CSA 2 Exam Flashcards

1
Q

General Tenderness

A

Caused by I’s & T’s

  • inflammation
  • infection
  • tumor
  • trauma
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2
Q

Tenderpoints

A

a specific point which exhibits acute tenderness when probed

Looking for changes in

  • fascia
  • muscle tendrils
  • CT
  • nerve fibers
  • changes in vascular elements
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3
Q

Tender points are…

A

a sign of somatic dysfunction

we do not treat tender points

we treat somatic dysfunction

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4
Q

Treatment of Somatic Dysfunction

diagnosed by tender points

A

Treat

  • most tender one
  • if more than one, treat most central one
  • treat most significant first
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5
Q

what initiates somatic dysfunction?

A

muscle spindle reflex

nociceptors

nocifensive reflexes

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6
Q

why and how is somatic dysfunction maintained?

A

guarding mechanism to protect from further damage

at spinal cord level or at higher CNS levels

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7
Q

Who described strain/counterstain?

A

Lawrence Jones DO

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8
Q

What is counterstrain?

A

a passive positional OMT that places a segment in a position of comfort

an indirect treatment to restore/reset nociceptive and proprioceptive activity

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9
Q

When was counterstrain described?

A

1955 by Lawrence Jones

published in 1964 in JAOA as “Spontaneous Release by Positioning”

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10
Q

What is the main diagnostic criteria for counterstrain?

A

Tenderness

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11
Q

What is the minimum resolution we want?

A

70%

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12
Q

Why do we return the patient back to neutral slowly?

A

to prevent re-inciting the nociceptive reflex

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13
Q

What happens during the 90 secs treat?

A

holding the muscle in a shortened position decreases the proprioceptive and mechanoreceptive input

allows CNS to reset normal resting length and tension parameters

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14
Q

Advantages of Counterstrain

A

non-traumatic

easy, safe, effective

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15
Q

Disadvantages

A

takes time

treatment reactions lasting up to 24-36 hours

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16
Q

What is strain?

A

severe over-stretching of a muscle

stretched muscle has increased freq of impulse

17
Q

What are tensegrity systems stabilized by?

A

Tensegrity systems are stabilized by

continous tension (soft tissue) with discontinuous compression (bone)

18
Q

Mechanotransduction

A

the process of mechanical signals being converted into biochemical changes

19
Q

Prestressed tensegrity structures are…

A

a series of discontinuous compression-resistant elements held w.i. a web of continuous tension elements

20
Q

Geodesic tensegrity structures are…

How do they differ from prestressed tensegrity structures?

A

tensegrity structures stabilized through force triangulation

individual elements are capable of alternating btw generating tension or resisting compression

21
Q

Tenets of tensegrity structures

A
  • intrinsically self-stabilizing
  • flexible while minimizing damage
  • return to original shape (elastic)
  • efficient, lightweight, + strong
22
Q

Who researched biotensegrity at the cellular level?

A

Ingber

23
Q

Who researched tensegrity?

A

Fuller

24
Q

In vitro vs in vivo study

A

in vitro

  • microfilaments = bent
  • microtubules = straight

in vivo

  • microfilaments = straight
  • microtubules = bent
25
Q

Outcomes of mechanotransduction are…

A
  • modifications in cell physiology
    • activate cAMP production
  • modifications in gene expression
    • activation through deformation of microfilaments + microtubules
      • chromatin remodeling + nuclear pore open/closing
  • modifications in intracellular bchm
    • activation of Ca ion channels (TRPV4)
      • alters focal adhesion