CVD - Basic ECG Flashcards

1
Q

What are some additional lead placements in ECG? What are the indications?

A
  • Right ventricular leads V4R-V6R: if suspecting right ventricular infarction
  • Posterior leads V7-9: if suspecting posterior ischaemia
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What is a systemic ECG interpretation (order of things)?

A
  • Rhythm and rate
  • Cardiac axis
  • PR interval
  • QRS complexes
  • ST segments and T waves
  • QT interval
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What is the ratio of p waves to QRS in normal sinus rhythm?

A

1:1 ratio

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

How do Premature atrial complexes (“atrial ectopics” look like on ECG?

A

early, narrow complex QRS followed by compensatory pause

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

How do Premature ventricular complexes (“ventricular ectopics”) look like on ECG?

A

early, broad complex QRS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

How does Atrial fibrillation look like on ECG?

A

absence of p waves, irregularly irregular rhythm

Need to comment on ventricular response rate

> 100 – “rapid ventricular response rate”

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

How does Atrial flutter look like on ECG?

A

“saw tooth” appearance of p waves due to large re-entrant pathway in atrium, REGULAR 300bpm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

How do you pick left axis/right axis deviation on ECG?

A

Look at Lead I & II/aVF.

  1. Normal axis: positive, positive
  2. Left axis dev: positive, negative (Ladies Adore Diamonds; in diamond shape)
  3. Right axis dev: negative, positive (Rover Adores Digging; in the shape of dug bone edges. opposite of diamonds)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

When do you get left axis deviation?

A
  • Left anterior hemiblock
  • Ischaemic heart disease
  • Cardiomyopathy
  • Hypertension
  • Wolff-Parkinson-White syndrome - right sided accessory pathway
  • (NB LV hypertrophy is not a cause)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

When do you get right axis deviation?

A
  • Normal finding in children and tall thin adults
  • RV volume/pressure overload: RV hypertrophy, ASD, VSD, pulmonary embolus
  • Lung pathology: COPD, PE
  • Dextrocardia
  • Wolff-Parkinson-White syndrome - left sided accessory pathway
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

When do you get extreme right axis deviation?

A
  • Lead transposition
  • Ventricular tachycardia
  • Emphysema
  • Hyperkalaemia
  • Paced rhythm
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What does atrioventricular dissociation indicate?

A

Complete heart block

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

(3) types of abnormalities of the QRS complex you can see on ECG

A

–Voltages eg. increased in LV hypertrophy, decreased in cardiac amyloidosis
–Q waves
–Conduction eg. left or right bundle branch blocks

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

How do you diagnose LVH on ECG “on voltage criteria”?

A
  • Sum of S in V1/V2 & R in V5 or V6 (whichever is larger) ≥ 35 mm (7 large squares)
  • or R in aVL ≥ 11 mm

NB: Echocardiography is a more accurate test for LV hypertrophy than ECG criteria alone

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What are pathological Q waves?

A

–marker of electrical silence which implies established full thickness death of myocardium i.e scar

– > 25% height of the corresponding R wave
(and/or > 40 msec width and > 2mm in depth)

–Present in more than 1 contiguous (adjacent) lead

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Classic morphology for LBBB on ECG

A

–“WilliaM” – W in V1(often not obvious) and M pattern in V6
–Inverted T waves lateral leads V5-V6, I, aVL
–No septal Q waves

17
Q

Classic morphology for RBBB on ECG

A

–“MarroW” – M (rSR’) pattern in V1 and W in V6
–Inverted T waves V2-V3
–Slurred S wave in V6

18
Q

(4) causes of ST segment changes

A

– Myocardial ischaemia/infarction
–Pericarditis (widespread ST segment elevation)
–LV hypertrophy with “strain” pattern (ST segment depression)
–Drugs eg. digoxin

19
Q

Inferior leads of ECG

A

II, III, aVF

20
Q

Lateral leads of ECG

A

High lateral: I, aVL

Lateral: V5-6

21
Q

Anteroseptal leads of ECG

A

V1-4

22
Q

Briefly discuss the progressino of acute ischaemic changes seen on ECG

A
  1. ST elevation (a key sign of acute myocardial infarction requiring urgent treatment)

+/- T wave inversion (often persists long term)

  1. Q waves: if full thickness infarction (previous infarction)
23
Q

How does pericarditis present on ECG?

A

Widespread saddle shaped ST elevation

24
Q

What does horizontal ST depression suggest?

A

significant myocardial ischaemia

25
Q

What is a “strain” pattern on ECG?

A

ST depression with T wave inversion

26
Q

(3) causes of T wave abnormalities

A

–Myocardial ischaemia
–LV hypertrophy/strain, digoxin effect
–Systemic issues eg. electrolyte imbalances (K+,Mg2+, Ca2+)

27
Q

What (2) morphologies of T waves are most useful in diagnosing MI?

A

Biphasic or inverted T waves

28
Q

Briefly describe Wolff-Parkinson White syndrome

  • structural abnormality
  • consequences
  • (2) ECG changes
A

Accessory pathway bypasses atrioventricular node

  • leads to earlier (pre)-excitation of the ventricle than would occur with conduction through normal pathway
  • may lead to rapid regular tachycardias

ECG:

  • Short PR interval
  • Delta wave reflecting pre-excitation (early excitation) of the ventricle