Coursecram Flashcards

1
Q

what is the endocrine system?

A
  • all the hormone secreting cells of an organism
  • composed of endorcrine and neuroendocrine glands
  • these glands secrete their hormones and neurohormones into body fluids (blood, hemolymph, etc. )
  • think “within the body”
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2
Q

what is histamine?

A
  • local regulator
  • regulation of immune response and and inflammation
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3
Q

what are local regulators

A

signals that act like hormones but never enter any circulatory system

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4
Q

what are pheromones

A

they are signals that leave the organism and act at a distance on another organism

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5
Q

what are the 3 signalling strategies?

A
  • autocrine (self signal)
  • paracrine (signal beside it, histamine)
  • endocrine (within)
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6
Q

what are the four groups of hormones?

A
  • amino acid derivatives: dopamine, melatonin, thyroid hormones, adrenaline
  • peptide hormones: proteins, prolactin,insulin
  • steroid: cholestoral derivatives, sex hormones, cortisol
  • fatty acid derivatives: in insects
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7
Q

what type of soluble are steroid hormones?

A

lipid soluble

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8
Q

what are the unique properties and receptor location of peptide hormones?

A
  • encoded by genes
  • soluble in blood
  • receptors found on the plasma membrane
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9
Q

what are the unique properties and receptor location of steroid hormones?

A
  • insoluble in blood
  • need carrier proteins
  • made by enzymes
  • receptors are found in the cytoplasm
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10
Q

what are the steps in the endocrine signalling pathways?

A
  • synthesis: translation and enzymatic
  • transport: either soluble or insoluble (need carrier proteins)
  • reception: only cells with the appropriate receptor expressed can respond
  • transduction
  • response
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11
Q

what are the 2 types a receptor can be?

A
  • angonists: stimulatory
  • antagonist: inhibitory
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12
Q

what are the 2 general kinds a receptor can be?

A
  • angonists: stimulatory
  • antagonist: inhibitory
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13
Q

what can hormone concentration affect for receptors?

A

activity

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14
Q

what are peptide hormones synthesized by?

A

genes that are expressed

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15
Q

what are steroid hormones synthesized by?

A

enzymes

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16
Q

which can travel through the blood stream? soluble or insoluble hormones

A
  • soluble: can travel
  • insoluble: bind carrier proteins in the blood (Albumin and Globulins)
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17
Q

what are signal transduction pathways?

A
  • extracellular signals that produce one or more effects in structure, function, of the gene expression profile of the target cell
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18
Q

what are the types of receptors? what are the types thar use second messengers?

A

use 2nd messengers
* GCPR
* Ligand gated ion channels
* receptor enzyme

don’t use it
* intracellular receptor (influences gene expression- steroids)

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19
Q

what do second messengers do?

A

amplify response and most signalling cascades use them

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20
Q

what are protein kinases?

A

act on each other and transmit signals by a casecase of phorphorylations that cause conformational change in phosphorylated protein

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21
Q

what does phosphorylation usually change in a protein?

A

changes it from inactive to active form

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22
Q

what are the antagnostic hormones in the human endocrine system? what is their general roles

A
  • general role: maintain homeostasis

Regulation of Glucose
* insulin: decreases blood sugar levels by stimulating uptake at the tissues
* glucagon: increases blood sugar levels by stimulating glucose released by the liver into the blood

Regulation of Sodium Levels
* ADH: less water in urine
* ANP: more sodium in urine
* alsosterone: more sodium in urine

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23
Q

what happens when you eat, in terms of insulin?

A
  • blood sugar spikes
  • insulin released from pancres
  • causes glucose to be taken out of the blood (by tissues) and stuffed into the cells
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24
Q

what happens when you fast, in terms of glucagon?

A
  • blood sugar is depelted
  • glucagon released
  • releases glucose from liver and into the blood
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25
Q

what does increased sodium levels cause?

A
  • stimulates hypothalamus
  • which stimulates the posterior pituitary
  • which release ADH
  • ADH causes for more water to be abosrbed, and for there to be less water in urine
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26
Q

what does the hypothalamus do?

A
  • stimulate release of hormones into the blood
  • secretes tropic hormones
  • stimulates the posterior pituitary
  • pupper master
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27
Q

what is the anterior pituitary controlled by?

