D1 Flashcards

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1
Q

<p>the major cause of pediatric burn?</p>

A

<p>scaled injury</p>

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2
Q

<p>characteristics of scaled burn-in child abuse?</p>

A

<p>mainly due to immersion to hot liquid
Clear demarcation with no splash mark
Involvement of back, buttock, and leg
sparing of the flexural part(ankle, knee, and hip flexure)
lesion inconsistent with the history
Delay in presentation
Uniform burn degree especially in case of severe burn</p>

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3
Q

<p>what action should be taken?</p>

A

<p>contact with childhood protective service immediately</p>

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4
Q

<p>unentational burn caracter?</p>

A
<p>Mainly spillage from upper
Proximal upper extremity, face, and proximal trunk
Presence of splash mark
non-uniform burn depth
asymmetric lesion</p>
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5
Q

epidemiology of tinea capitis

A

transmitted by direct contact or fomite

dermatophyte(Trichophyton and microsporidium)

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6
Q

CM?

A

scaly, erythematous skin lesion with hair loss
+-black dote on the site of lesion
+- Tender LDP
scaring and pruritis may be there

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7
Q

managment?

A

oral grisofulvin/terbinafin

treat contact with selinium selfide or ketokonazol shampo

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8
Q

alopecia areata?

A

Due to autoimmune
Clear area of hair loss w/o scaling
Discoid rash

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9
Q

Seborrheic dermatitis?

A

scaley, oily erythematous rash(dandruff)
around the hairline, ears, or nose; or in the center of the chest or back.
High risk of hair breaking

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10
Q

keratosis pilaris?

A

Characterized by retained hair b/n the hair follicle
Small painless papules
rough skin texture
mottled perifollicular erythema
exacerbated by dry and cold weather
commonly affect the posterior surface of the upper arm

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11
Q

Treatment when necessary?

A

use when necessary
emollient
topical keratolytic(salicylate, uric acid)

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12
Q

Pathogenesis of henock-schonlein purpura pathogenesis?

A

IgA mediated leukocytoclastic vasculitis

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13
Q

CM?

A

Triads(Palpable purpura,Arthritis/Arthralgia
and Abdominal pain,intususuption)
A renal disease similar to IgA nephropathy

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14
Q

Laboratory?

A

Normal platelet
Normal coagulation
Nephritic syndrome sign

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15
Q

Managment?

A

suportiv(RHD and NSAID)

IV glucocorticoid in sever case

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16
Q

Infectious complications of atopic dermatitis?

A

Impetigo
Eczema herpeticum
Molluscum Contagiousum
Tinea corporis

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17
Q

Impetigo CXS?

A

S.A and S.P(can progress to cellulitis and abscess)
Painful
nonpruritic
Hoony crust lesion

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18
Q

Eczema herpeticum CXS?

A
HSV
Painful
Vesicular
Punched out
hemorrhagic crusting
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19
Q

Molluscum Contagiousum CXS?

A

PV

Flesh-colored papule with central umbilication

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20
Q

Tinea corporis CXS?

A
trichophyton rubrum
Pruritic
erythematous
Central clearing
raised and erythematous border
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21
Q

Eczema herpeticum treatment?

A

acyclovir indicated in children

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22
Q

Atopic dermatitis presentation?

A

In young children
Dry, scaly, and erythematous lesion in chicks and extensor surface
In older children
Dry, thickened skin in antecubital fossa and popliteal area

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23
Q

Tinea pedis etiology?

A

Trichopytone(MCC;TP.rubrum)

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24
Q

CM?

A

Acute: Pruritic, burning pain erythematous vesicle/bulla
Chronic: erythematous, pruritic, Interdigital scale/fissure with an extension around.

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25
Q

treatment?

A

Topical antifungal
Azoles(miconazol)/terbinafine
Systemic(fluconazole, terbinafine) in severe case: for the patient did not respond to topical
Keep feet dry

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26
Q

What about scabies

A

Papule,pustule and burrow

treat with permethrin

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27
Q

effect of UV exposure?

A

Sunburn
Photoaging
Skin cancer

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28
Q

Sunburn symptom?

A

Changes in skin tone, such as pinkness or redness.
Skin that feels warm or hot to the touch.
Pain and tenderness.
Swelling.
Small fluid-filled blisters, which may break.
Headache, fever, nausea, and fatigue,
if the sunburn is severe.
Eyes that feel painful or gritty.

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29
Q

prevention?

A

exposure
sunscreen
clothing

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30
Q

exposure reduction?

A

Avoid all if you can
Avoid age < 6 mont
Avoid exposure 10:AM to 4:PM

31
Q

sunscreen?

A

Use SS cream protecting power 30-50
15-30 min before exposure
reapply every 2 hours and after swimming

32
Q

Clothing?

A

Long sleeves
Broad hat
Dark color
Tight weav

33
Q

Alopecia areata pathogenesis?

A

Genetic predisposition

Autoimmune hair bulb distraction

34
Q

CM?

A

painless, nonscary patchy hair loss
narrowing of hair shaft near the surface
positive hair pulling test(extract 5-5 hair at once)
recur but regrow during TX
It May be associated with AID(SLE, Autoimmune thyroid disease and vetiligo)

35
Q

mild/moderate hair loss?

A

Mild/Moderate:topical or intralesional CS

Sever: Topical immunosuppressant (diphenylcyclopropenone) with oral CS

36
Q

What about discoid SLE involving hair?

