Counterstrain Flashcards
Theories of somatic dysfunction initiating
Mechanoreceptor intiated
Nociceptor initiated
Nocifensive reflexes responding to nociception activation
Metabolic and blood flow imbalance
Counter strain defined
Indirect treatment that passively places a segment of muscle(s) in the position of comfort
Halts inappropriate nocieption and proprioception actions
Tenderpoint
Point on an anatomical location which exhibits tenderness when probed.
-usually at musculotendinous junctions to belly of muscles
Generic tenderness is caused by what?
Inflammation, infection, tumors, or trauma
SOMATIC DYSFUNCTION IS NOT TISSUE DAMAGE often accompanies it though.
Treatment of tenderpoint
Not actually treating the tenderpoint. Since it usually manifested via a somatic dysfunction of the involved joint and muscles that cross said joint.
Tenderpoints are not symptoms, they are signs
Positioning phases during CS
Gross movement: actually placing patient in classic CS position
Fine tuning: small movements to the parts that further decrease the sensitivity at the tender-point
Treatment position is often very specific (too much or too little reduction can cause increased tenderness
Why do we shorten the muscle for 90 seconds
Gives enough time for proprioception and mechanorecption to decrease. This allows the CNS to rest to normal resting length, remove pressure of small vessels, allow better blood flow, decrease noxious stimuli and increase tone of sympathetic nervous system.
Treatment with regards to patient
Patient should remain relaxed throughout the treatment.
Physician finger should monitor the tenderpoint and and only apply pressure every 30 seconds to recheck
After treatment, Patient should remain relaxed and be placed back into neutral passively by the physician ONLY. Physician should move slowly and keep finger on tenderpoint
Advantages of CS
Non-traumatic
Increases patient confidence quickly if done properly
Easy and effective
Easy to record diagnosis
Disadvantages of CS
Size difference between patient and physician can be problematic
Quantifying pain is completely subjective to patient
Can have reactions afterwards up to 36 hours
- water and acetaminophen/ibuprofen should be used when needed
Mechanism of a joint at rest
Primary sensory nerve endings of muscle spindle fibers send normal rate of impulse to the CNS to induce tonic muscle contraction
Mechanism of strain
Severe overstretch of one muscle and understretching of another
Overstretched = increases impulses from primary and secondary nerve endings
Undertretched = decreased impulses from primary and secondary nerve endings
Results in imbalance of muscles which elects pain.
- Strain is almost always NOT the point of injury*
- can elicit a sharp reflex muscle contraction if returning to neutral too quickly*
Nocifensive reflex
Seen in strains
understretched muscle cannot straighten out to normal and the overstretched muscle causes hyper stretching of B when it tries to return to neutral (pain).
Mechanism of counter strain in patients with strain
Moving the patient back to the position of the strain turns off the CNS action. The physician then returns the understretched muscle or abnormal proprioceptive muscle to a shortened position.
This allows the overstretch muscle to return to normal when the CNS activity returns
Must be done passively to not reactivate understretched muscle and slowly to not reactivate muscle spindle fibers in the overstretch muscle.
Difference between trigger and tender points
Trigger point:
- located in muscle tissue
- presents with characteristic pain pattern
- elicits radiating pain
- taut band is present
- elicits twitch response
- follows dermatome map
Tenderpoint
- all the opposites