CXR, ABG, ECG Flashcards

1
Q

Whats the check for xray quality?

A

PRIM
P- Projection
R- Rotation (SP-clav dist)
I - Inspiration 7 ant ribs
M - Markings

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2
Q

What are the structures to consider in a CXR?

A
  • Heart 50%
  • White = effusion if unilateral and solid. Pneumonia if unilateral and fluffy. Oedema if bilateral and fluffy. Fibrosis, bilateral and honeycomb
  • Trachea - central, deviation towards a collapse and away from a pneumothorax
  • Mediastinum can wide with right upper lobe collapse (tracheal dev) or aortic dissection (no trach)
  • Blunted costophrenic angles in pleural effusions
  • Sail shadow behind heart suggests left lower lobe collapse.
  • Not clear apices - TB or tumor
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3
Q

What are the ABCDE signs of pulm. oedema on CXR?

A

A - Alveolar oedema (bat wings)
B - Kerly B ligns (intersitital oedema)
C - Cardiomegaly
D - Diversion of blood to upper zones (upper zone vessels larger than lower)
E - Pleural effusions

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4
Q

Whats the consideration for an arterial blood gas oxygen level for a patient on supplemental oxygen?

A

You would expect them to have a grossly higher level of PaO2 even if it falls within the normal range they can still be grossly hypoxic.

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5
Q

Whats a trick method for calculating a normal PaO2?

A

Approximately: FiO2 - 10 in kPa (PaO2 should exceed this)

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6
Q

How do we distinguish the types of respiratory failure on an ABG?

A

Type 1: low or normal PaCO2 i.e fast/normal breathing: Caused by damage to heart or lungs causing SOB

Type 2: High PaCO2. (slow/shallow breathing), blue bloaters of COPD. or neuromuscular failure or restrictive chest wall abnormalities.

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7
Q

What are some tips and tricks for determining the cause of acid: base disturbance being resp or metabolic.

A

If only PaCO2 abnormal = Resp cause.

If only HCO3 = metabolic cause.

If both increased or decreased = compensation. Abnormal pH = partially compensated.

If PaCo2 and HCO3 abnormal in opposite directions then indicates coexistent metabolic and respiratory disease

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8
Q

What are some rough causes of Resp acid and alkalosis, and meta acid and alkalosis?

A

Resp Alk: Rapid breathing; Anxiety or disease.

Resp acid: Type two resp. failure.

Metabolic alk: Vomiting, diuretics, and conns syndrome.

Metabolic acidosis: DKA, lactic acidosis, renail failure, intoxication; ethanol, methanol, ethylene glycol. Many causes. Anion gap further elucidates cause.

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9
Q

Whats the mneumonic for remembering BBB and ECG changes?

A

William morrow:

LL
1st deflection of QRS; V1 = down (W), V6 = up (M)

RR
1st deflection of QRS; V1 = up (M), V6 = down (W)

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10
Q

Describe the heart blocks:

A

First degree: Constant PR but is longer than 1 large square.

Second degree (Type 1) : Increased PR and then missing QRS

Second degree (type 2): 2-3 P waves for a QRS

Complete heart block: No association between P and QRS

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11
Q

Whats the QRS width?

A

<3 small squares = no bundle branch block. 3+ = BBB

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12
Q

Whats the height of QRS and indicates?

A

Add largest deflection in V1 and V6. If exceeds 3.5 large squares = left ventricular hypertrophy. If small complexes consider pericardial effusion

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13
Q

What does ST elevation look like and indicate?

A

Infarction: elevated in SOME leads

Pericarditis: Saddle shaped and raised in ALL leads.

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14
Q

What does ST depression look like and indicate?

A

Ischaemia or infarction: ST segment flat and depressed in SOME leads. - Check troponin to distinguish.

Digoxin tx: ST segment downsloping in all leads.

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15
Q

What are some common T wave anomalies:

A

If more than 2/3rds of QRS throughout = peaked = hyperkalaemia.

Inversion: Normal in AVR and I (Top middle two leads), in other leads suggests old infarction or LVH

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16
Q
A