Concepts Exam#2 (Ch. 37) Flashcards

1
Q

Shock

A

widespread abnormal cell metabolism occurring when gas exchange with oxygenation and tissue perfusion needs aren’t met sufficiently to maintain cell function.

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2
Q

Progressive stage of Schock

A

Life-threatening emergency - Vital organs can tolerate this situation for only a short time before being permanently damaged.

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3
Q

MODS

A

Multiple Organ Dysfunction Syndrome

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4
Q

Multiple Organ Dysfunction Syndrome

A

Sequence of cell damage caused by massive release of toxic metabolites and enzymes

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5
Q

Shock Syndrome

A

Problems resulting form it occur in a predictable sequence

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6
Q

Mean Arterial Pressure Factors include

A

Total blood volume, Cardiac output, size and integrity of the vascular bed, esp. capillaries. If TBV and CO increase, so does MAP, vice versa.

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7
Q

Hypovolemic Shock

A

Occurs when too little circulating blood volume decreases MAP, resulting in inadequate total body perfusion and oxygenation.

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8
Q

Common Problems leading to Hypovolemic Shock

A

Poor clotting with hemorrhage and dehydration.

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9
Q

Cariogenic Shock

A

Occurs when the heart muscle is unhealthy and pumping is impaired.

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10
Q

Distributive Shock

A

Occurs when blood volume is not lost from the body but is distributed to the interstitial tissues where it cannot perfuse organs. Caused by blood vessel dilation, pooling of blood in venous and capillary beds, and increased capillary leak.

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11
Q

Septic Shock

A

Widespread infection that triggers whole-body inflammation.

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12
Q

Sepsis

A

Presence of infection systemic manifestations. Infectious organisms have entered the bloodstream. Results in widespread inflammation, known as system inflammatory response syndrome (SIRS)

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13
Q

Anaphylactic Shock

A

Extreme type 1 allergic reaction. Begins within seconds to minutes after exposure to a specific allergen in a susceptible person.

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14
Q

Anaphylaxis Results in

A

Widespread loss of blood vessel tone, with decreased BP and CO.

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15
Q

Neurogenic Shock

A

Type of distributive shock resulting in low BP, occasionally with a slowed heart rate, that is attributed to the disruption of the autonomic pathways within the spinal cord. It can occur after damage to the CNS such as spinal cord injury.

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16
Q

SIRS

A

Widespread inflammation from increase of infectious organisms entering bloodstream… Triggered as a result of infection escaping local control.

17
Q

Multiorgan Dysfunction syndrome (MODS)

A

When shock conditions continue for longer periods without help, resulting acid-base imbalance and increase metabolites cause so much cell damage in vital organs that they are unable to perform their critical functions. Once MODS occurs, recovery from shock is no longer possible.

18
Q

MODS Manifestations

A

Rapid loss of consciousness, non palpable pulse; cold, dusky extremities; slow, shallow respirations; unmeasurable oxygen saturation.

19
Q

Who should be assigned to assess vital signs of a patient who is at risk for or suspected of having hypovolemic shock?

A

Registered Nurse rather than a LPN/LVN or UAP.

20
Q

These organ systems characterize MODS

A

Respiratory, Renal, Cardio, Neurologic, Hepatic, Hematologic

21
Q

MODS Causes

A

Occurs as a secondary phenomenon. Include unrestrained inflammatory reaction or infectious response due to a severe injury or illness. Septic shock is most common reason for MODS.

22
Q

Main indicators of Shock presence or progression

A

Systolic Blood Pressure changes, pulse rate changes and quality.

23
Q

Interventions and Response to Shock

A

Focus on reversing the shock, restoring fluid volume to normal range, preventing complications.

24
Q

More interventions to Shock

A

Oxygen therapy, IV Therapy (Crystalloids), Plasma, Protein-containing colloid fluids restore osmotic pressure and fluid volume (whole blood, packed RBCs, Plasma),

25
Q

Drug Therapy for Shock

A

Vasoconstrictors (Dopamine, Norepinephrine), Inotropic Agents (Dobutamine), Agents Enhancing Myocardial Perfusion (Sodium - Nirtoprusside)

26
Q

Drug Alert - Monitor patient closely because drugs that dilate coronary blood vessels can cause systemic vasodilation and increase shock if patient is volume depleted

A

Example - Nitroprusside

27
Q

Nursing Interventions for Shock

A

Monitor Vitals q15min until shock is controlled, Hemodynamic monitoring (intra-arterial monitoring, Mixed venous Oxygen saturation, pulmonary artery monitoring, pulmonary capillary wedge pressures), Insertion of Central Venous Pressure catheter

28
Q

Risk Factors for Hypovolemic Shock

A

Diuretics, diminished thirst, immobility, aspirin, anticoagulants, ginkgo biloba

29
Q

Risk Factors for Cariogenic Shock

A

Diabetes mellitus, presence of cardiomyopathies

30
Q

Risk Factors for Distributive Shock

A

Diminished immune response, reduced skin integrity, presence of cancer, peripheral neuropathy, strokes, hospital stay, malnutrition, anemaia

31
Q

Action Alert - Use only normal saline for infusion with blood or blood products because the calcium in Ringer’s Lactate induces clotting of the infusing blood.

A

Nothing