Contributing Factors to LBP Flashcards

1
Q

Biomechanical Contributing Factors

A

noxious input from abnormally loaded tissue is driving the problem

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2
Q

Biomechanical Contributing Factors - four main issues

A
  1. movement is slower and more cautious in individuals with LBP
  2. for niche activities where there is LBP, individuals spend a prolonged period time at end rage of lumbar movement (eg. cyclists spend a long time in excessive lumbar flexion)
  3. potenital sub-optimal load sharing between the lumbar spine and hip
    - people with LB tend to use Lx early on in movement
    - have greater contribution of Lx to movement
    - have decreased hip range and hip asymmetry
    - have poor hip muscle function
    - have dysfunctional load sharing
  4. Greater compressive loads in individual with LBP whilst performing activities (mainly lifting) when compared to individuals without LBP
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3
Q

Structural Pathology - when is it important?

A
Important for individuals with 
- serious medical condition 
- radiculopathy 
- degenerative central canal stenosis 
However - must remember that these abnormalities sometimes exist in individuals without any LBP 

For OLBP - evidence shows that imaging and changes to structure are not helpful at all

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4
Q

Muscular Dysfunction Contribution - Punjabi Model of Spinal Function

A

Passive sub-system - passive support and guidance to the joint
Active sub-system - intrinsic muscle characteristics (size, length and orientation of the muscle)
Control sub-system - skill required in order to appropriately activate the muscles

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5
Q

Musc Dysfunction - Local Muscles

A

inner unit

  • act on the lumbar spine and also attach to it
  • short, medial and deep back muscles
  • main job in stabilising
  • can act segmentally on the spine
  • have osseous/broad insertions
  • more oxidative - high fatigue resistance
  • able to move the lumbar spine independently from the thoracic spine
  • control joint stiffness
  • control segmental movement trajectory
  • unable to balance large external loads
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6
Q

Musc Dysfunction - Global Muscles

A

outer unit

  • act on the trunk but don’t have direct attachment to the lumbar spine
  • long, lateral and superficial
  • have tendinous insertions
  • more glycolytic biology - less fatigue resistance
  • unable to have segmental control - move both the lumbar and thoracic spine together
  • have little effect on specific intervertebral control
  • unable to compensate for loss of local function
  • designed for global rigidity
  • produce stabilisation under large external forces via co-contraction
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7
Q

Musc Dysfunction - Altered Musculature

A

wasting of posterior muscles
increased percentage of fat in muscles
wasting of the psoas muscles
transition from slow twitch to fast twitch fibres

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8
Q

Musc Dysfunction - resultant functional problems

A

trunk muscles are deconditioned
leg muscles are deconditioned
impaired control of the back muscles
altered lifting mechanics

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9
Q

Musc Dysfunction - deconditioning of the trunk muscles

A

weak lumbar extensors
poor endurance of lumbar extensors and flexors
- main contributing factor
- predictor of future back pain
- alligns with aggravating activities described by patients

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10
Q

Musc Dysfunction - deconditioning of leg muscles

A

Pts with LBP more likely to have

  • weaker hip abductors
  • reduced endurance of hip abductors
  • weaker knee extensors
  • weaker hip extensors
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11
Q

Musc Dysfunction - impaired control of back muscles

A

lose flexion relaxation - extensors don’t turn off fully during flexion
asymmetric muscle recruitment
erratic muscle contraction
decreased force rate development
take longer to recover equilibrium with sudden load release
take longer to recover equilibrium with sudden load
delayed off set sudden release
delayed onset of appropriate muscles with sudden load
greater co-contraction of muscles - lose precision of muscle contraction
take longer to recover equilibrium with arm movement
increased disruption to balance

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12
Q

Musc Dysfunction - altered lifting mechanics

A
earlier muscle activation 
takes longer to switch muscles off 
greater co-contraction of msucles 
greater asymmetry in muscle contraction 
same with other functional tasks - walking, standing, sit to stand etc.
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13
Q

Neural Processing - contribution to LBP factors

A

increased efficiency of nociceptive processing
less attentive to non-noxious inputs
put more importance on danger than safety
representation of the back changes
back feels different

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14
Q

Neural Processing - increased efficiency of nociceptive processing

A
  • increased sensitivity over the back
  • increased sensitivity elsewhere in the body
  • enhanced facilitation and temporal summation of pain
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15
Q

Neural Processing - less attentive to non-noxious input

A
  • poorer tactile acuity
  • poorer proprioceptive activity
  • less sensitive to muscle vibration - don’t initiate a response to muscle stimulus
  • poorer overall balance
  • display sensory neglect like features
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16
Q

Neural Processing - seem to weigh danger over safety

A

Brain always gives greater attention to evidence that supports its predictions
Then acts in order to resolve any prediction error

Sensitivity = method of minimising prediction error, try to align evidence with predictions
Means that
- danger messages become more precise and trustworthy
- safety message becomes less precise and trustworthy

17
Q

Neural Processing - representation of the back changes

A

Neuro-immune changes in the sensorimotor areas of the brain
Sensory smudging - shift of area in the brain that is activated when the back is stimulated
Motor smudging - loss of demarcation (boundaries) between body areas in the motor cortex

Makes the back feel…

  • foreign and disconnected
  • distorted
  • hard to control
  • reinforced the model of the unhealthy self
18
Q

Psychosocial Contributors to LBP

A
Yellow Flags
Attitudes and beliefs
Behaviours
Compensation issues
Diagnosis and treatment issues 
Emotions
Family 
Work
19
Q

Psychosocial - Attitudes and Beliefs

A
  • belief that LBP is indicative of a serious medical condition
  • belief that LBP is degenerative
  • belief that pain signals damage
  • belief that activity is harmful
  • belief that all pain needs to be abolished before returning to activity
  • belief that the back is vulnerable
  • low self efficacy
  • belief that they won’t get better
  • belief that pain is uncontrollable
20
Q

Psychosocial - Behaviours

A
  • report extreme levels of pain
  • passive coping mechanisms
  • inflexibility in coping
  • activity avoidance
  • social withdrawal
  • boom/bust approach to activity
21
Q

Psychosocial - Compensation

A
  • previous claim or sickness benefits
  • disputes over claim/access to benefits
  • unhappy with how employer has dealth with the issue
  • perceived injustice - someone else is to blame
  • lack of financial incentives to return to work
22
Q

Psychosocial - Diagnosis and Treatment issues

A
  • previous advice to withdraw from work/activity from other HCP
  • conflicting diagnosis and explanations
  • catastrophising and threatening language
  • dramatisation of the problem
  • continual passive treatment
23
Q

Psychosocial - Emotions

A
  • fear of re-injury
  • hypervigilance
  • depression - causally mediated
  • irritable/angry
  • under stress and feel a loss of control
24
Q

Psychosocial - Family

A

Overprotective

  • reinforces fear and catastrophisation
  • contributes to disablements

Punitive - don’t believe they are injured
- patient feels as though they have to prove they are sick

25
Q

Psychosocial - Work

A
  • belief that work if harmful
  • repetitive of boring work
  • lack of control over work
  • job dissatisfaction
  • poor relationship with colleagues
  • negative experience of workplace management
26
Q

Lifestyle Contributors to LBP

A

sedentary lifestyle
obesity and diet
smoking and alcohol
sleep

27
Q

Lifestyle - Sleep Education

A
  • regular bed and rising times
  • eliminate noise and light from bedroom
  • regulate bedroom temperature
  • get up from the bed if awake
  • avoid caffeine, alcohol and nicotine
  • remove phone and computers from the bedroom
  • avoid screens and bright lights before bed
  • minimise napping
  • regular exercise