CV Emergencies Lecture Flashcards

1
Q

Pericarditis is pleuritic like central CP that worsens with ___________ and improves with ________

A

Worsens when pt is supine

Imroves with sitting

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2
Q

Pericarditis is aggravated by..

1.
2.
3.

A

Movement

Coughing

Swallowing

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3
Q

MC cause of pericarditis?

A

Viral!

(Coxsackie)

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4
Q

What will you hear during a lung exam in a pt with pericarditis

A

Friction Rub

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5
Q

3 Ps of pericarditis?

A

Position
Palpation
Pleuritic

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6
Q

Duration= hours to days

Quality= sharp

A

Pericarditis

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7
Q
  • *1. Viral**
    2. Uremia
    3. Radiation
    4. Autoimmune
    5. Drug induced
    6. Trauma
    7. Early post MI
    8. Neoplastic
A

causes of pericarditis

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8
Q

True or False..

other, less common causes of pericarditis, include:
TB, acute bacterial infections, fungal

A

True

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9
Q

ECG changes occur within hours of onset of pain

initial changes= diffuse ST segment elevation in ALL leads EXCEPT aVR and VI

later changes= normalization of ST elevation followed by T wave flattening and T wave inversion (this can be days or longer)

A

Pericarditis

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10
Q

All pericarditis pts should get what image, because there may be a small effusion

A

Echo-doppler

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11
Q

Tx of pericarditis..

A

Bedrest
NSAIDs

**avoid oral anticoagulation!

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12
Q

Mortality rate in diagnosed cases = 8%

Mortality rate in undiagnosed cases= 40-50%

A

PE

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13
Q

Risk factors:

Stasis (pregnancy, obesity, varicosity, bed rest, immobilization, surgery, incresed age)
Cardiac D/Os
Hypercoagulability
(OCPs, polycythemia, Factor V Leiden, etc)
Trauma
Chemo
Smoking

A

PE

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14
Q

97% will have at least 1 of the following:

Tachypnea
Dyspnea
Pleuritic CP

A

PE

*1/3 will have tachycardia

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15
Q

ST, NSST-T Changes on ECG

O2 may be decreased; repiratory alkalosis

CXR often normal

D-Dimer is non specific

A

PE

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16
Q

Image of choice for PE?

A

Helical (Spiral) CT Angiography

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17
Q

Pain (deep, visceral, crushing, heavy squeeze) longer than 30 mins
+
diaphoresis

..should be highly suspicious of?

A

MI

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18
Q

Can be caused by….

CAD
Plaque rupture
Occlusive thrombus
No perfusion of effected myocardium

A

MI

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19
Q

Atypical presentation of MIs are commonly seen in….

A

Elderly
Women
Diabetics

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20
Q

If a person is having an MI, is there pulse and BP high or low?

A

Can be either!

*usually depends on what type of MI they are having (location)

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21
Q

The sympathetic NS is stimulated, causing
increased pulse
increased HR

..usually seen in what type of MI?

A

Anterior MI

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22
Q

Parasymp NS is stimulated, casuing a
decreased pulse
decreased BP

..usually seen with what type of MI?

