CXR, ABG, ECG Flashcards
Whats the check for xray quality?
PRIM
P- Projection
R- Rotation (SP-clav dist)
I - Inspiration 7 ant ribs
M - Markings
What are the structures to consider in a CXR?
- Heart 50%
- White = effusion if unilateral and solid. Pneumonia if unilateral and fluffy. Oedema if bilateral and fluffy. Fibrosis, bilateral and honeycomb
- Trachea - central, deviation towards a collapse and away from a pneumothorax
- Mediastinum can wide with right upper lobe collapse (tracheal dev) or aortic dissection (no trach)
- Blunted costophrenic angles in pleural effusions
- Sail shadow behind heart suggests left lower lobe collapse.
- Not clear apices - TB or tumor
What are the ABCDE signs of pulm. oedema on CXR?
A - Alveolar oedema (bat wings)
B - Kerly B ligns (intersitital oedema)
C - Cardiomegaly
D - Diversion of blood to upper zones (upper zone vessels larger than lower)
E - Pleural effusions
Whats the consideration for an arterial blood gas oxygen level for a patient on supplemental oxygen?
You would expect them to have a grossly higher level of PaO2 even if it falls within the normal range they can still be grossly hypoxic.
Whats a trick method for calculating a normal PaO2?
Approximately: FiO2 - 10 in kPa (PaO2 should exceed this)
How do we distinguish the types of respiratory failure on an ABG?
Type 1: low or normal PaCO2 i.e fast/normal breathing: Caused by damage to heart or lungs causing SOB
Type 2: High PaCO2. (slow/shallow breathing), blue bloaters of COPD. or neuromuscular failure or restrictive chest wall abnormalities.
What are some tips and tricks for determining the cause of acid: base disturbance being resp or metabolic.
If only PaCO2 abnormal = Resp cause.
If only HCO3 = metabolic cause.
If both increased or decreased = compensation. Abnormal pH = partially compensated.
If PaCo2 and HCO3 abnormal in opposite directions then indicates coexistent metabolic and respiratory disease
What are some rough causes of Resp acid and alkalosis, and meta acid and alkalosis?
Resp Alk: Rapid breathing; Anxiety or disease.
Resp acid: Type two resp. failure.
Metabolic alk: Vomiting, diuretics, and conns syndrome.
Metabolic acidosis: DKA, lactic acidosis, renail failure, intoxication; ethanol, methanol, ethylene glycol. Many causes. Anion gap further elucidates cause.
Whats the mneumonic for remembering BBB and ECG changes?
William morrow:
LL
1st deflection of QRS; V1 = down (W), V6 = up (M)
RR
1st deflection of QRS; V1 = up (M), V6 = down (W)
Describe the heart blocks:
First degree: Constant PR but is longer than 1 large square.
Second degree (Type 1) : Increased PR and then missing QRS
Second degree (type 2): 2-3 P waves for a QRS
Complete heart block: No association between P and QRS
Whats the QRS width?
<3 small squares = no bundle branch block. 3+ = BBB
Whats the height of QRS and indicates?
Add largest deflection in V1 and V6. If exceeds 3.5 large squares = left ventricular hypertrophy. If small complexes consider pericardial effusion
What does ST elevation look like and indicate?
Infarction: elevated in SOME leads
Pericarditis: Saddle shaped and raised in ALL leads.
What does ST depression look like and indicate?
Ischaemia or infarction: ST segment flat and depressed in SOME leads. - Check troponin to distinguish.
Digoxin tx: ST segment downsloping in all leads.
What are some common T wave anomalies:
If more than 2/3rds of QRS throughout = peaked = hyperkalaemia.
Inversion: Normal in AVR and I (Top middle two leads), in other leads suggests old infarction or LVH