CVA Flashcards
RED FLAGS
DIFFERENTIALS
- Hemiplegic migraine
- Hypoglycaemia
- Tumour
- Seizure
- Bell’s palsy
RISK FACTORS
- Age
- Diabetes
- Previous TIA
- Hypertension
- AF
- Smoking
- Alcohol
CAUSE
- Atherosclerosis
- Ruptured aneurism
- Thrombi
- Embolism
TYPES
- Haemorrhagic: due to aneurism, chronic hypertension weakened blood vessel wall rupture. If it occurs in the Subarachnoid space, it is an SAH increase ICP.
- Ischaemic:
Embolic– fat, gas, amniotic fluid occludes the cerebral blood vessels
Thrombus – atherosclerosis can occur anywhere in the vasculature - LDLs entre tissue & are oxidated into soap cells creating plaque reduce the size of the vessel lumen or rupture rupture causes clot formation that can dis/relodge in the cerebral vasculature vessel occlusion hypoxic neurons cell death.
TIA– an ischaemic stroke that resolves within 20min as the occlusion dislodges again & allows the brain tissue to reperfuse.
PATHOPHYSIOLOGY
- Neurons can only survive for 3-5min without O2 Ischaemic neurons Inflammation vascular permeability BBB fluid leaches into tissue.
- Ischaemic neurons calcium influx Glutamate & Ca cascade causes apoptosis to cells surrounding that hypoxic tissue brain death that will expand if hypoxia is not reversed.
MANIFESTATIONS
- Head: ALOC, headache, confusion, memory loss, facial droop, unequal pupils, visual disturbance, dysphagia, dysarthria aspiration.
- Torso: respiratory compromise.
- Limbs: Hemiparesis, paraesthesia.
Stroke symptoms may not resolve or resolve incompletely
TREATMENT
DRAB: O2 (careful not to over oxygenate vasoconstriction). C: IV access. D: Ondansetron (reduce risk of increased ICP). E: Position 45° (increase perfusion + decrease cerebral oedema).
Secondary Survey: Thorough NSA, CVA + reassessments. NHISS-8, modified Rankin score, stroke referral
PARAMEDIC ROLE
Prevent secondary injury. Identify if it’s a LVO in order to Tx to a hospital capable of endovascular clot retrieval. Find time of onset, determine whether the Pt needs thrombolysis or EVCR.
DEFINITIVE CARE