CXR and ECG Flashcards

1
Q

Approach to CXR analysis

A

Assessment
- Verify Patient name, DOB, date of exam
- Exposure (fine lung markings should be visible)
- Rotation
- Expansion

ABCDEFG:
1. Air where it shouldn’t be (under diaphragm, pneumothorax, mediastinum, skin)
2. Bones
3. Cardiac silhouette and size
4. Diaphragm
5. Effusion and Equipment (ETT, NGT, ECG)
6. Fields (lung fields) - should be symmetric without haziness, blotches or dots
Great vessels

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2
Q

DDX for white out on CXR

A
  • Consolidation: trachea MIDLINE, contralateral lung normal, ribs normal, may see air bronchograms
    • Collapse: Trachea pulled towards SAME side, crowding of ribs in affected side, contralt lung is hyperinflated
    • Fluid (effusion): trachea midline or towards OPPOSITE side, contralateral lung will be normal/collapsed, meniscus effect
    • Mass: trachea midline or towards OPPOSITE side, contralateral lung will be normal/collapsed, rounded appearance to lesion
    • Lobectomy or atresia - trachea towards SAME side, hyper inflated contralateral lung, may see staples post lobectomy
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3
Q

ECG analysis

A
  1. Rate = 300/(large boxes between QRS)
  2. Rhythm
  3. Axis - look at leads I, II, AVF
    1. QRS axis
      1. Normal values
        1. 0-1: RAD (NEG I/POS AVF)
        2. > 1YO: NORMAL AXIS (POS 1/POS AVF)
    2. P wave axis - normal if positive p wave in I, and aVF
      1. Low atrial rhythm if neg in II and or AVF
    3. Segments
      1. P wave
        1. Bifid or long = LA enlargement
        2. High/peaked = RA enlargement
      2. PR interval
        1. Duration= 3-5 small squares (120-200ms)
          1. > 200ms = heart block
          2. <120ms = pre-excitation
      3. QRS
        1. Duration = < 3 small squares (<120ms)
          1. Narrow = Supraventricular
          2. Broad = ventricular
        2. Height
          1. Tall V1-3: RVH
          2. Tall V4-6: LVH
        3. Morphology
          1. Delta wave = WPW
      4. T wave
        1. Progression
          1. Inversion V1-3 at birth which progressively revert in reverse order
          2. Persisent inversion of V1 after 8yo is normal
      5. ST segment (should be isoelectric)
      6. QT
        1. Should be < 1/2 RR interval
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4
Q

ASD

ECG, CXR and clinical findings in a 5yo

A

Incomplete RBBB
ESM at upper LSE
CXR - cardiomeg (RV hypertrophy)
plethora
prominent PA
Fixed split S2

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5
Q

ECG features L ventricular hypertrophy

A

Large R wave voltages in V5, V6
Deep S wave in V1
(note normal ranges change with age)
LAD

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6
Q

4/6 pan systolic murmur at LSE

A

VSD (voltages due to L ventricule dilation which normalises over time)

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7
Q

VSD

ECG, CXR findings

A

CXR - cardiomegaly, pulmonary plethora, hyper expanded lungs
ecg - rvh, lvh

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8
Q

R ventricular hypertrophy ecg findings

A

rad
R wave dominance v1
S wave dominance V5, V6
upright t wave v1

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9
Q

loud systolic murmur
mild cyanosis 88%
R V hypertrophy

?what heart defect

A

TOF pre op

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10
Q

What do tall p waves indicate?

A

Right atrial dilatation/hypertrophy

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11
Q

LONG bifid p wave in lead II

A

L atrial dilatation/hypertrophy

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12
Q

Conditions that cause R atrial hypertrophy

A

Large ASD

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13
Q

Conditions that cause left atrial hypertrophy

A

Mitral regurgitation or stenosis
Large VSD or duct
Cardiomyopathy

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14
Q

Causes of L axis deviation

A

LVH esp with volume overload (large VSD)
LBBB (wide QRS)
Tricuspid atresia
AV canal defect

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15
Q

R atrial enlargement = what on ECG?

A

p wave hight >3mm (3 small squares)

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16
Q

L atrial enlargement = what on ECG?

A

bifid p waves
prolonged > 10ms (2.5 small squares)

17
Q

What condition is assoc w Extreme axis deviation?

A

AVSD