A

Inferior MI

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23
Q

An S3 heart sound during an MI indicates ______ dysfunction

A

LV

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24
Q

Transient MR murmur with an MI indicates _________ dysfunction

A

Papillary muscle

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25
What dose of ASA is typically given to an MI pt?
160-325 mg
26
For a STEMI, \>2mm ST elevation in ________ leads \>1mm ST elevation in _____ leads in 2 adjacent (contiguous) leads
\>2mm in **precordial** leads \>1mm in **limb leads**
27
3 drugs that can be used to treat MI pain
Nitro Morphine Beta blockers
28
dose= 0.4mg SL Q5 minutes (for up to 3 doses)
Nitro
29
Avoid nitro is systolic BP is below...
90 mmHg
30
Very affective drug in anterior MIs Can lead to venous pooling **dose= 2-5 mg IV. repeat if needed** if BP drops, elevate legs and give IVF
Morphine sulfate
31
Cardiac ultrasound/doppler (echocardiogram) should be obtained in an MI pt within...
24 hours \*will show wall motion abnormalities of MI, EF, others.
32
Best way to treat/reperfuse an MI?
Pecutaneous Coronary Intervention (PCI)
33
Ideal goal= to keep ischemic time to under.....
120 mins! (so from onset of symptoms to PCI time)
34
**PCI is more complete in reperfusion of infarct artery and decreasing re-occulusion liklihood** in comparison to what alternative tx method...
Fibrinolytics
35
If PCI is not available, what is MI tx?
Fibrinolytics
36
Start fibrinolysis within _______ minutes of onset of symptoms
30 mins
37
tPA, tenecteplase, reteplase
Fibrinolytic drugs
38
- Hx of cerebrovascular hemorrhage - Stroke within 1 year - Marked HTN (**S\>180, D\>110**) - Active internal bleeding - Recent head trauma - Recent major surgery (**within 3 weeks**) - \>65 yo
Absolute/relative contraindications for fibrinolytics
39
After a thrombolytic is given to an MI pt who needs it, what needs to be done next?
**Full anticoagulation** with unfractionated Heparin or LMW Heparin (goal PTT=50-75 secs)
40
Post PCI stent, what treatment needs to be established?
Long term ASA use
41
If a PCI center is not readily available (artery open in 120 mins), but is reachable by transfer ..what can you give in the mean time?
Fibrinolytics (PCI follows)
42
Risks of giving fibrinolytics before PCI?
If PCI done too early, before thrombolytic wears off, there is an increased risk of bleeding
43
Spontaneous tear in intima of aorta allows blood to dissect into media, separating aortic wall
Aortic Dissection
44
Associated with **long standing, poorly controlled HTN**- repetitive torque to ascending/descending aortic wall
Aortic dissection
45
Which type... Dissection **starts in aortic arch** **proximal to L subclavian artery**
Type A Dissection
46
Which type... Dissection starts in **proximal descending aorta _beyond the subclavian artery_**
Type B Dissection
47
Severe CP often radiating to/down back HTN usually present ECG= LVH common; NSST-T changes; ischemia if coronaries are involved
Aortic dissection
48
Image of choice for an aortic dissection?
Multiplanar CT
49
Must decrease BP ASAP! **target SBP= 100-110 mmHg** Give Beta blockers! (Labetalol, Esmolol)
Aortic dissection tx
50
Target SBP for an aortic dissection pt?
100-110 mmHg
51
What type of dissection DEFINITElY requires surgery?
Type A
52
What type of dissection... Requires urgent surgical repair if there is an arterial branch occlusion or a progressive dissection BUT If pt stabilizes and dissection does NOT invove branch arteries, CAN do surgery or can just medically treat BP and get annual CT scans
Type B dissection
53
How do you distinguish between unstable angina (UA) and a NSTEMI?
NSTEMI will have abnormal cardiac markers (CK, MB, troponins) that indicate cell necrosis \*no cell necrosis in UA
54
If UA is left untreated, what are they are high risk for developing?
MI in the following days/weeks
55
If NSTEMI is left untreated, what can happen?
Progression to larger MI/death
56
ECG abnormal 50% of time: ST depression T wave inversion (**signs of ischemia**)
Unstable angina NSTEMI
57
Pain at rest Prolonged \>10 mins Substernal CP Radiation Nausea, dyspnea, diaphoresis
UA/NSTEMI \*women and elderly present different
58
For a CP pt, you should get an EKG within...
10 mins
59
Serial cardiac markers should be checked every...
6-8 hours
60
\>65 At least 2 risk factors for CHD Prior coronary stenosis ST segment deviation on presenting ECG 2+ anginal episodes in 24 hours ASA in the last 7 days Elevated cardiac markers
TIMI Risk Score (risk factors for adverse events)
61
Why might you see an ECG with low voltage?
Fluid present (ie tamponade with effusion) COPD (increased air surrounding heart) Obesity
62
If troponins and ECG are negative for a CP (possible UA) pt, what should you do within the next 24-48 hours?
Stress test/imaging \*if + for ischemia= refer. possible cath \*if - for ischemia, discharge
63
Tx for NSTEMI/UA pts
**ASA\*\*** Beta blockers UFH,LMW Heparin (CCB can be used 3rd line or alternative to Beta blockers if contraindicated)
64
NSTEMI pt with.... \*Recurren Angina/ischemia \*Elevated troponins \*ST depression \*CHF or EF \<0.40 \*Sustained vtach \*PCI within last 6 mo \*Prior CABG
