COMPS:Unit 1: EKG Extra Stuff You Need to Know!!! Flashcards
What is ECG of EKG???
- Represents the electric impulses of the heart
- hearts functioning SHORT TERM
- Halter/Telemetry
- LONG TERM
ECG ===
- Graphic rep of hearts electrical activity
- provides info about hearts function
Why do PTs need to learn EKG??
4 Reasons:
- Basic anatomy & phys of heart–> norm vs. patho
- PT implications of rhythms –> norm vs. patho
- Diff b/w benign vs. life-threatening arrhythmias
- Read physicians notes on 12 lead EKG AND understand implications
Myocytes….
What are they?
cells of myocardium
3 Properties of Myocytes
- Automaticity
- Rhythmicity
- Conductivity
Properties of myocytes
1. Automaticity
explain…
*Discharge e-stim w/out nerve impulse (automatic)
-
SA Node (natural pacemaker of heart)
- 60-100bpm
-
AV Node (backup pacemaker)
- 40-60bpm
- kicks in to keep adequate HR
-
Perkinje (next backup Pacemaker)
- 30-40bpm
Properties of myocytes
2. Rhythmicity
explain…
*Spontaneous depolarization/repolarizatoin
*rhythmically fires
Properites of Myoctyes
3. Conductivity
explain..
*Carry rhythmic control from cell to cell (W/in OWN cells)
*Carry muscle to muscle
*NO NEURAL INPUT NEEDED
A DEPOLARIZATION wave will be a ________deflection
*Influx of Na+
POSITIVE deflection
A REPOLARIZATION wave will be _______ deflection
NEGATIVE deflection
ECG and Electrical Activity of the Myocardium
The Conduction System
see pics
ECG and Electrical Activity of the Myocardium
The Conduction System
More pics
Conduction System
P Wave===
Atrial Depolarization
*SA node fired B/L Atria
Conduction System
QRS Complex ====
Ventricular depolarization AND contraction
*AV node fired
NOTE: Atrial Repolarization hidden in QRS complex
Conduction system
T wave===
Ventricular Repolarization
Conduction system
Pics
Conduction System
All ECG components broken down
see pics
What lead is MOST COMMONLY recorded in an ECG?
Lead 2 !!!
Lead II:
Wave of Depolarization moving toward positive electrode==
+ Deflection
Lead II:
Repolarization moving towards positive electrode==
- Deflection
Lead II is used to determine ______ and ______
Rate and Rhythm
*Matches the angle of the heart along the axis of depolarization
Off of Lead II…
What do we interpret?
- Rate
* 3 methods
* ONLY METHOD 1 FOR IRREGULAR RHYTHMS
- Rate
- Regularity
- P wave
* Y/N?
* Upright?
* 1 for ea. QRS?
- P wave
- PR interval
* tiny boxes x.04
- PR interval
- QRS width
* tiny boxes x.04
- QRS width
Off of 12 Lead
What do we interpret?
- Axis
- Hypertrophy
- Ischemia/Infarction
PR Interval
What is NORMAL?
.12 (3 sm. boxes)–.20 (5 sm. boxes)
QRS width
What is NORMAL?
.06 (1.5 sm. boxes)–.10 (2.5 sm. boxes)
Rate
3 methods to determine rate
- R waves per 6sec strip x 10==Rate
- 300, 150, 100, 75, 60, 50…(make sure one of the QRS is right on the line)
- 300/# of Lg. boxes w/in 2 R waves
Measuring PR interval
Where to Where?
see pics
QRS Width
Where to Where ?
see pics
Normal Rhythms:
What are they
5
*ALL FROM SA NODE*
- NSR
- Sinus Tachy
- Sinus Brady
- NSR w/ Pause
-
Sinus Arrhythmia
- arrhythmia literally means “irregular rhythm”
- all else will be NORMAL
Normal rhythms
Whats Different
Whats Normal
-
NORMAL
- PR interval
- QRS width
-
DIFFERENT
- rate
- regularity
Sinus Bradycardia
Keep in mind…
NORMAL BUT Rate <60
*In high lvl athletes this is normal
Sinus Tachycardia
Keep in mind…
NORMAL BUT Rate >100bpm
Sinus Arrhythmias OR aka…
The Inspiration (faster) vs. Expiration (slower) one
*IRREGULAR so must use Method 1***
Sinus Pause OR
Other names ?
