cvs regulation Flashcards

1
Q

CO x TPR

A

BP

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2
Q

SV x HR

A

CO

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3
Q

quantity of blood pumped by heart per minute

A

CO

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4
Q

volume of blood ejected with each heart beat

A

SV

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5
Q

inhibits automaticity

A

PNS

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6
Q

Ach activates Kach channels in cardiac cells –> open kach channels –>

A

SA AV node with cholinesterase –> decay quickly

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7
Q

enzyme that destroy Ach

A

cholinesterase

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8
Q

Ach released quickly activates special Ach-regulated K+ channels –>

A

brief latency –> rapid decay of response

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9
Q

rapid decay of response –>

A

beat by beat control of SA and AV nodal function

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10
Q

enhances automaticity

gradual decay of response

A

SNS

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11
Q

site: aortic arch, carotid sinuses

A

baroreceptor reflex

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12
Q

> 100-180 mmHg –>

A

increase PNS, minimal SNS

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13
Q

< 100-180 mmHg –>

A

increase SNS, absent PNS

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14
Q

increase blood volume –> increase HR

A

Bainbridge reflex

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15
Q

increase blood volume –>

A

bainbridge reflex

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16
Q

decrease blood volume –>

A

baroreceptor reflex

17
Q

increase SNS –> increase HR

A

inspiration

18
Q

increase PNS –> decrease HR

A

expiration

19
Q

exaggerated when vagal tone is increased

A

Sinus Arrhythmia

20
Q

stimulus peripheral chemoreceptors

A

hypoxia
hypercapnea
acidosis

21
Q

peripheral chemoreceptors –> stimulation medullary vagal centers –>

A

decrease HR

22
Q

stimulate endocardial receptors of ventricle –>

A

dec HR

dec TPR

23
Q

ANS: SNS –?

A

increase HR

24
Q

PNS –?

A

decrease HR

25
Q

higher centers

A

increase HR

26
Q

Baroreceptor reflex: increase BP –?

A

decrease HR

27
Q

baroreceptor reflex: decrease BP –?

A

increase HR

28
Q

bainbridge: increase blood volume –?

A

increase HR

29
Q

inspiration –?

A

increase HR

30
Q

expiration –?

A

decrease HR

31
Q

chemoreceptor reflex:

PC02 P02

A

decrease HR

32
Q

invoked in response to changes in resting length of myocardial fibers

A

frank-starling mechanism

33
Q

increase ventricular contraction –>

A

greater stroke volume

34
Q

increase in fiber length alters cardiac performance by

A

altering number of myofilament crossbridges

35
Q

increase ventricular volume –>

A

increase cardiac fiber length

36
Q

optimal fiber length –> increase filling pressure –>

A

overstretch fibers –> LV dysfunction

37
Q

decrease contraction interval –> increase contractile force –>

A

increase Ca++

38
Q

sino mahal mo???

A

si lllooooyyyyddddd