress response)
- Emergency airway/intubation equipment on hand
- administer humidified O2 prn
- Promote hydration (po/iv) and nutrition
- Prevent spread of infection, isolate,PPE

Administer medications:
- dexamethasone,
- antipyretics,
- epinephrine inhalations
- antibiotics if epiglottis type

Concern is agitation and low sats

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

acute epiglottitis

A

Acute Epiglottitis
- organism: H.influenzae type b
- Most Serious – high risk of airway obstruction
- Most common age 2-8 years

Clinical manifestations
- Usually abrupt onset
- retractions, fever, Inspiratory stridor
- Frog-like croaking sound on inspiration
- 4 D’s - drooling, dysphonia, dysphagia, distress (agitation)
- Tripod position

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

croup score

A

0-2 mild
3-7 moderate
8-11 severe
12-17 impending respiratory failure

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

foreign body aspiration

A

Can aspirate anything and Symptoms depend on location of obstruction
- Initial: coughing, gagging, wheezing, dyspnea
- Treat with abdominal thrusts and back blows if full obstructions occurs
- Laryngotracheal: stridor, hoarseness, drooling → sign they cant swallow, coughing
- Diagnosed by history and xray, removed by endoscopy
- Can lead to aspiration pneumonia if not removed quickly

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
influenza
Types A and B - Spread by droplet/contact - Severe in infants Symptoms: - dry throat, dry cough, photophobia, myalgia, lack of energy, fever/chills - Can lead to viral pneumonia, encephalitis, secondary bacterial infections - Lasts 4-5 days - In mild cases supportive management at home - no asprin (reye syndrome) In severe cases: - Tamiflu (oseltamivir) 48hrs Prevention: - flu shot!
26
pertussis
Caused by: - Bordetella pertussis - Primarily occurs in children under 4 and not immunized - High morbidity and high risk of mortality - PREVENTION is the key - Immunization - Life-long immunity after infection nsg care: - droplet precaution (nasal swab) Management: - Supportive care - erthyromicin
27
pneumonia
Pneumonia - inflammation of the lungs --> alveoli fill w fluid Etiology In children,usually: - Viral (RSV, influenzas) - Bacterial - Aspiration Pneumonia: Nursing Care Plan - Goal; Decrease respiratory effort, prevent exhaustion and/or Increas eefficacy Interventions: - chest xray - Frequent assessment, close observation depending on presentation - Position, clear airway (suction), offer O2 - Humidified highflow NP - Antibiotics - Promote hydration and nutrition
28
CF
Autosomal recessive trait - CF gene inherited from both parents (both parents must be carriers) – - Increased Viscosity of Mucous Gland Secretion - Results in mechanical obstruction - Thick mucus accumulates, dilates, precipitates, coagulates and blocks glands, ducts, passageways - 90% have a blocked pancreatic duct - Malnutrition - Respiratory tract and pancreas are predominantly affected
29
Patho to dehydration
1. decreased fluid intale/increased fluid loss 2. rapid ecf loss (children have less ecf) 3. electrolyete imbalances 4. Icf loss 5. cellular dehydrations 6. hypovolemic shock 7. death key ecf losses - diarrhea and vomitting - blood loss/ wound drainage - insensible losses
30
types of dehydration
isotonic - Na loss = water loss - ecf losses - V+D --> insensibble losses --> decreased PO intake - most common type for children (ECF is high in sodium) hyotonic loss - Na loss > water - ECF to IC space --> increases ECF loss - edema - V+D, burns, renal issues hypertonic - Na loss
31
hypovolemic shock | s/s
CO = HR x SV Compensation: - ↓ Stroke Volume (SV) related to ECF losses causes ↑ - HR to maintain Cardiac Output (CO) - ↓ BP a late sign of shock - Hypotension is followed by rapid decompensation Signs of Shock: - ↑HR - Altered LOC - Poor perfusion
32
dehydration tx for mild moderate and severe
Mild (<5%) - rehydrate with ORS (50ml/kg) over 4 hours - replace ongoing with ORS moderate (5-10%) - rehydrate with ORS (100 ml/kg) over 4 hours - replace ongoing with ORS Severe (> 10%) - IV recussitation with NS or LR (2-40ml/kg) for 1 hour - reasses and repeat if neccessary - begin ORT when pt is stable - replace ongoing loss with ORS
33
Cardio assessement