A

tropic hormones

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28
Q

describe the pathway of cortisol.

A
  • hypothalamus releases CRH which is a tropic hormone which stimulates the release of other hormones
  • the anterior pituitary then releases ACTH to the adrenal cortex
  • this then releases cortisol: stress
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29
Q

what is phytoestrogens?

A

plants posses estrongen like compounds, which can cause for vertebrates to become feminized due to too much exposure from phytoestrogens

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30
Q

what are the 4 functions of the nervous system

A
  • recieve external and internal signals
  • transmit external and internal signals
  • integrate/interpret all signals
  • coordinate/command an action/response to those signals
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31
Q

what are the two components of the nervous system and their function?

A
  • CNS: brain and nerve cord
  • PNS: all neurons and their projections outside the CNS
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32
Q

what is integration carried out by?

A

interneurons

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33
Q

what is the general structure of a neuron?

A
  • dendrites: recieve inputs from other neurons or the environment
  • nucleus
  • cell body/soma: has nucleus and organelles
  • nodes of ranveir: location of action potential
  • axon: transmit impulses
  • myelin sheath: allow saltatory/jumping conductions
  • axon termini
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34
Q

what is a nerve tract?

A

a bundle of axons

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35
Q

what is a ganglion

A

a collection of neuron cell bodies

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36
Q

what is the myelin sheaths role, and what is it formed by?

A
  • formed by glial cells
  • acts as insulating material
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37
Q

where does the action potential run down and go?

A

runs down the azon and then it has to be relayed off to the next cells, when it reaches the axon termini

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38
Q

how are the glial cells subdivided? what is each of their functions?

A
  • microglia: defense and savenging of the immune cells of the CNS
  • astrocytes: maintenance and support
  • oligiodendrocytes: insulating of neurons for electrical transmission, outside of the CNS called schwan cells, form the myelin sheath
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39
Q

is a reflex arc voluntary or involuntary?

A

involuntary

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40
Q

what is an effernt neuron

A

motor neuron

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41
Q

what were the steps in evolution related to the nervous system?

A
  • more nerves and nerve bundles
  • concentration of nerves (ganglia)- cell bodies organized
  • specialization of function of cells
  • more complex synaptic contacts
  • trend toward cephalization (creating a brain)
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42
Q

what is cephalization?

A

tendency toward bundles of nerves around the head

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43
Q

what is the hindbrain

A
  • survive
  • central pattern generator (stores the pattern of neuronal connections)
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44
Q

what is the midbrain?

A
  • router
  • conector to forebrain
    vision and hearing
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45
Q

what is the forebrain?

A
  • decision maker
  • center for integration of complex pathways
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46
Q

how is the PNS seperated, and what are their roles?

A
  • sympathetic: fight or flight
  • parasympathetic: rest and digest
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47
Q

what is the common neurotransmitter of the PNS?

A

adrenaline

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48
Q

what are the differences in duration for hormonal vs neural stimulation of fight or flight?

A
  • neural stimulation of fight or flight is immediate and short
  • hormonal stimulation of fight or flight is a longer lasting response
    *
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49
Q

what is the resting membrane potential in resting neurons?

A

-70mV

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50
Q

is there more sodium or potassium inside or outside of the cell?

A
  • more sodium outside
  • more potassium inside
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51
Q

what is an action potential?

A
  • if the stimulus is strong enough the neuron fires a nerve impulse, which is an action potential
  • action potentials move along the axon, to cell body, to synaptic termini
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52
Q

explain the steps of an action potential.

A
  1. reach threshold: due to small depolorizations
  2. rapid depolarization: fast opening of sodium channels open
  3. repolarization beginds: small opening of potasssium channels open
  4. repolorization continues
  5. hyperpolarizations: potassium channels are slow to close, so too much flows out, making it very negative
  6. resting potential restored: sodium/potassium pump is always on
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53
Q

what does the speed of action potential conduction depend on?

A
  • axon diameter: broader is faster, due to less resistance (in bigger one)
  • myelination: myelination is faster
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54
Q

what are synapses?

A

junctions that control the communication between a neuron and another cell (neuron or not)

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55
Q

what is the transmitting cell called?