Sinus Block
Sinus Arrest
NSR w/ pause
*skipped beat/pause
*SA node fails to fire
Atrial Arrhythmias
*Occur in Atria!
5:
- Wandering Atrial Pacemaker
- @ least 3 diff. P-wave shapes!
- PAC
- has a P wave! diff. shape OR inverted
- Atrial Tachycardia (SVT)
- Atrial Flutter (Sawtooth)
- A-Fib
**ALL ectopic==> SA fires when should NOT
ONLY NORMAL THING W/ ATRIAL ARRHYTHMIAS
QRS Width bc QRS complex is ventricles and Atrial Arrhythmias are occurring in the Atria
Wandering Atrial Pacemaker
Keep in mind…
- Random ectopic foci
- P-wave constantly changing
- @ least 3 diff.
PAC
Keep in mind
- P-wave embedded in the T (Tall T)==PAC
- OR
- Tall T==PAC
Premature Beats
Compensatory (use 3 line rule)
match 1 and 3 up w/ 1 and 3 w/ premature beat in the middle and 1 and 3 should still line up….if NOT==NON-comp
- Pause following ectopic beat which allows reg rhythm to resume w/ next normal beat @ its orig. projected timing
Premature Beats
NON-compensatory
1 and 3 will NOT line up!!!
- Pause not long enough to allow rhythm to resume its original rhythm and timing
Which PAC compensation (NON-comp vs. COMP) represents a healthier SA Node?
Complete COMPENSATORY Pause
*SA goes back to firing Normal
3+ PAC in a row====
Atrial Tachycardia OR SVT
Atrial Tachycardia
3+ PAC in a row*
Other names?
Paroxysmal Atrial Tachy
Paroxysmal Junctional Tachy
Supraventricular Tachy (SVT)
Atrial Tachycardia or SVT
*remember this is w/ NO ACTIVITY we call it SVT otherwise WITH Activity we call it Sinus Tachy
Keep in mind…
- Tachy occurs ABOVE AV node
- rate always elevated
- common
- NOT benign
- Usually older adults w/ comorbidities
Rate for SVT (Tachycardia)
150-250bpm
EXAMPLE
NSR into SVT
see pics
Rate for “Flutter”
250-350bpm
Atrial Flutter
Keep in mind…
- Atrial rate will be > ventricular rate
-
Usually has a RATIO (report it!)
- ex. 4:1 Atrial Flutter
Fibrillation Rate
350+
SUPER FAST!
RATES LISTED
SVT
A-Flutter
A-Fib
- SVT==150-250
- A-Flutter==250-350
- A-Fib==350+
Atrial Fibrillation
Keep in mind
- Chaotic, squiggly line MESS b/w QRS complexes
- Rate: 350+
- Irregular MUST use method 1
A-Fib and Ejection Fraction
- Norm EF==60%
-
W/ A-Fib
- EF is diminished (by ~15%) w/ significant blood remaining in atrium
A-Fib
Keep in mind w/ CLOTS
A-fib you are @ risk for CLOTS!!!
- The atria are filled w/ ectopic foci
- The atria will shake like Jello
-
Pooled blood builds up in jello atria
- == HIGH RISK FOR DVT
-
Pooled blood builds up in jello atria
PT Implications A-Fib
*****
- Check t/o sessions for s/s of Low CO
- Neuro screen regularly
- ex. smile @ me
- CV/Pulm screen regularly
- PulseOX
- chest pain
- VITALS!!!
PT Implications of A-Fib
When the Ventricular Rate goes ABOVE 100
Rate >100bpm w/ A-Fib
- A-Fib w/ Rapid Ventricular Rate (A-fib w/ RVR)
- Pot. for LOW CO
Rates:
Tachy vs. Flutter vs. Fibrillation
- Tachy (Atrial Tachy/SVT)
- 150-250 Atrial AND Vent
- Flutter
- 250-350 Atrial rate
- Fibrillation
- 350+ Atrial rate
Junctional Rhythms
*Only @ AV NODE*** so it will have that AV Node rate (40-60bpm)
3:
- PJC — NO P Wave
- Junctional (Idiojunctional)
- Accelerated Idiojunctional (SVT)
- Tachy occurs @ or ABOVE AV node or OUTSIDE VENTS
ONLY NORMAL THING W/ JUNCTIONAL ARRHYTHMIAS
QRS Width
PJC
NO P OR INVERTED P
*Also “Same ht. T==PJC”
Junctional Rhythm
Idiojunctional
Keep in mind..
