Content for Exam 1 Flashcards
Eye opening GCS grades
(4 = spontaneous opening of eyes; 3 = opening in response to speech; 2 = eye opening to pain; 1 = no eye opening response)
verbal response GCS grades
(5 = verbal response demonstrate personal orientation to location; 4 = confused conversation is the best verbal response; 3 = inappropriate words in response to question; 2 = incomprehensible sounds; 1 = no verbal response)
motor response GCS grades
(6 = follows motor commands; 5 = localizes a painful stimulus; 4 = withdraws specifically from pain; 3 = abnormal flexion response to pain; 2 = abnormal extensor response to pain; 1 = no motor response)
Rancho Los Amigos Levels of Cognitive Function (LOCF)
Widely used in BI
To track cognitive and behavioral responses of pt
Level I (no response, coma) to Level X (purposeful, appropriate)
Coma Recovery Scale (CRS-R)
Better sensitivity for lower levels – coma<->veg state<->min cons state
Terminal Swing
Critical Events: Knee extension to neutral (or within 5 degrees)
Begins with vertical tibia
Ends with initial contact
Anterograde
inability to learn new material after injury
Retrograde
inability to remember things prior to brain injury
PTA (post-traumatic amnesia)
loss of memory of event surrounding brain injury and inability to process info after BI – length of PTA is a prognostic factor for recovery
Amnesia
deficits in memory
Agnosia
inability to recognize familiar objects, persons, sounds, shapes, or smells – visual, tactile (astereognosis), auditory
Dysarthria
Problems in oral motor aspects of articulation
Aphasia
loss of ability to understand or express speech due to brain damage, mostly in dominant hemisphere
Wernicke’s/receptive/fluent aphasia
deficits in auditory/written comprehension with well articulated speech marked by nonsense words or substitutions
Broca’s/expressive/non-fluent aphasia
deficits in expressing thoughts using speech, limited vocabulary, but comprehension relatively preserved.
Stereognosis
Ability to recognize form of objects by touching and feeling, without visual input
Objects like keys, comb, coins
Graphesthesia
ability to recognize letters, numbers or designs ‘written’ on skin
Barognosis
ability to distinguish weights of different objects by holding them
Assessment of Spasticity
Force/Velocity dependent increased resistance to stretch
Clasp-knife response
Modified Ashworth Scale (0-4 grades)
Sign of UMN lesion - pyramidal tracts
Assessment of Rigidity
Resistance to stretch is independent of force/velocity
Lead-pipe response - constant resistance
Cogwheel response – rachet-like jerkiness
Sign of UMN lesion – extra-pyramidal tracts, basal ganglia lesions like Parkinson’s.
Modified Ashworth Scale
Assessed during PROM with pt in supine
If testing a muscle that primarily flexes a joint, place the joint in a maximally flexed position and move to a position of maximal extension over one second
If testing a muscle that primarily extends a joint, place the joint in a maximally extended position and move to a position of maximal flexion over one second
put muscle in its shortened position
MAS scoring for assessing spasticity
0 No increase in muscle tone
1 Slight increase in muscle tone, manifested by a catch and release or by minimal resistance at the end of the range of motion when the affected part(s) is moved in flexion or extension
1+ Slight increase in muscle tone, manifested by a catch, followed by minimal resistance throughout the remainder (less than half) of the ROM
2 More marked increase in muscle tone through most of the ROM, butaffected part(s) easily moved
3 Considerable increase in muscle tone, passive movement difficult
4 Affected part(s) rigid in flexion or extension
General Tone assessment
0 No response (flaccidity)
1+Decreased response (hypotonia)
2+ Normal response
3+ Exaggerated response (mild to moderate hypertonia)
4+Sustained response (severe hypertonia)
Hypotonia
Decreased or absent tone, flaccidity, limbs are floppy, hyper-extensible joints, stretch reflexes are dampened (hyporeflexia)
Primitive/tonic reflexes
should be integrated during infancy, can persist with delayed development or re-emerge following brain injury. Tonic reflexes change postural tone rather than producing overt movements
Reflex integrity scale
0: no reflex
1+: hypo-reflexive
2+ normal
3+: hyper-reflexive
4+ Clonus
UMN Lesion: increased/brisk reflexes
LMN/Peripheral sensory lesion: hypo-reflexive
Babinski Sign
Big toe extends and other toes fan out.
Sign of UMN lesion in adults.
