Cx Spine Stabilization Flashcards

1
Q

what are the superficial neck flexor and extensor muscles?

A

flexor:
SCM and anterior scalene
Extensor:
splenius capitus

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2
Q

superficial muscles tend to become hypo or hypertonic?

A

hypertonic - protective spasms, inhibiting local muscles

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3
Q

what are the deep neck stabilizers?

A
multifidus (segmental attachments)
longus capitis and longus colli (anterior stability)
semispinalis cervicis (lower cervical posterior stability)
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4
Q

deep neck stabilizers are static or dynamic stabilizers?

A

dynamic

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5
Q

what two criteria are required for “clinical instability”? (NOT panjabi)

A

1) increase in NZ of C-sp ROM

2) less resistance to passive motion in the elastic zone than one would expect

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6
Q

according to Panjabi, what are the three subsystems of spinal stability?

A

passive system
active system
neural system

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7
Q

what three things does the passive subsystem do (C-sp stability)?

A
  • provides stabilization of the elastic zone
  • limits the size of the neutral zone
  • provides neural subsystem info on vertebral position and motion
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8
Q

what four things does the active system do (C-sp stability)?

A
  • generates forces to stabilize the spine
  • controls the motion that occurs within the neutral zone
  • contributes to maintaining the size of the neutral zone
  • provides the neural subsystem with info on forces generated by muscles
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9
Q

what does the neural subsystem do other than receive info from the other two systems (C-sp stability)?

A

from the info: it determines the requirements for stability and acts on spinal muscles to produce the required forces

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10
Q

what is Panjabi’s definition of spinal instability?

A

“Inability of the spine under physiologic loads to maintain its pattern of displacement so that no neurologic damage or irritation, no development of deformity, & no incapacitating pain occur”

  • essentially this is an increase in NZ that the three subsystems cannot compensate for, resulting in poorly coordinated mvt within this increased NZ leading to symptoms
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11
Q

what is the cause of this increased NZ with C-sp instability? what are four contributing factors?

A
  • degeneration and mechanical injury of spinal stabilizers

- contributing factors: Poor posture, repetitive microtrauma, macrotrauma, poor motor control of ms

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12
Q

for Hypermobility of the C-sp, what are some MOIs?

A
  • macrotrauma (MVA, FOOSH)

- poor posture (repetitive or sustained…)

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13
Q

what is the pain site and pain type for hypermobility of the C-sp?

A

pain site: local

pain type: feeling unstable, lack of control, may feel noise

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14
Q

what is the pain pattern for hypermobility of the C-sp?

A

pain increases with prolonged static postures, plus there is a feeling of fatiguability and inability to hold head up. Sharp pain with sudden movements

pain decreases with self or therapist manipulation, or with external support

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15
Q

what might you see on a patient with hypermobility (observation)?

A
  • poor posture

- cutaneous creases

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16
Q

what might you notice during ROM assessment on a hypermobile C-sp?

A
  • painful and increased ROM
  • clicks, clunks
  • creases with movement
  • movement is not smooth, look for aberrant movement
17
Q

what might you see with PAs and PPIVMs with a hypermobile C-sp?

A
  • increased NZ, R1 & R2 and soft or normal EF
18
Q

what might you notice during a biomechanical assessment of a hypermobile C-sp?

A
  • Cranio-cervical flexion test (CCFT) = increased use of superficial muscles and reduced use of deep neck flexors.
19
Q

name one condition associated with C-sp hypermobility/instability…

A

UNLT 1 may be +ve

20
Q

what are five principles to include when dealing with C-sp hypermobility/instability?

A
  • train tonic endurance of deep ms: should be a slow, tension type contraction
  • exercise should be pain-free
  • add outer unit after good inner unit recruitment
  • incorporate in functional activities
  • note progression and increase pressure and reps accordingly
21
Q

what are four tests for C-sp hypermobility/instability that have to be performed in ordeR?

A

1) low resistance - CCFT
2) add deep neck extensors
3) add anterior mobilizers
4) add posterior mobilizers

22
Q

what is the ultimate goal of conservative Rx for C-sp hypermobility/instability? What are three elements of treatment that can be used to achieve this goal?

A

Decrease stress on involved segments by:

  • patient education on posture, which will decr stress on segments and put them in a more efficient position.
  • mobs above and below hypermobility may decr stress on passive system.
  • neuromuscular control exs: improve function of the active subsystem, in hopes that the deep muscles compensate for the increase in NZ that happens with hypermobility/instability. Improving coordination and quality of mvt of deep muscles may also help to inhibit superficial ones.
23
Q

what three elements of a HEP have been shown to improve outcomes?

A
  • deep neck flexors strengthening
  • stretching ms that tend to tighten (U trap, levator)
  • strengthen scap adductors and retractors

Note how these three are all the most typical postural corrective measures ever.