- Color - Nutritional status/Feeding/weight gain or loss (Infants) - imput/ output - Respiratory – rate and ease of breathing - Sweating?!!! (A sign of cardiac issues) - Presence of oedema - Size/shape of chest - Auscultation (Heart sounds/ Murmers Lung sounds – crackles, wheezes decreased/no air entry) - HR, BP, O2 saturations - Pulses and warmth of extremities - Presence of clubbing (Related to hypoxia // chronic resp issues like COPD )
34
PDA
Patent ductus arteriosis (PDA) Etiology: prematurity --> the more preterm – the higher likelihood of PDA - They will have mixing of blood → less effective oxygenation bc of that - Wil be acyanotic Clinical Signs: - increasing respiratory distress due to pulmonary edema; - apnea and bradycardia; - bounding pulses Management: - fluids (furosemide) - oxygen - indomethacin (to help close it) - Surgical ligation
35
CHD presentation after birth
Shock in the 1st hour after birth - Severe respiratory distress - Cyanosis - Poor perfusion Or nonspecific and difficult to differentiate from respiratory causes - If cyanosed – and oxygen helps, this can rule out cyanotic heart disease. - If cyanosed and oxygen does not help, this can indicate cyanotic heart disease. - do not to give cyanotic heart issue babies very much oxygen its not gonan help and it shifts their circulation
36
VSD and ASD Patho
VSD - shunt creation between the right and left ventricles - It allows oxygen-rich blood to move back into the lungs instead of being pumped to the rest of the body. ASD - a birth defect of the heart in which there is a hole in the wall (septum) that divides the upper chambers (atria) of the heart. normal heart pressure and o2 sats - pressures are higher in left
37
CoA and TOF
- Coarctation of the aorta is a narrowing (constriction) in a part of the body's main artery (aorta). - The heart must pump more forcefully to send blood through the aorta and on to the rest of the body TOF - Ventricular septal defect, overriding aorta, pulmonary stenosis and right ventricular hypertrophy. - These problems result in cyanotic, or blue, skin on babies because of a lack of oxygen.
38
TGA
- Aorta and pulmonary artery switched - oxygen-poor blood from the body enters the right side of the heart but the blood is pumped directly back out to the rest of the body through the aorta. - Oxygen-rich blood from the lungs entering the heart is pumped straight back to the lungs through the main pulmonary artery. - End up with very cyanotic picture because body can't access the pulmonary system - PDA in this case would be important
39
infective endocaditis
- Infection of valves/inner lining of heart Causes: - Altered blood flow and turbulence inside the heart - Damage to valvular endothelium - increases fibrin and deposition of platelets --> Microorganisms grow and form vegetation on the endocardium - Lesion may invade adjacent tissues (valves and myocardium) - Risk of emboli
40
nsg priorities for all CHDs
- contractility (medications – eg digoxin) - vasodilaters (ACE inhibitors --> to reduce resistance - eg captopril) - Remove excess fluid and sodium (decrease pre-load) (medications – diuretics) - Rest - Nutrition without fatigue (NG feeds) - Comfort and positioning (+/- sedation) - Improve oxygenation & decrease oxygen consumption - Preparation for invasive procedures and surgery
41
rheumatic fever
Rheumatic Fever - Inflammatory disease which occurs after group A β hemolytic streptococcal pharyngitis - Rheumatic heart disease: most significant complication of Rheumatic Fever. - Causes damage to heart valves; usually mitral valve
42
s/s of increased ICP in chold/infant
infant - Change in feeding, vomiting - Altered LOC (irritability, restlessness, poor consolability, lethargy) - Bulging fontanels - High-pitched cry - increased HC (head circumference) - separation of cranial sutures Child - Nausea/Vomiting - Altered LOC - Diplopia/blurred vision - Slurred speech - Seizures
43
Late s/s of increased ICP
- Alteration in pupil size and reactivity - Decreased motor or sensory response - Coma - Posturing (decerebrate/decorticate) - Papilledema - Projectile vomiting - Changes in Vital Signs - Bradycardia - Irregular or decreased respirations - Cheyne Stoke
44
Hr and increased temp on ICP
Decreased heart rate and resp rate which causes --> Decreased O2 and increased CO2 which causes --> Decebral vasodialation to increased O2 --> Causes increased ICP which further decreases resp rate Increased Temp (or instability) which causes --> Increased metabolic needs of brain which causes --> Seizures related to increased ICP which causes --> Increased metabolic rate and thus increased ICP
45
bacterial meningitis
