CVA Impairments Part 3 Flashcards
What types of asymmetries are seen w/Hemiplegic gait?
- Spatial asymmetries
- decreased step length
- Temporal asymmetries
- decreased single-limb stance time
- increased swing time
- intra-limb ratio of swing: stance time
- additional
- decreased WB in stance
- decreased weight shift in stance
- decreased step height in swing
how does the temporal features of hemiplegic gait impact overall gait?
decreased gait speed
List some other common features (not including the LE) of hemiplegic gait
- UE
- decreased or absent arm swing
- Trunk
- ipsilateral lateral trunk lean
- forward trunk lean
T/F: the ipsilateral lateral trunk lean that may be observed in hemiplegic gait is due to trunk weakness
FALSE
the trunk is often spared
the weakness may be coming from the hip (glute med weakness pulling the trunk down)
Impact of Hemiplegic Gait on the pelvis/hip
heel strike → mid-stance
- ↓ pelvic rotation
- ↓ hip flexion
- ↑ hip IR
- ↑ hip adduction
- Trendelenberg
Impact of Hemiplegic gait on the knee
heel strike → mid-stance
3 common knee patterns:
- Increased knee flexion (particularly at IC)
- decreased knee flexion during early stance phase, followed by knee hyperextension in mid to late stance
- Excessive knee hyperextension throughout most of the stance phase
Impact of Hemiplegic gait on the foot/ankle
heel strike → mid-stance
- ↓ tibial progression
- ↓ ankle DF
- Lack of heel strike
- Foot flat initial contact
- Foot slap after initial contact
- Instability at foot/ankle complex à inversion, supination
- Pes planus
Impact of Hemiplegic gait on the pelvis/hip
mid-stance → terminal stance
- decreased pelvic rotation
- decreased hip extension/terminal stance
- hip flexion during forward progression
Impact of Hemiplegic gait on the knee
mid-stance → terminal stance
- decreased knee extension
- knee buckling
- delayed movement into knee flexion in prep for swing phase
Impact of Hemiplegic gait on foot/ankle
mid-stance → terminal stance
- May still see decreased tibial progression (step-to pattern)
- decreased heel off at terminal stance
Impact of Hemiplegic gait on pelvis/hip
Initial swing → terminal swing
- decreased hip flexion
- hip hiking
- circumduction
- increased compensatory ER
Impact of Hemiplegic gait on the knee
initial swing → terminal swing
- initial swing → mid-swing = decreased knee flexion
- mid-swing → terminal swing = decreased knee extension
Impact of Hemiplegic gait on foot/ankle
Initial swing → terminal swing
- poor foot clearance
- toe drag
- decreased ankle DF
- increased inversion
What are some additional considerations w/post stroke gait? (i.e other factors that influence it)
- Tone abnormalities
- Somatosensory deficits
- Vision deficits
- Coordination deficits
- Perceptual deficits
how can tone abnormalities influence post-stroke gait?
- Spasiticity
- movements might appear stiff, en-block movements
- clonus will cause jerky movements at joints
- UE spasticity patterns commonly exacerbate during gait
- Hypotonia
- buckling LE
- Floppy UE
how can somatosensory deficits influence post-stroke gait?
- variable foot placement at IC
- risk for ankle rolling
how can vision deficits influence post-stroke gait?
- Visual field losses or loss of visual acuity
- tripping over obstacles on floor
- hitting door frames, walls
- decreased awareness of oncoming obstacles
- veering while walking
- Dysconjugate gaze
- visual disruption → LOB
how can coordination deficits influence post-stroke gait?
- Cerebellar or Sensory originates
- fractionated, dyskinetic swing phase
- fractionated, dyskinetic arm swing
- slower movements
- variable foot placement
- trunk ataxia → LOB
- cerebellar only: EOM incoordination → visual disruption → LOB
how can perceptual deficits influence post-stroke gait?
- Visuospatial neglect
- visual → veering, curved trajectory of gait path
- decreased awareness of oncoming obstacles
- difficulty following directions involving neglected side
- Sensory neglect
- similar to somatosensory deficits, though often more severe
- Motor neglect
- involved hemibody often “left behind”
- Pusher’s syndrome
- increased extension in uninvolved UE (if holding AD) and LE in stance
- lateral shift of COG outside BOS leading to falls
- absent reactionary strategies → falls
- increased extension in uninvolved UE (if holding AD) and LE in stance