Babinski sign is normal in infants up to 1 years age, should integrate by 2-3 years
Strategy level
decides the goal of movement, best movement strategy to achieve the goal, represented by - association motor areas, basal ganglia and cerebellum
Tactics level
decides sequence of muscle contraction in space and time, represented by primary motor cortex and cerebellum
Execution level
stimulate motor neurons to activate muscles appropriately for movements/postural control, and feedback(cerebellum)
Ataxia
most common problem associated with cerebellum, results on ‘ataxic gait’ – poor trunk control, wide BOS, arms high guard position, irregular stepping.
Dysdiadochokinesia
impaired ability to perform rapid alternating movements, eg, supination/pronation
Dysmetria
inability to judge the distance, over- or under-shooting.
Dyssynergia
inability to perform smooth movements using synergistic muscle, breaking into components
Rebound phenomenon
inability to stop motion when resistance is released, pt might hit himself/herself when resistance is removed, cerebellum problem
Bradykinesia
slow movements, expressed as decreased arm movements and shuffling and festination during gait, Parkinson’s gait. Basal ganglia issue
Akinesia
progression to inability to initiate movements – freezing episodes, basal ganglia issue
Athetosis
involuntary writhing
‘worm-like’ movements.
Also a clinical sign of athetoid CP
Basal ganglia issue
Chorea
involuntary irregular jerky movements mostly seen in UE, cannot be inhibited, clinical sign of Huntington’s disease, basal ganglia issue
Dystonia
sustained involuntary contractions of agonist/antagonist muscle, abnormal posturing, torsion spasms, basal ganglia issue
Hemiballismus
large-amplitude movements, sometimes violent, on one side of body, an arm or leg
Gross motor tests
Involve large muscle groups to test ability to crawl, kneel, stand, walk w/o abnormal postures w/o LOBs
Also test balance – called equilibrium tests
Fine motor tests
Involve small distal muscle groups, test fine motor activities like finger dexterity tasks
Fine motor tests are also called non-equilibrium tests
Steady-state balance
Quite sitting/standing
Ability to control the location of the body’s center of mass within the area defined by the base of support under predictable, quasi-static conditions. This includes the ability to adapt motor behavior to meet the demands of different task and environmental conditions
Proactive/Anticipatory balance
Ability to generate postural adjustments prior to the onset of and during voluntary movement for the purpose of either countering an upcoming postural disturbance due to voluntary movement or realigning the body’s center of mass prior to changing the base of support. This includes the ability to adapt motor behavior to meet the demands of different task and environmental conditions.
Reactive balance
Ability to respond to sensory input that signals a need for a response to ensure successful maintenance of postural control. The need for a response is unanticipated but may be generated externally (perturbation originating external to body) or secondarily to an internally generated movement. This includes the ability to adapt motor behavior to meet the demands of different task and environmental conditions
Ankle strategy
Used when sways are small and slow
Move COM of body as a block about ankle joints
Muscles are activated distal-to-proximal
During forward sway, gastrocnemius hamstrings paraspinals
During backward sway, tib ant quads abdominals
Used on firm support surface
Hip strategy
Used with larger and/or faster sways
Moves COM by flexing/extending/abd/adducting hips
Muscles are activated proximal to distal
Forward sway, abdominals Quads.
Backward sway, paraspinals Hamstrings
Used when support surface is small/narrow/compliant
Suspension strategy
Moves COM down by flexing hips/knees and
flexing trunk
May progress to squating
Regulatory features
specific aspects of the environment shape movement, size, shape, and weight of a cup, type of surface a person walks on. Movement control strategies need to conform to or adapt to these features
NON Regulatory features
aspects that are not critical to accomplishing movement, Gait with lights on vs off, noise and distraction
May affect performance, but movement does not need to conform to these features
Initial conditions
Observe initial posture, environmental conditions to check if person is in a position to perform the task
Preparation phase
Did the person understand your instructions to get in the ‘ready-position/ready-posture” to be able to correctly initiate the task – observe for stability, alignment, verticality, symmetry of posture
Initiation phase
The initial movement of the task – observe for amplitude, timing and direction of movement
Execution phase
The majority of the dynamic movement phase of the task – observe amplitude, coordination (smoothness, sequencing, timing), direction, speed, symmetry of the movements
Termination phase
Posture at the end of the task – observe for stability, alignment, verticality, symmetry of posture
SCOOTING WHILE LYING DOWN essential components
In initiation, the essential component is the hook lying position, in execution phase, essential component is bridging
ROLLING essential components
in initiation phase, essential component is cervical flexion/rotation and hip flexion/adduction
SUPINE-TO-SIT AT EDGE OF BED/MAT essential components
In initiation phase, possible essential component: “Partial sitting posture” by flexing neck, trunk, hips to lift off of surface