CVA Impairments 1: motor based Flashcards

1
Q

Mild ipsilateral weakness can be seen post-CVA and is most notably observed in ________________

A

proximal muscles

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2
Q

Common patterns of weakness between muscle pairs:

A

Ext > flex (EXCEPT: DF > PF)
ER > IR
ABD > ADD
Evertors > Inverters

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3
Q

Motor control

A

the underlying substrates of neural, physical, and behavioral aspects of movement

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4
Q

Reactive (feedback) and proactive (feedforward) movement is associated with

A

motor control

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5
Q

Motor Plan

A

an idea or plan for purposeful movement that is made up of component motor programs.

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6
Q

Motor program

A

An abstract representation that when initiated results in the production of a coordinated movement sequence.

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7
Q

Motor learning

A

A set of internal processes associated with feedback or practice leading to relatively permanent changes in the capability for motor skills.

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8
Q

Motor Recovery

A

the reappearance of motor patterns present prior to CNS injury performed in the same manner as prior to injury

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9
Q
A
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10
Q

Motor compensation

A

The appearance of new motor patterns resulting from changes in CNS

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11
Q

Adaptation and substitution are associated with

A

motor compensation

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12
Q

Common impairments of motor control

A

Abnormal synergies and apraxia

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13
Q

UE flexion synergy

A
  • Scapular retraction/elevation or hyperextension
  • Shoulder ABD, ER
  • Elbow FLEX*
  • Forearm supination
  • Wrist finger flexion
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14
Q

LE Flexion synergy

A
  • Hip flexion*, abd, ER
  • Knee flex
  • Ankle DF, Inversion
  • Toe DF
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15
Q

UE Extension Synergy

A
  • Scapular protraction
  • Shoulder ADD*, IR
  • Elbow EXT
  • Forearm pronation
  • Wrist and finger FLEX
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16
Q

LE Extension Synergy

A
  • Hip EXT, ADD*, IR
  • Knee EXT
  • Ankle PF*, Inversion
  • Toe PF
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17
Q

Apraxia

A

Inability to plan and execute purposeful movements that cannot be accounted for by any other reason.

18
Q

Ideomotor Apraxia

A

Inability to produce movement on command but able to move automatically, conceptualization of task remains intact.

19
Q

Ideational apraxia

A

inability to produce movement on command or automatically, complete breakdown of conceptualization of task

20
Q

Stage 1 of Motor Recovery

A

period of flaccidity immediately following acute episode.

21
Q

Stage 2 of Motor Recovery

A
  • limb synergies/components may appear
  • minimal voluntary movement
  • spasticity begins
22
Q

Stage 3 of Motor Recovery

A
  • Voluntary control of movement synergies
  • Increase spasticity (may become severe)
23
Q

Stage 4 of Motor Recovery

A
  • Movement combos that do not allow the paths of synergy are mastered (difficult > ease)
  • Spasticity begins to decline
24
Q

Stage 5 of Motor Recovery

A
  • More difficult movement combos are learned
  • Synergies start to lose their dominance over motor acts.
25
Q

Stage 6 of Motor Recovery

A
  • Disappearance of spasticity
  • Individual joint movements become possible and coordination approaches normal
26
Q

What four factors have a significant influence in a patient’s progression through the stages of motor recovery

A
  1. Initial weakness
  2. Presence of spasticity
  3. Cognitive deficits
  4. Access to rehab
27
Q

Why is MMT not the best option to evaluate with a post-CVA patient?

A

MMT requires selective capacity (ability to isolate a single joint movement)

28
Q

The MDC for UE and LE of the Fugl-Meyer Assessment is

A

UE 5.4 points
LE 5 points

29
Q

The MCID for the Fugl-Meyer Assessment for UE/LE is:

A

10 points for both

30
Q

The MCID of the Rivermead Motor Assessment is

A

3 points

31
Q

If unable to complete graded exercise testing what is the recommended exercise intensity and modifications to compensate for intensity.

A

Light to moderate exercise is recommended w/increased frequency and duration

32
Q
A
32
Q

Dysynergia

A

fragmented movement patterns (movements occur in sequence of component parts rather than a single and coordinated smooth output)

33
Q

Asynergia

A

loss of ability to associate muscles together for complex movements

34
Q

Ataxia

A

uncoordinated movements that manifest when voluntary movements are attempted

35
Q

Classification of ataxia:

A

Cause or Location (limb, truncal, gait)

36
Q

What is tone

A

muscle’s resistance to passive stretch

37
Q

Describe the progression of tone abnormalities with an UMN injury

A

temporary hypotonia (Acute) > development of spasticity (subacute/chronic)

38
Q

Common UE areas of spasticity

A

Scapula retractors, downward rotators
Shoulder IR, adductors
Elbow flexors
Forearm pronators
Wrist/hand flexors, finger adductors

39
Q

Common LE areas of spasticity

A

Hip adductors, IR, extensors
Knee flexors
Ankle/foot PF, inverters, toe flexors

40
Q

Primitive and tonic reflexes can return in patients with

A

extensive brain damage