A

The lesion is erythematous and scaring

Another site SLE lesion

37
Q

Allergic contact dermatitis pathogenesis?

A

Require previous exposure(Immune sensitization)
Due to T cell RXN(Type IV)
Ethology are(URUshiol(from the plant(Poison ivy, oak, and sumac), nickel and neomycin)

38
Q

CM?

A
Start after 12 hr
Pruritis
Erythematous rash with a streak(especially in uroshiol)
Develop edema and vesicle
weeping drainage and crusting
usually, resolve in 1-3 week
39
Q

managment?

A

avoid exposure
removed exposed cloth
topical and oral corticosteroid

40
Q

Scabies pathogenesis?

A

Infection bay Sacrobitis scabi bite

Spread by direct person to person contact

41
Q

CM?

A

extremely pruritis
burrow/small erythematous papule/vesicle
wave space, flexor wrist, extensor elbow, axilla, umbilicus, and genitalia

42
Q

Treatment?

A

Topical permetrin or oral Ivermectine

43
Q

Perianal streptococcal infection sign?

A
Caused by S.Pyogenes
Sharpley demarcated, erythematous  and painful perianal rash
Constipation due to the pain
Bloody stool
Contact in recent day
44
Q

Managment?

A

Oral BLA

45
Q

Staphylococcal scalded skin syndrome pathogenesis?

A

Caused by exfoliativ toxin-producing S.A infection
The source is usually the umbilicus/circumcision site in the neonate
But in adult nares and skin
Toxin damage keratinocyte attachment within the epidermis

46
Q

CM?

A

Prodrom: Fever eriteblity and skin tenderness
Then, generalized erythema,
flaccid bullae/blister dominate in the flexural area
spare mucosa
Niklisky sign
Scaled skin lesion due to epidermal peeling

47
Q

Managment?

A

Naficiline/vancomycin

Supportive(wound care)

48
Q

Diagnosis?

A

clinical

Bulle is sterile

49
Q

Bullous impetigo?

A

The localized form of SSSS

50
Q

Seborrheic dermatitis pathogenesis?

A

associated with colonization by Malassezia furfur

Affect part of the body that have a sebaceous gland

51
Q

CM?

A

A peak in infancy/adulthood
Erythematous plaque/yellow gressy scale
Located on the scalp, face(eyebrows/eyelid.nasolabial fold and posterior ear), umbilicus and diaper area.
pruritis and pain is not typical

52
Q

Tx?

A

Mild: gentle emollient and non-medical shampo
severe: Glucocorticoid cream and topical ketoconazole

53
Q

Bullous and non-bullous impetigo D/C?

A

Etiology
CM
TX

54
Q

Etiology?

A

B: S.A
NB: SA & GAS

55
Q

CM?

A

B: enlarged flaccid bullous lesion with yellow fluid
—raptured lesion with a collarette of scale at the periphery
NB:Papules with pustule with honney crust lesion

56
Q

Treatment?

A

NB:Topical/if extensive :oral
B: Oral

57
Q

common childhood pigment disorder?

A

Cafe-au-laits spots
Congenital dermal melanocytosis
Congenital melanocytic nevus

58
Q

Cafe-au-laits spots?

A
Flat,hyperpigmented patches
Associated MAS(McCune Albright syndrome)/NF
59
Q

Congenital dermal melanocytosis?

A

Blue-gray patches
more common in African American
Common in lower back and buttock
Fades away with decades(no treatment require)
Due to the presence of melanocytes in the dermis

60
Q

Congenital melanocytic nevus?

A

Benign melanocyte proliferation
Increase density of hair lesion
5 % risk of melanoma

61
Q

Infantile hemangioma managment?

A

Also known as strawberry hemangioma
Can present with a patch of telangiectasia at birth
Proliferate age 0-1, bright red raised nodule
Involution: age 1-9,Deeper red/violet,regretion in size
Is capillary tumor due to endothelial proliferation

62
Q

managment?

A

observation

topical beta-blocker(at site complication will be anticipated)

63
Q

Complication?

A

Ulceration/scaring
Vision impairment if near eye
Life-threatening if the near airway

64
Q

cherry hemangioma?

A

common benign vascular tumor in adult
Dilation of capillary and PCV
usually widespread at trunk and face
enlarge with age

65
Q

Pathogenesis of T.Versicolour?

A

Malaysia globosa

exposure to the skin to hot and humid air

66
Q

CM?

A
Hypo/Hyperpigmented lesion
may have mild erythema
face in children
trunk and UE in above
\+- fine-scale and pruritis
67
Q

Diagnosis?

A

KOH: hyphae and yeast(spaghetti in metaball)

68
Q

Tx?

A

Topical ketoconazole, Terbinafine or selenium sulfide

Generally nystatin not treat dermatocyte

69
Q

Pityriasis rosea?

A

viral prodrome
classically begin by single salmon colourd macule
then multiple lesions in trunk and extremity(Christmas tree)
erythematous
eventually, desquamate
Pruritis

70
Q

managment?

A

usually self-limited

antihistamine and topical steroid for pruritis

71
Q

erythema toxicum neonatorum?

A
asymptomatic
erythematous papule and pustule
full-term neonate in first 2 week
spare hand and palm
not need treatment(resolve by itself)
72
Q

bed bug skin lesion?

A

pruritic
small puncture
maculopapular in linear group
on close free rea

73
Q

spider bite?

A

solitary
papule/pustule
weal/+-pruritis

74
Q

managment?

A

suportive(CS and anti histamin)

decontamination