A

presynaptic cell

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56
Q

what is a recieving cell called

A

the post synaptic celll

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57
Q

what is acetylcholine?

A
  • type of neurotransmitter
  • one of the most widepsread
  • released at neuromuscular junctions
  • excitatory in brain and skeletal muscles
  • inhibitory in cardiac muscles
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58
Q

what happens when the action potential reaches the synaptic termini?

A

releases chemical messengers (neurotransmitters)

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59
Q

what do neurotransmitters do with an action potential?

A
  • bind receptors in the post synaptic cell membrane and trigger other changes (open ion channels, initiaye other signalling changes)
  • after activating their receptors, neuro transmitters are rapidly degraded or re abosorbed by pre synaptic cell (limits duration of stimulus)
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60
Q

describe what happens in the pre and post synaptic cell?

A
  1. AP opens voltage gated calcium channels
  2. calcium influx triggers vesicles docking (exocytosis event) and neurotransmitter is released in synapse
  3. neuro trans bind to post synaptic cell at receptor and these initate a response (inhibitory or excitory)
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61
Q

what can neurotransmitter signalling be stopped by?

A

degredation at the synapse or reuptake

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62
Q

what is sumation?

A

the integration of both stimulatory and inhibitory stimuli

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63
Q

what does a NT cause at an excitatory synapse?

A

a slight depolarization of the membrane and is called an excitatory post synaptic potential (EPSP)

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64
Q

what does a NT cause at an inhibitory synapse?

A

a hyper polarization of the membrane and it is called an inhibitory post synaptic potrntial (IPSP)

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65
Q

what determines if a post synaptic neuron will fire an AP>

A

the summation of all the EPSP and IPSP that reach the axon hillock at any time

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66
Q

what is the axon hillock

A

where the axon originates and where the voltage gated sodium channels are

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67
Q

what does IPSP cause?

A

repolarize cell slightly, make more neg

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68
Q

what does EPSP cause?

A

depolarize cell slightly, make more pos

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69
Q

what is learning?

A

the process of acquiring new info, and making new connection

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70
Q

what is memory?

A

process of storing info in stable neuronal connections, maintaining connections

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71
Q

what is plasticity?

A

ability to change synaptic connections and function properties of neurons

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72
Q

how does plasticity strengthen the communications between two neurons?

A

making more connections or reinforcing existing connections

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73
Q

what is the hot pepper receptor type?

A

heat receptor

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74
Q

what is muscle tissue responsible for?

A

the generation of force and motion essential for many life purposes including: locomotion, circultion, digestion and the manipulation of objects

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75
Q

what is the most abundant tissue in the vertebrae body?

A

muscles

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76
Q

what is the organization of muscles?

A
  • tissue is arranged into bundles of muscle fibres
  • muscle fibres are comprised of thinner threadlike structures caled myofibrils
  • myofibrils are made up of individual actin (thin) and myosin filaments (thick)
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77
Q

what are the main proteins responsible for contraction of muscle fibers?

A

actin and myosin

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78
Q

how is force generated?

A
  • when myosin pulls the actin polymer which causes the thick filament to slide over the thin filament
  • a cross bridge forms between actin and myosin
  • AP is hydrolysed, releasing enegy to the myosin head
  • the resulting molecular changes in myosin cause the powerstroke
  • interacting is described by slidng filament model
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79
Q

what is actin and what is myosin (think in terms of common function. symbol)

A

actin is the cable
myosin is the engine

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80
Q

what type of protein is myosin?

A
  • motor protein
81
Q

what is a motor protein?

A

protein that changes the shape with ATP hydrolysis to generate force

82
Q

what are the roles of tropomyosin and troponin?

A
  • tropomyosin: wraps around actin and blocks myosin binding sites
  • troponin: regulates tropomyosin
83
Q

what does contraction pull on?

A
  • contraction pulls the actin
  • pulls the thin filaments to the center of the sacromere
84
Q

how are thick and thin filaments arranged

A

in sacromeres

85
Q

what type of pairs do muscles usually come in?

A

antagonistic pairs

86
Q

what does contraction cause for the sacromere?

A

shortening

87
Q

what are the 3 types of muscle conditioning?

