Credit 3 Extra Flashcards

1
Q

Describe ventricular tachycardia and it’s ECG changes

A

Impulses are repetitively generated from one or more ventricular foci.
Ventricular tachycardias differ in morphology, rate, regularity, and duration.

The P waves are normal, have no fixed relationship to wide and bizzare QRS complexes.
- They occur before, during, or after the QRS complex, but have no fixed relationship = P wave differ from beat to beat.

Ventricular tachycardias can be:

  • Uniform: All QRS complexes are of one, there is one focus from which all QRS complexes originate
  • Multiform: Called multifocal. There are different sites of origin
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2
Q

What is typical for multifocal premature ventricular complexes?

A

For example from lead II

  • a single premature ventricular beat occurs
  • then two normal beats.
  • and finally 3 premature beats in a row.

When these 3 premature beats occur in succession it is called a run and now having a different configuration, meaning morphology is different.

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3
Q

Describe ventricular fibrillation and it’s ECG changes

A

Characteristic are rapid irregular, unorganized deflections on the ECG.

Changes on the ECG:
- P waves, QRS complexes, and T waves are not detectable.
There are continuous positive and negative oscillations that are chaotic and bizzare activity of heart.

Coarse fibrillation – a deflection of high amplitude than fine fibrillation has.

A terminal rhytm resulting in NO ventricular contractions causing cardiac arrest.

Triggers may be
- myocardial ischaemia, hypothermia and drugs

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4
Q

Define and characterize right bundle branch block

A

Characteristics:
A delay in conduction to the right ventricle leading to that the right ventricle will continue to be depolarized.

Changes on the ECG tracings:

  • Decrease the size of the R wave in leads I,II, III and AVF
  • Large and wide S waves in leads I,II,III and aVF
  • Cause of a prolonged conduction time the QRS complex is wider than normal
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5
Q

Define and characterize left bunde branch block

A

Characteristics:
Delayed depolarization of the left ventricle

Changes on the ECG tracings:

  • Wider QRS complex
  • An increase in the R wave height in the lead II
  • The T wave is large and opposite in polarity to the QRS complex
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6
Q

Mechanism of development of myopathies caused by ischaemic imbalances

A

Downer Cow syndrome
Also called
- Maternal obstetric paralysis
- Obturator paralysis

Occurs often in the first 2 - 3 days after calving, in heavy milk producers.

Characterized by

  • Inability to stand
  • Affected hindlimbs are commonly directed behind the cow in a frog-leg attitude.

Due to:

  • Unsifficient blood supply to affected areas of muscles causing them to undero athropy and later necrosis
  • Traumatic injuries: during parturition due to slipping or forced to stand up too quick
  • Difficult parturition due to eg. an oversized calf
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7
Q

What are typical for AV blocks?

A

Characteristics:
The normal sequence of cardiac activation includes slowed conduction through the AV node, allowing sequential activation of the atria and then ventricles.

Conditions:
enhanced vagal tone, drugs, structural diseases of the AV node
Altered intraatrial conduction + altered conduction in both bundle branches simultaneously result in
Classified into:
First-, second-, and third degree AV blocks

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8
Q

Describe Premature atrial complexes

A

Occurs when an ectopic site depolarizes at a rate faster than the sinus node.

Rare in dogs and cats but can be associated with

  • Atrial enlargement
  • Acid/base or electrolyte abnormalities
  • Hypoxia

Causes changes on ECG;

  • A normal appearing QRS complex but that occurs too early (premature)
  • An abnormal P wave
  • P - P duration is highly variable depending on origin of the ectopic impulse
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9
Q

Describe sinus arrhytmia

A

The rhytm originate from the sinus node but the rhytm is irregular

Defined as rhytmic speeding and slowing of the rhythm so that the variability of the R-R interal is greater than 10%

A wandering pacemaker; P wave is varying in shape and is biphasic or will reverse its polarity.

A chaning heart rate accompanied by shifting the pacemaker site within the SA node and / or atrium

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10
Q

What is respiratory sinus arrhytmia?

A

When the sinus arrhytmia is linked to the respiratory cycle, when the rate increase with inspiration and slows on expiration

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11
Q

Describe sinus block

A

When the impulse is blocked before exciting the SA node

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12
Q

What are the types of abnormalities in impulse conduction (conduction disturbances)?

A
  • Atrioventricular blocks (AV blocks)

- Bundle branch blocks

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