Cranial Introduction Anatomy and General Movement Flashcards
who is the founder of cranial osteopathy?
William Garner Sutherland
history of cranial osteopathy
1931
1939
1943
1944
1947
1953
2013
- people though the head did not move
1931: introduction of osteopathic cranial manipulative medicine concepts (blunt bone bill)
1939: techniques to treat cranial dysfunction (the cranial bowl)
1943: presented at osteopathic convention
1944: publishe din JAOA
1947: osteopathic cranial academy estab;ished by his students to provide greater knowledge and research
1953: sutherland cranial teaching foundation
2013: glymphatics was discovered
what are the two definitions of cranial manipulative medicine
- diagnosis and treatment by an osteopathic practitioner using primary respiratory mechanism and balanced ligamentous tension
- system og diagnosis and treatment first described by william G. southerland
prior to sutherland the cranium was considered?
fused with no capacity for movement or dysfunction
what are the 5 anatomical-physiological elements of primary respiratory motion?
there is motility of the brain and spinal cord
the csf fluctuates
the intracranial and intraspinal membranes move
the cranial bones move
the movement between the sacrum and ilium depend on the motion at the SBS
what does “primary” in primary respiratory mechanism mean
cranial motion is a fundamental life process that is it controls all other processes like cellular and lung respiration
what does “respiratory” in primary respiratory mechanism mean
cranial motion has an ebb and flow like the breath (the breath of life)
what does mechanism in primary respiratory mechanism mean
pattern of tissue and fluid movement occur with a purpose
indications for craniosacral treatment
stresses affect the PRM
after birth
trauma (suture overlap)- orthodontics/posture/car accident
dentistry (holding jaw open)
toricolis
plagiocephaly (flat head)
feeding difficulties (compression of cranial nerves 9,10,11)
vertigo
concussion
headache
tmj
tinnitus
mechanical stresses of birth and dural membrance occur during delivery, treatment facilities _ remodeling
persistant suture overlap may result in the bones growing together forming a _
vault
synostosis
side effects of cranial OMM
headache, tinnitus, dizziness
SBS strain treatment may cause an alteration to?
heart rate, blood pressure, respiration, and GI tract
absolute contraindications for cranial OMM
acute (24hr) intracranial bleed of CVA
acute skull fracture
relative contraindications for cranial OMM
coagulopathy
seizure history
increased intracranial pressure
**exacerbation of neurologic condition
in the inherent motility of the brain and spinal cord what is the contractile element in the brain
the glial cells which are mobile (have the capacity of moving spontaenously)
csf is produced in the
choroid plexus which is in the ventricles
pumped across the brain and spinal cord
what is the driving force of CSF expansion/movement
cranial ryhtmic impulse (CRI)
what is the rate of CRI
10-14
motion characteristics of the CRI
RRADS
rate: 10-14 bpm
rhytmn: regular/tide of an ocean with some variations
amplitude: amount of movement (mm of movement)- somatic dysfunction may have decreased amplitude
direction: linear and symmetric, in SD it will be assymmetric
strength: good strong motion? does it have vitality, is it weak?
the contiguous dural folds become the _, _ , and _
this gives the mobility of the intracranial membranes
falx cerebri
falx cerebelli
tentorium cerebelli
(inside is the venous sinus)
the tentorium and the falx create a _ _ _
reciprocal tension membrane
what is the reciprocal tension membrane (RTM)
this is a functioning unit that holds the bones of the vault and base uunder constant tension
dura is so tough
the RTM allows but limits _
motion
the RTM acts as a _ storing energy in _ and releasing in _
spring
flexion
extension