A
  • atrophy: decrease in the number of myofibrils per fiber
  • strength training: increase myofibrils per fiber
  • endurance training: increase blood vessels and mitochondria in fibers
88
Q

what are the 3 types of locomotion?

A
  • hydrostatic: muscles and fluid pressure
  • exoskeleton: extenal structure
  • endoskeleton: internal
89
Q

what are the 2 types of muscles?

A

smooth and straited

90
Q

how can straited muscle be further divided?

A

cardiac and skeletal

91
Q

what happens at the muscular junction?

A
  • axons divide into terminals containing acetylcholine
  • regions of muscle under axon terminal is folded into junctional folds to increase SA
  • ach receptor is ligand gated ion channel
  • calcium flow into muscle leading to the depolarization and an AP
92
Q

describe the motor analogy of muscle contraction

A
  • myosin is the motor, converts ATP energy into movement
  • actin filaments are the tracks
  • ATP is the fuel
93
Q

what are autotrophs? Heterotrophs? it is how an organism obtains _____?

A
  • obtains carbon
  • auto: utilize CO2 as carbon source, carry out carbon fixation
  • Hetero: cannot fix carbon, and must synthesize carbon compounds using organic molecules created by other organisms
94
Q

what are phototrophs? what are chemotrophs? this is how an organism obtains __?

A
  • obtains energy
  • photo: use light as energy source
  • chemo: obtain energu from organic or inorganic molecules, usually via redox reactions
95
Q

animals are chemo or phototrophs?

A

chemo

96
Q

what are the exceptions of animals that need to eat food?

A
  • corals: have endosymbiotic relationship with microbiomes, don’t do lots of their own digestions
  • nudibranch: feed on cnidarians and eat using stining cells
  • riftia: harvest bags of bacteria
  • Tape worms: don’t have gut, exterior surface absorbs food
97
Q

what compartments does the digestive system contain?

A

fluid compartments: intracellular, extracellular

98
Q

what is movement between the fluid compartments of digestive system?

A

movement occurs through passive diffusion, facilitated diffusion, or active transport

99
Q

what is intracellular digestion?

A
  • phagocytosis
  • often in unicellular eukaryotes
  • used by protists to engulf solid particules by the cell membrane to form internal food vacuole called phagosome
  • form of endocytosis
  • involved in obtaining nutrients
  • can be used to remove pathogens and dead cells in immune system
100
Q

what is the problem with intracellular digestions?

A

there is an increased chance of infection, therefore there was selective pressure against intracellular digestion

101
Q

what are the benefits and tradeoffs of extracellular digestion

A
  • digestive enzymes secreted into the environment

Benefit: decreased risk of infection
Tradeoff: nutrients can wash away

102
Q

what is the benefit and tradeoff for blind sacs?

A

benefit: allowed continous contact with nutrients, not washed away

tradeoff: nutrients and waste were getting mixed up

103
Q

what is the benefit and tradeoff for unidirectionality digestion?

A
  • mouth and anus different holes
  • earliest example of specialization

benefit: allows step wise digestion, digest more complex nutrients
tradeoff: requires specialization

104
Q

what does chewing allow for?

A

increased surface area for digestion

105
Q

what is the alimentary canal?

A
  • contains openings at both ends
  • anterior end: ingestion
  • middle portion: storage and initial digestion
106
Q

what does the complete digestive system have?

A

regional specialization

107
Q

describe the parts of the stomach.

A
  • stomach: muscular, stretchable sac, mixes and stores ingested food, control passage of food in small intestine
  • gastric juice: dissolve food into liquid mixture
  • protection: pepsin
  • goblet cells: line the stomach and secrete mucous layer for protection
108
Q

what are the cells of the gastric pit?

A
  • goblet cells: secrete mucous to protect stomach
  • cell X: parietal cells that secrete acid
  • chief cells: secrete inactive form of pepsin to digest proteins
109
Q

what is the major site for chemical digestion?

A

duodenum

110
Q

what does increased SA allow for digestion?

A

slow process of digestion of complex nutrients and absorption

111
Q

what is an enterocyte?

A

interstinal cell

112
Q

where are proteins are sugars aborbed into?

A

directly into the blood

113
Q

what are the four compartments of the ruminant’s stomach?