Patho - organisms from the nasopharynx invade the underlying blood vessels, cross the blood–brain barrier, and multiply in the CSF) - inflammation, exudation, white blood cell accumulation, and varying degrees of tissue damage. - cerebral edema, purulent exudate, fibrin, or adhesions may occlude the narrow passages and obstruct the flow of CSF Usually abrupt onset - Fever/Chills/ Headache - Vomiting - Seizures (often the initial sign) - bulging fontanelles - infants may not have nuchal rigidity - Nuchal rigidity: May progress to opisthotonos - Positive Kernig and Brudzinski signs Signs and symptoms peculiar to individual organisms: - Petechial or purpuric rashes (meningococcal infection), especially when associated with a shocklike state - Joint involvement (meningococcal and Haemophilus influenzae infection) - Chronically draining ear (pneumococcal meningitis)
46
Care for meningitis
- A medical emergency (and ICU care) - Isolation precautions - Initiation of antimicrobial therapy - Maintenance of hydration - Maintenance of ventilation - Reduction of increased ICP - Management of systemic shock - Control of seizures - Control of temperature - Treatment of complications
47
hydrocephalus
- caused by an abnormal buildup of cerebrospinal fluid in the ventricles (cavities) deep within the brain - Ventricles engorged with CSF → infants can compensate well - Can be congenital or acquired Management - For congenital they will place a shunt to drain inside body → can get blocked or infected - Relieve, prevent ICP r/t hydrocephalus - Monitoring and early intervention for problems related to surgery
48
scoliosis | what is it? Angle? Types?
What - Lateralcurvature of the spine that usually involves spinal rotation (Structural or Non-Structural). - Curves with Cobb angle > 10 degrees = scoliosis. - Curves with Cobb angle < 10 degrees = spinal asymmetry and have no long-term significance. Classification of Scoliosis A. Early Onset Scoliosis B. Late Onset Scoliosis - Congenital Scoliosis - Neuromuscular Scoliosis - Idiopathic Scoliosis
49
neuromuscular scoliosis
Neuromuscular Scoliosis - Result of muscle imbalance and lack of trunk control - Early detection is important as early intervention may help improve function and delay progression - Lots are wheelchair ambulators (ie dont walk most of the time) → we want to help them with their posture until we must operate - Function can include: ambulation, positioning in wheel chairs, prevention of pressure ulcers, and cardio-pulmonary function
50
idiopathic scoliosis
3 subcategories: - Infantile: 0 to 3 years - Juvenile: 4 to 9 years - Adolescent: ≥ 10 years Adolescent Idiopathic Scoliosis (AIS) - most common form; 80 – 85% of cases. - The primary concerns regarding scoliosis often involve activities of daily living, as well as aesthetic appearance - When the scoliosis angle increases over 70 degrees, other organ systems can be affected (Heart/lungs)
51
AIS assesments
Inspection - Shoulder height asymmetry - Pelvic/hip asymmetry - Head in line with center of sacrum Adam’s forward-bending test (AFBT) - Assess degree of rotation and deformity - Patient bends forward at waist with knees straight and palms together - Thoracic or lumbar prominence noted Scoliometer (optional) - Measures angle of trunk rotation: run along spine from top to bottom during forward bend test more signs - Cafe au lait spors
52
diagnostic evaluation +cobb angle and risser signs
Xray required to confirm diagnosis and is indicated if: - Clinically evident scoliosis based on physical exam - Thoracic or lumbar asymmetry on physical exam - Family history of scoliosis 3 Foot Spine Xray (PA/LAT) identifies: - site of deformity (thoracic or lumbar) and curve pattern - Magnitude of curve (Cobb angle) - Skeletal maturity (Risser sign + Sanders) - Spondylolysis/Spondylolisthesis Cobb Angle - Degree of curvature measured according to Cobb method. - Gold standard for assessing degree of curve. - Essential to determine management of scoliosis and risk of progression. risser sign (pelvis) - Degree of ossification and fusion of iliac crest/apophysis. - Used to assess skeletal maturity using xray. - Maturity occurs in a stepwise fashion from lateral to medial.
53
Management of AIS
Monitor patients if… - Cobb angle <25 ° & still growing at time of presentation - Curve more than 45° and finished spinal growth. Treat patients if still growing and… - Cobb angles >25° considered for bracing if skeletally immature (Risser 0-2). - Cobb angles >45-50° at time of diagnosis will likely require surgery.
