Craniofacial growth, Developing dentition Flashcards
What’s the incidence of ankylosis in primary dentition?
7 – 14% in primary dentition
Most often affects lower Ds, followed by lower Es, upper Ds, upper Es
associated with agenesis of succadaneous tooth
multiple teeth seen as frequently as single
Does early correction of unilateral posterior crossbite eliminate morphological and positional asymmetries of the mandible?
yes
What # of weeks define the embryonic period?
The first 8 weeks of life.
What is the most favorable eruption sequence in max permanent dentition?
61245378
612 regular
want 4s to come in (1st PM)
Then 5s (2nd PM)
Then 3s (canines)
Last 7s (so that 3s can take up leeway space)
Name 6 pharyngeal fistulae, cysts, and tumors associated with branchial arch malformation
- Thyroglossal duct cyst/ectopic thyroid (2nd pouch)
- Hemangiomas
- Thymic anomalies (e.g. DiGeorge)
- SCM tumor of infancy (sternocleidomastoid)
- Cervical teratoma/dermoid cyst and midline cervical cleft
- Lymphangioma/cystic hygroma
What is hypertrophy?
Increase in size of individual cells
What radiographs will best locate a supernumerary tooth?
2 PAs or occlusal films reviewed by the parallax rule
also: two PAs either using two projections taken at right angles to one another or the tube shift technique (buccal object rule or Clark’s rule) or by CBCT
What are the 6 stages of histodifferentiation?
- Initiation
- Proliferation
- Histodifferentiation
- Morphodifferentiation
- Apposition
- Mineralization and Maturation
What are the mechanisms of formation for intramembranous bone formation?
Periosteum and sutures, generally in areas of tension
no cartilagenous precursors
If a patient has crowded primary dentition, what % of the time will this patient have crowding in the permanent dentition?
100%
Moyer’s Analysis
Measures the M-D width of the mandibular incisors
Maxillary tooth predicted by mandibular teeth
Tanaka and Johnson also uses m-d width of lower incisor: Y = A + B (X) where
Y is sum of m-d widths of unerupted canines & PMs
X is sum of m-d widths of lower incisors
A and B are constants
Y = 11 + 0.5X for maxillary arch Y = 10.5 + 0.5X for mandibular arch
SNB average?
80˚
>80 then prog
<80 then retro
What is a low birth weight?
2,500 g at birth (5.5 lbs)
Where are most supernumerary teeth located?
Maxilla – 80-90%, ½ of that in the anterior area
Most common site – mesiodens
Second most common – paramolar (max. molar)
25% of mesiodens erupt spontaneously
Calcification of the permanent teeth
Will a permanent molar w/ part of its occlusal surface clinically visible and part under the distal of the second primary molar “jump” and self-correct?
NO
This is the impacted type, not the self-correcting type.
After the age of 7, definitive tx is indicated to manage and/or avoid early loss of the primary second molar and space loss
What is considered a full termbirth?
37-41 wks of completed gestation
T/F The majority of pts have Class I malocclusion.
T. 53%
Class II – 32%
Class III – 14 %
Open bite 5%
deep bite 10%
severe overjet >6mm 15%
29% have malocclusion where tx is highly desirable or mandatory (6.5 mill)
What arch has the stapes, styloid process, and stylohyoid ligament?
2 – Hyoid arch.
Also has lesser cornu/upper portion of body of hyoid, posterior digastric and muscles of facial expression
CN 7
What do the mandibular processes merge to create?
Lower lip and mandible
Systemic conditions implicated in delayed primary exfoliation and permanent eruption
- Vit D resistant rickets
- Endocrine disorders
- CP
- Celiac
- Prematurity/ low birth weight
- diabetes
- Genetic disorders (there are 26! Including OI, Cleidocr dyspl, ED, Chondroectod D, Albright’s, Gorlin, MPS, Gardner, Down, Apert, Achondroplasia, Cherubism and more)
What arch develops into muscles of mastication?
1st arch, also mylohyoid, anterior digastric, tensor palatini, tensor tympani
CN5 – Trigeminal nerve
What are some rules of thumb for timing of primary tooth loss affecting successor eruption?
Loss of 1˚ tth before age 5 => delays premolar
Loss of 1˚ tth after age 8 => accelerates premolar
Loss prior to completion of crown of successor → delays
Loss after crown completion → accelerates eruption
Which branchial arch has the most frequent anomalies?
2nd (95%)
1st arch only has 1% of all branchial anomalies
T/F:
Baume Type I is non-spaced dentition.
False
Baume Type I – generalized spacing of primary dentition (2/3)
Baume Type II – non-spaced (1/3)
Bilateral posterior crossbites in children (primary teeth)
Manifestation of a true skeletal constriction
Associated with dolicocephalic growth, open bite
Midlines are symmetric, no notable shift of mandible
2-3% of posterior crossbites
Treated with expansion
If a patient has no spacing in the primary dentition, what % of the time will this patient have crowding in the permanent dentition?
66%
What branchial arch is the major contributor to the facial structures?
1st arch mainly
some contribution from the 2nd
Ectopic eruption of permanent molars occurs in up to ___% of the population
3%
new guideline
more common in children with cleft lip and palate (25% of kids with CL/P)
Interference with cranial vault growth leads to ___________
Craniosynostosis
“copper-beaten skull”
e.g. Apert’s and Crouzon’s syndromes
What 3 ways do you make up for incisor liability?
- Intercanine width development
- Facial placement of lower anteriors
- primary spacing
If a pt has 3-6mm spacing in the primary dentition, what % of the time will the pt have crowding?
20%
Space regaining appliances
Fixed: active holding arches, pendulum appliances, Jones jig
Removable: Hawley appliance with springs, lip bumper, and headgear
What is development?
Increase in complexity
1st branchial pouch anomalies
Atretic eustachian tube → recurrent OM
eustachian tube diverticuli
absence of tympanic cavity, mastoid antrum
perforated tympanic membrane
bifid tongue
If you cannot palpate the canine bulge and there is radiographic overlapping of the permanent canine with the formed root of the lateral during mixed dentition, what should you do?
EXT the primary canine
Functional Matrix theory
- Functional demands of the craniofacial complex controls growth
- influence of “capsular matrices”
- Moss and Salentijn
- says that bones don’t grow but are grown: soft tissue grows – bone and cartilage respond
What is the precursor for the mandible?
Meckel cartilage
1st arch also has malleus, incus, sphenomandibular ligament
What is a normal nasolabial angle?
100-110˚
T/F
Undermining resorption happens with light, continuous forces on the pdl.
Heavy forces result in hyalinization.
F light forces result in “frontal resorption”
T. heavy forces result in “undermining resorption” and pdl become hyalinized because blood supply to the pdl becomes occluded
Are most children mesial, flush, or distal step?
Mesial 60%
flush 30%
distal 10%
(source??)
Handbook says
mesial 15% “incidence”
flush 75% “incidence”
distal no percentage given
What are the lateral lip and maxilla derived from?
Fusion of median nasal and maxillary processes
What percentage of primary supernumeraries have permanent supernumeraries?
1/3 of cases
What two points make Frankfurt Horizontal?
Po- Or
Porion-Orbitale