A
  • rumen: where processed mechanically and exposed to bacteria to break down cellulose
  • reticulum: allows the animal to regurgitate and reprocess particulate matter
  • omasum: further mechanical processing
  • abosmassum: the true stomach, where digestive enzymes break down bacter as to release nutrients
113
Q

what are the four compartments of the ruminant’s stomach?

A
  • rumen: where processed mechanically and exposed to bacteria to break down cellulose
  • reticulum: allows the animal to regurgitate and reprocess particulate matter
  • omasum: further mechanical processing
  • abosmassum: the true stomach, where digestive enzymes break down bacter as to release nutrients
114
Q

what is the ruminat’s stomach

A

contain enterosymbiotic bacteria that live in the rumen and can digest cellulose

115
Q

what are the 3 types of feeders?

A
  • continous feeders
  • opportunistic feeders: eat when food is availble
  • metabolic suppressors: can temporarily reduce metabolic rate and body temp (poikilotherms)
116
Q

what is a gastrovascular cavity?

A

one opening (mouth) and branches that reach all the cells of the inner layer, cells digest food, nutrients diffusue into outer layer of cells

117
Q

what are open circulatory systems?

A

no distinction between blood and in the interstatial fluid

118
Q

what is a closed circulatory system?

A
  • cardiovascular system
  • blood is confined to vessels and is seperated from tissues
  • blood flow may be seperated into pulmonary (right side) and systemic circuit (left side)
  • general components: heart, bessels, artieries, capillaries
119
Q

what is the problem with an open circulatory system?

A

as body size increases, it becomes harder to distribute nutrients

120
Q

what is the lymphatic system?

A

open system that collects overflow fluid from the capillaries back to the venous system

121
Q

why are some vessels blue and some are red?

A

The arteries (red) carry oxygen and nutrients away from your heart, to your body’s tissues. The veins (blue) take oxygen-poor blood back to the heart.

122
Q

what is the pacemaker? what is its role?

A
  • a group of pacemaker cells
  • modified cardiomyocytes that have a leaky sodium channel, which causes it to underho a regular slow depolarization
  • action potential spreads through gap junctions across ventricles of the heart, allows coordinated contraction
123
Q

what is the pacemaker? what is its role?

A
  • a group of pacemaker cells
  • modified cardiomyocytes that have a leaky sodium channel, which causes it to underho a regular slow depolarization
  • action potential spreads through gap junctions across ventricles of the heart, allows coordinated contraction
124
Q

simply describe arteries, capillaries, veins, roles

A

artieries: blood away from the heart
capillaries: exchange
veins: blood to the heart

125
Q

what is velocity in the veins caused by?

A

muscle contractions

126
Q

what are erythrocytes?

A
  • red blood cells
  • transport oxygen
  • oxygen carrying capacity is determined by the number of red blood cells in circulation
127
Q

what are leukocytes?

A
  • white blood cells
  • monocytes, lymphocytes, defense and immunity
  • neutrophils (phago), eosinophils (allergic), basephils (inflammation)
128
Q

what are platelets?

A
  • blood vessel repair
  • not cells
  • mediate blood clotting
129
Q

what is the protein that binds oxygen?

A

hemoglobin

130
Q

what is the structure of hemoglobin?

A
  • 2 alpha
  • 2 beta
  • each carry heme (iron carrying)
  • 4 oxygens can bind one Hb tetramer
131
Q

what is the biggest driver for oxygen affinity?

A

pH

132
Q

how have fish managed to use their anatomy to create countercurrent flow?

A

as blood moves to the surface of gills, the blood moving the other way can bind O2

133
Q

what does negative pressure (created) from the diaphragm contraction do?

A

inflates the lungs

134
Q

what is an immune response?

A

the ability to recognize foreign or dangerous macromolecules and eliminate them

135
Q

what is a lymphocyte?

A

non granular white blood cells that can cross into the lymph system

136
Q

what is an antigen?

A

molecule specifically reckognized as foreign or dangerous by cells of the immune system

137
Q

what are antibodies?

A

highly specific immunoglobulin proteins that reckognize and bind to specific antigens

138
Q

what are the two classes of soluble factors released during innate immune response?