Should favor **Cath/revascularization**
65
Increase in pericardial fluid, increasing intrapericardial pressure, **resulting in compression of the heart** **\*limitation of ventricular diastolic filling, leading to _decreased stroke volume and cardiac output_**
Cardiac tamponade
66
Marked elevation and equilibration of LV and RV LA and RA pressures elevated **Marked decrease in cardiac output** \***\*\*RA and RV collapse seen on echo**
Cardiac tamponade
67
1. decreased arterial pressure 2. increased systemic venous pressure 3. quiet heart
Beck's Triad **seen with cardiac tamponade**
68
1. Hypotension 2. Distended neck veins 3. Distant heart sounds
Beck's triad (Cardiac Tamponade)
69
Cardiac output is very _____ sensitive
Volume
70
Intrapericardial pressure is markedly increased RV and LV volumes diminshed ## Footnote **inspiration results in a decreased LV volume, resulting in a systolic BP drop \>10 mm**
Pulsus Paradoxus
71
Increased venous return Slight increase RV volume Slight decrease LV volume RV output increases, LV output decreases ## Footnote **minimal drop in systolic BP (2-4 mm)**
what NORMALLY occurs with inspiration **pulsus paradoxus is an exaggeration of this**
72
Pulsus paradoxus can be seen with cardiac tamponade true or false?
true
73
Diagnostic image of choice for cardiac tamponade?
Echocardiogram
74
Tx for cardiac tamponade?
Pericardiocentesis IVF to increase preload
75
Pericardectomy and pericardotomy are necessary in 25% for...
recurrent tamponade
76
Acute MI or severe ischemia Progression of HF Acute HF superimposed on chronic HF Acute volume overload of LV
can all cause **acute pulmonary edema**
77
D/C of meds Excessive Na intake Myocardial ischemia Tachyarrhythmias Intercurrent infection
Precipitating factors of acute pulmonary edema
78
Severe dyspnea\*\*\* +/- pink, frothy sputum Cool extremities, cyanosis Diaphoresis Anxious, restless, unable to breath ## Footnote **JVD\*\*\***
Acute pulmonary edema
79
``` Lungs= diffuse crackles, rales, wheezing Heart= tachy, S3 ``` "butterfly" pattern of alveolar edema on CXR
Acute pulmonary edema
80
Tx for acute pulmonary edema?
O2! Morphine IV diuretics (Furosemide)
81
Drug that helps acute pulmonary edema because.. **venodilator**, decreases PCW/LA pressure Decreases anxiety
Morphine
82
Can be paroxysmal or persistent 95% of the time, is seen with underlying cardiac or pulmonary pathology often called "holiday heart"
A fib
83
Atrial depolarization 400-600/min
A fib
84
If new/untreated, ventricular tachycardia ## Footnote **120-180 bpm**
A fib
85
What is the first priority in an A fib pt?
Examine hemodynamic stability \*most patients will be stable \*\*\*\*if not stable, DC cardioversion | (pulse, BP, symptoms)
86
Risk of thromboemboli in A fib if ongoing for longer than...
48-72 hours
87
If A fib pt is hemodynamcally stable (most are), what is the initial goal and how do we achieve it?
**Rate control!** **with IV diltiazem**
88
If A fib is present for \>48-72 hours, must fully anticoagulate for how long before attempting cardioversion?
3 weeks
89
Cardioversion to NSR + drug rx to maintain NSR \*this increases CO
Rhythm control of a fib
90
Leave in A fib, control ventricular rate + anticoagulate (warfarin)
Rate control of A fib
91
**Ibutilide** can be used for rapid conversion of recent onset...
A fib and A flutter
92
Asymptomatic pt with BP \>220/120 mmHg High BP (\>200/110) with **optic disc edema or progressive target organ (kidneys, heart) complications** Perioperative HTN
Hypertensive Urgencies
93
Warrant BP lowering within a few hours
HTN **urgencies**
94
Warrant substantial BP lowering **within 1 hour** to avoid severe morbidity or death (BP often very elevated..DBP\>130mmHg)
HTN **emergencies**
95
Hypetensive encephalopathy Hypertensive nephropathy Malignant HTN
Hypertensive **emergencies**
96
HA Irritability Confusion Altered mental status (cerebrovascular spasm)
Hypertensive encephalopathy
97
Hematuria Proteinuria Progressive kidney dysfunction (arteriolar necrosis and intimal hyperplasia)
Hypertensive nephropathy
98
Encephalopathy or nephropathy PLUS papilledema! ## Footnote **predisposes to progressive renal dysfunction if not rapidly fixed**
Malignant HTN
99
↑↑BP with: Intracranial hemorrhage Aortic dissection Pre-eclampsia/eclampsia Unstable angina Acute MI
Hypertensive emergencies
100
DO NOT use meds to lower BP unless greater than..
200/100
101
In a HTN emergency, goal is to lower BP by 25% within ___ hours, then more gradual lowering (over 2-6 hours) to a BP ~160/100
2 hours
102
**Nicardipine** and **Clevipine** are two potent CCBs that are used for
HTN emergencies (Nicardipine=1st line)
103
\_\_\_% of abdominal aneurysms are below renal arteries
75
104
Palpable, pulsatile, non tender mass ## Footnote **abdominal ultrasound allowes for accurate dimension measures**
AAA
105
\>60 yo with risk factors for AAA: - family hx of AAA - presence of PAD/atherosclerosis - presence of peripheral artery aneurysms
Screen with ultrasound for AAA
106
AAA \>6.5 cm
SURGERY always!!
107
AAA \>5 cm
probable surgery
108
AAA \>5 cm has a 20-40% rupture chance over...
5 years