Convergence Insufficiency Flashcards
What’s the normal near point of convergence?
Between 6 - 10cm (Abraham et al., 2015)
What is Convergence Insufficiency?
Convergence insufficiency is present if the near point of convergence (NPC) is less than 10 cm or if it can be maintained at this level only with effort
What is the prevalence of convergence insufficiency?
2.7% to 17.6% (Ma et al., 2018)
What’s the difference between Primary & Secondary CI?
Primary CI
CI initial defect
Secondary CI
CI secondary to other defect e.g. significant heterophoria
What are some predisposing factors for Primary CI?
- Genetic: Wide interpupillary distance IPD
- Environmental: demands of occupation
e.g. use of mainly distance vision and uniocular vision.
What are some precipitating factors in Primary CI?
- Illness
- Stress
- Increased close work demands
(Jenkins, 1999)
What did Nunes et al. (2019) find about Primary CI?
Of 292 people aged 10 - 14 6.8% previously had CI, 3.1% also had AI (accommodation insufficiency) and 6.8% have AI without CI
What might CI be Secondary to?
- Strabismus e.g. large X or primary intermittent nr XT
- Systemic Disease e.g. TED
- Drugs
- Mechanical Deficit e.g. Duanes
- Neurological Disease e.g. Parkinsons, PSP
- Refractive Error
- Accommodative Anomalies
What Mechanical & Neurogenic Strabismus might CI be Secondary to?
- Large exophoria
- SO palsy (vertical deviation makes convergence difficult)
- Duanes type C
- TED
What did Burke et al. (1993) find out about TED and CI?
22% of those with active TED have secondary CI
When might CI be secondary to Rx?
- Uncorrected high hypermetropia
May choose not to accommodate to avoid ET - Overcorrected hypermetropia
Have relaxed accommodation - Acquired Myopia
Eliminates need to accommodate at near so less accommodative convergence - Presbyopia
New glasses reduces accommodation stimulus and so accommodative convergence
What did Jenkins (1999) find out about Presbyopia?
New gls reduce accomm stimulus and therefore accommodative conv
What neurological disease might CI be secondary to?
- Parkinson’s Disease
- Progressive Supranuclear Palsy (PSP)
What did Irving et al. (2016) find out about secondary CI and neurological disease?
31.3% of those with Parkinson’s Disease had secondary CI
What drugs might CI be secondary to?
- Over 100 drugs can affect accommodation and therefore possibly convergence (Mazow et al., 1989)
e.g. tranquilizers like Lorazepam for anxiety (but found it doesn’t affect accommodation Speeg-Schatz et al., 2001)
What did Cuiffreda et al. (2007) find out about Traumatic Brain Injury and Secondary CI?
Age 8-91, n = 160, TBI
CI 42.5%
(41.1% accommodative dysfunction – mainly insufficiency)
What did Alvarez et al. (2012) find out about Traumatic Brain Injury and Secondary CI?
Age 5-89, n = 557, TBI
CI 9% without other ocular problems
CI 23.3% TBI group (with and without ocular problems)
(24.2% accomm dysfunction)
What are the symptoms of CI?
Intermittent horizontal diplopia at near
Asthenopic symptoms:
- Frontal headaches
- Eye strain/eye ache
- Photophobia
- Nausea
- Epiphora
- Blurred near vision
What do we need to ask in a case history about CI?
Symptoms?
- When do symptoms occur?
- When did symptoms begin?
- Did anything happen at the time symptoms started?
GH?
- Medication
- Previous treatment
What factors should be considered in CI?
Their refractive error (& what they’re wearing)
What did Momeni-Moghaddam et al., (2013) find out about CI symptoms?
Symptoms occur when NPC (near point of convergence) is greater than 10cm
What did Momeni-Moghaddam et al., (2013) find indicates a diagnosis of CI?
- 124 medical students
- Convergence Insufficiency Symptom Survey (CISS)
15 questions, symptoms during close work, in adults ≥ 21 = symptomatic - 33.9% asymptomatic 8.4cm
61.1% symptomatic 11.7cm
NPC 9.5cm cut off for symptoms
What did Horwood (2014) find out about CISS (Convergence Insufficiency Symptom Score)?
- 171 students, naïve, 18 - 26 years
- Symptoms common without signs of poor convergence
- Some with reduced convergence have no symptoms
This indicates using both questionnaire and clinical judgement as part of our assessment because people will have symptoms of CI without convergence insufficiency
When investigating convergence insufficiency what do we need to consider?
- Accommodative Amplitude/NPA (BEO, Monocularly, Age)
- Facility
(+/- 2DS, binocularly, Nott dynamic RET) - OM
(for differentially diagnosing secondary cause) - Fusional amplitude
(Sheard’s criterion - fusional reserve must be twice amount of heterophoria for it to remain nicely controlled) - Fundus & media check
- Refraction
When would we say a strabismus was secondary?
If someone had a 3BI XT would say Primary CI but if someone had like 15BI XT you’d call this secondary