A
  • cytokines: act as an alarm, signalling breach of the barriers
  • complement: enhance inflammation and attract phagocytes (patrol)
139
Q

how does a phagocytic cell reckognize something it should eat?

A
  • potential pathogens (mostly bacteria) contain PAMP (pathogen associated molecular pattern) which are molecules found on pathogens
  • phagocytes carry PRR’s (pattern recog. recep.) which will bind and reckognize PAMP
140
Q

what is the interaction between phagocytes and potential pathogen?

A
  • phagocytes destroy potential pathogens and keep a souvenir (anti gen)
  • then they present the antigen
141
Q

what is the role of B cells?

A

can produce anti bodies, or become memory cells which continue to produce anti bodies after an infection has been overcome

142
Q

what are the similarties and differences between innate and adaptive immunity?

A

innate
* rapid response
* B cells: target foreign pathogens
* T cells: target aainst diseased self cells

Adaptive
* slow response
* more specific
* B cells: generate antibodies against bullies
* T cells: recognize diseased traitor cells

Similarities
* innate reports to help T cell to coordinate adaptive immunity

143
Q

what produces anti bodies?

A

B cells

144
Q

describe what happens the different times you’re exposed to pathogen

A
  • first exposure: you need to identify and fight
  • 2nd time: now you know how to defend, faster recovery/defense
145
Q

what is hypertonic and hypotonic?

A

hyper: more solute inside cell, water flows in (freshwater-cell gets to big/swells)
hypo: less solute inside cell, water flows out (salt water- shrinks)

146
Q

what is osmolarity?

A

a property of a solution

147
Q

what is molarity

A

the concen. of a solute in a solution

148
Q

what is tonicity?

A

solutions affect on cell volume

149
Q

what is osmotic pressure?

A

force produced by movement of water

150
Q

compare fresh vs salt water fishes

A
  • fresh: environment is hypotonic, fish are hypertonic to their environment, water flows in
  • salt: environment is hypertonic, fish are hypotonic to their environment, water flows out
151
Q

what are osmoconformers?

A
  • adjust the osmolarity of the body to the environment
  • can’t survive in the extremes
152
Q

what are osmoregulators?

A
  • body maintains constant osmolarity
153
Q

what are ionoregulators vs ionoconformers?

A

regulators: concen. of ions in the blood are tightly regulated
conform: allow concen. of ions in blood to very in the environment

154
Q

what is the difference between stenohaline and euryhaline?

A
  • steno: narrow tolerance for changing salt concen
  • eury: tolerate a range of salininty
155
Q

what are the 3 processes involved in he formation of urine?

A
  • filtrations: passive loss of wasye
  • reabsorption: active reabsorption of nutrients
  • secretion: active exctretion of waste, toxins, etc
156
Q

what is the kidney?

A

collection of nephrons that filter the blood and excrete wastes

157
Q

what is the function of the loop of henle?

A
  • to create an osmotic gradient that can be used to retain water at the collecting ducts
  • relatively passive process
  • the longer the loop, the greater the ability to create more concen. urine and absorb more water
158
Q

what is the pathway of filtrate through the nephron?

A
  • renal artery
  • glomerulus in Bowman’s capsule
  • proximated convulated tubule: active in secretion of wastes, reaborpstion of ions
  • loop of henle: establish a concen gradient in interstitium
  • distal convulated tubule: monitor the filtreate and pressure, responsive to blood pressure
  • collecting duct: allows water reaborspotion with ADH
  • ureter to urethra out
159
Q

what is in the filtrate? what do we not want to see therw?

A

in it
* urea
* excess water
* excess ions
* water

not want
* glucose
* nutrients
* blood cells

160
Q

what controls the movement of things into and out of the filtrate?

A

into
* blood pressure into bowmans capsule
* active secretion

out
* making pee
* reaborption
* passive water retention

161
Q

what hormones act on the kidney and how?

A

RAAS: raises blood pressure

ADH: via HPA axis, opens aquaporins in collecting ducts, allows water retention

162
Q

what does the kidney produce in response to low blood pressure?

A

renin

163
Q

what does reaborption in distal convulated tubule cause

A

retention of fluids, increase blood volume, increase blood pressure

164
Q

what is parhenogenesis?