54
bracing for scoliosis
Bracing - Does not correct curvature!!! - Objective: Arrest curve progression. - Reduces chance of progressing to surgery - Must be skeletally immature (Risser 0-2) - TLSO (thoraco-lumbar-sacral orthosis) or Providence (night- time) brace routinely used. - Time in brace varies
55
post op teaching for scoliosis
- VS & Neurovascular checks with ankle dorsiflexion - Monitoring effect of PCA and PO analgesia ( nausea, vomiting, pruritis) - Monitor respiratory status + leg paresthesias - Assessment of peripheral neurologic signs in extremities is essential after spinal surgery (changes can occur up to 48 hours post op) - In/Out monitoring: Fluids, Drains – hemovac + foley - Activity - POD#1: Standing at bedside BID - POD#2: Walking BID or more. essential to progress recovery and avoid complications - Dressing intact and clean until discharge day then changed --> check Q4H.
56
discharge teaching for spinal surgery
- Follow up with surgeon 1-2 weeks post-op. - Activity restrictions – return to school 1 month post op, no contact sports x 6 months. - Frequent position changes - Monitor for signs of infection – fever, drainage, inc. pain - May shower 5-7 days post op, no baths or soaking for 6 weeks. Medications: - Narcotic PRN, - Tylenol str x3D then PRN - Celebrex x5D - PEG 3350 and Baclofen x5D
57
DDH and 5 risks
- DEVELOPMENTAL DYSPLASIA OF THE HIP (DDH) Risk Factors 5 F’s: - Family History - Fetal position – Feet first/ Breech presentation more likely - First born infant - increased risk with Females - Fluid – history of oligohydramnios (low amniotic fluid levels) - Family Hx of Breech position = Most Important RF
58
physicsal exam for DDH
Hip Exam: - Asymmetrical creases (thigh or gluteal) - Range of Motion (? Limited abduction) - Barlow or Ortolani maneuver - Galeazzi sign (knee level) Neck: - Preference to one side - Plagiocephaly (flat back of head) - Torticollis & strength (stiff neck) Spine: - Dysraphism (spina bifida) Upper & Lower Extremities: - Generalized motor assessment & reflexes - Foot abnormalities (MA or club foot)
59
swaddling and pavlick tx
- Cultures that keep the baby’s hips in an “M” style or wide position have lower rates of hip dysplasia - Hip-Safe swaddling methods are recommended for newborn infants. Pavlik Harness Treatment - Goal: To obtain and maintain concentric reduction of the hip and allow for healthy development of the acetabulum and hip joint. Success of Pavlik treatment - 80-95% How does it help? - Holds hips in a flexion position (>90-100 degrees) AND abduction position (optimal position for hip development) - Prevents adduction of the hip
60
secondary tx for DDH
When? - If the infant has failed the Pavlik harness treatment - If the child is over 6-12 months at diagnosis (grey area for Pavlik use) - We will put them in a cast instead types - Fixed abduction brace - Closed reduction under anesthesia & hip spica cast (can be tried up until 2 years of age) - Open reduction +/- pelvic osteotomy & hip spica cast
61
spica cast
what - A plaster cast from upper torso (nipple line) down tothe ankles Types - Bilateral full leg - Unilateral - Bilateral short leg (rare) - A-Frame/Petrie (phasing out) Objective: Immobilization of the hip, pelvis and/or femur
62
pain management for DDH surgery and position/transfers
- Epidural sometimes but mostly single shot epimorph (usually cast will help pain after 48 hours) - IV narcotics - PO narcotics - Antispasmodics for first 48 hours - Movement is difficult with a spica cast – on both patient and parents. (bean bag chair lol) - Reposition frequently and support legs with pillows - Watch feet for pressure sores Transfers depend on size of child - independent (parents) vs mechanical lift in older child. - Standard wheelchair not appropriate (use spika wheelchair) - Spica table
63
skin care for DDH
- Make sure cast is not too tight - Check skin for breakdown from cast. - Keep cast as clean and dry as possible - reverse Trendelenberg position is key → must be used so that the urine and BM go down and not towards the cast - Sponge bath only while in cast - Combat smells with eucalyptus oil Teaching - Clothing to the upper body only - has to be loose and larger than typical size. - Check skin daily for sores. - Casts are NOT waterproof – no bathing/swimming. - Watch food that may spill into the front of the cast or get trapped inside.