A

female haploid egg can spontaneously duplicate genome and produce diploid zygote q

165
Q

what is parhenogenesis?

A

female haploid egg can spontaneously duplicate genome and produce diploid zygote q

166
Q

what is budding?

A

offspring grows off of them

167
Q

what chromosome determines maleness?

A

y

168
Q

why are reptile sex determination messed up by humans?

A

it is determined by incubation temp, and sex ratios are skewed due to climate change

169
Q

what is ovipary?

A

lays eggs and developmet completed
* birds
* fully formed chick hatches

170
Q

what is ovovivpary?

A
  • young develop within the female
  • derive nutrition from yolk exclusively
  • no placenta
171
Q

what is vivipary?

A
  • young develop within female
  • derive nutrients via the mother
  • have placenta
172
Q

what type of feedback do sex hormones have, and why?

A

negative feedback to maintain homeostasis

173
Q

how do you get sex specific responses?

A

produce different receptors

174
Q

what is pre ovulation?

A

stimulate growth of the follicle

175
Q

what do FSH and LSH do for an oocyte?

A

mature it (stimulates meiosis)

176
Q

what does the egg contain?

A

oocyte and support cells

177
Q

what does the corpus letuem produce?

A

progesterone

178
Q

what does progesterone do?

A

maintain corpus luteum and uterine lining awaiting implantation

179
Q

what is the blastula?

A
  • population of identical cells in embryonic development
  • contains inner cell mass and trophoblast
  • the inner mass cells of the blastula inverts itself in a provess called gastrulation which forms 3 tissue layers
180
Q

what are the 3 tissue layers the gastrula forms?

A
  • endoderm: becomes most organs (inside)
  • mesoderm: become muscle (middle)
  • ectoderm: becomes skin and nervous system (outside)
181
Q

what does oxytocin do for partuirition (child birth)?

A

triggers the strenghtening contractions due to positive feedback loop

182
Q

what is the difference between positive and negative feedback?

A

Positive feedback occurs to increase the change or output: the result of a reaction is amplified to make it occur more quickly. Negative feedback occurs to reduce the change or output: the result of a reaction is reduced to bring the system back to a stable state.

183
Q

what is pheremone detection?

A
  • chemodetection
  • signalling factors
  • released into the environment
  • released by one sex to lure the other
  • bind to receptor on sensory euron triggering signalling pathway
    *
184
Q

what is taste and smell?

A
  • chemosensory responses: detect volatile chemicals in the environment
  • taste: sensory cell is not a neuron but a cell with microbilli, which stick out of the taste bud
  • smell: receptor sits on a neuron, neuronal sensors
185
Q

what. is magnetosensing?

A
  • detects magnetic poles and emissions
186
Q

what is the connection with bees and magnosensing?

A
  • bees land on flower and disrupt magnetic field
  • next bee can tell that the magnetic field has been disrupted
187
Q

what are the light sensing cells and what do they do?

A
  • cone and rod
  • detect the electromagnetic spectrum, energize and activate neurons
188
Q

where does the action potential leave int he eye

A

via the optic nerve

189
Q

is the eye an example of irreducible complexity?

A

no

190
Q

what are TRP receptors?

A
  • heat receptors
  • cell sigballing receptors
191
Q

which receptor is sensitive to heat and cold?

A
  • TRPA1: cold and menthol
  • TRPV2: heat and hot food
192
Q

what are homeotherms?

A

maintain a constant body temp and therefore respond to changes in ambient temps to maintain internal body temp

193
Q

what. isthe central thermostate for vertebrates?

A

hypothalamus

194
Q

what are four types of mechanireceptors?

A
  • merkel’s disks
  • meissner’s corpuscles
  • ruffini endings
  • pacinian corpulses

all sense different degress of touch of skin

195
Q

what are tropic hormones

A

a group of hormones that stimulate other endocrine glands so as to produce their particular hormones.

196
Q

how do you feel full?

A
  • when adipose is full of fat, secretes the hormone leptin that goes to the brain and signals that you’re full.content: stops hunger
  • when colon is full: gut releases peptide yy which goes to region of brain ans stops hunger
197
Q

what happens when your stomach is empty?

A
  • releases hormone ghrelin, which travels to the brain and stimulates appetite