- NSR of AV Node
-
NO SA Node so AV node takes over as Backup Pacemaker
- __These will have that AV Node rate of 40-60bpm
- R. CA MI, Necrosis
Accelerated Junctional Rhythm
OR aka
Accelerated Idiojunctional
Nodal (Junctional) Tachycardia
Accelerated Junctional Rhythm
Keep in mind w/ Rate
-
Accelerated junctional rhythm rate will be Faster than the normal junctional rhythm rate
- Accelerated will be >60
- normal junctional remember is b/w normal AV node 40-60bpm
-
Junctional SVT will be >100
- remember SVT == 150-250bpm
-
STILL NO Pwave though!
- embedded into the T (same Ht. T)
EXAMPLE Junctional SVT
see pic
Sinus Tachycardia vs. Paroxysmal Atrial Tachycardia (SVT)
SAME thing EXCEPT if there is a known cause for Tachy
DIFFs broken down
-
SINUS TACHYCARDIA
- Doing Exercise!!!
-
Atrial Tachycardia or SVT
- 150-250 for no apparent reason!!!
Accelerated Idiojunctional Rhythm vs. Nodal Tachycardia (SVT)
Same thing EXCEPT if known cause for Tachy
DIFFs broken down
-
Accelerated IJR
- AV node >60bpm
- DOING EXERCISE
-
Nodal Tachycardia or SVT
- 100-250 for NO REASON
Straight up SVT
“P wave BURIED in the T==SVT”
ALL Premature Beats (PAC, PJC, PVC) NEED what?
Underlying rhythms!!!!!
PVCs and counting RATE
NO CO from PVC —DO NOT COUNT PVC IN RATE!!!
**PAC and PJC you DO COUNT
Normal Rhythms come from
SA Node
Atrial Arrhythmias come from
ABOVE AV Node
*IN Atria
Ventricular Arrhythmias come from
VENTRICLES
Ventricular Arrhythmias
7:
- PVC—DO NOT COUNT IN RATE
- V-Tach
- V-Flutter (Torsades de Pointe)
- V-Fib
- Idioventricular (NOT ON TEST)
- Asystole (Flatlining)
- PEA** (NO PULSE)
PVC Unifocal
SAME SHAPE
PVC Multifocal
DIFFERENT SHAPE
PT Implications of PVC
Check 02sats regularly
Significant PVCs
HIGH PT IMPLICATIONS
*Terminate PT!!!
5:
- Multifocal PVCs
- 6-10 or MORE PVCs/Minute
-
Bigeminy
- PVC every other (2nd) beat
-
Trigeminy
- PVC every 3rd beat
- >3 PVC’s in a row==> V-TACH
PAC
PJC
PVC
>3 IN A ROW
- >3 PAC==SVT OR Atrial Tachycardia
- >3 PJC==SVT OR Accelerated Idiojunctional
- >3 PVC==V-TACH
***KEEP IN MIND for SVT vs. Sinus Tachy or SVT vs. Accelerated IJR if they are EXERCISING or NOT DOING ANYTHING!!!!
3 PVCS in a row==V-TACH
Causes:
Acute MI
CAD
HTN
Rxn to meds
Electrolyte imbalance
CLASSIC V-TACH Strip
“Pyramids”
see pics
Still regular***
- Remember >3 PVCs in a row!!! == VTACH
- SUPER WIDE BIZARRE QRS
WORSENING of V-Tach
Torsades de Pointes/V-Flutter
- Looks like ribbon
- need AED to save them
- No pulse OR thready pulse
WORSENING of A-Flutter
-Disorganized, just a squiggly line
-Ventricles like jello
Ventricular Fibrillation
*remember 350+ bpm
V-Fib
Causes:
- CAD
- Acute MI
- Toxicity from drug
- Electrolyte imbalance
HEART BLOCKS OR…..
The “Detours” one!!!!
*Heart Blocks b/w communication from SA—> AV Node
1st Degree Heart Block
Things to know:
-
Detour one!!!
- impulse has to go thru detour so LOOOOOOOONG PR intervals
- Very benign
- older people
2nd Degree Heart Block Type I
Mr. Weckenbach
Sketchy!!! Loves to be LATE, LATER, LATER, NOT COME HOME!!!
- *NO IMPLICATIONS FOR PT
-
Progressive elongation of PR interval
- Mr Weckenbach comes home later, later, later each night spending time w/ his mistress THEN DOES NOT come home and spends the night with his mistress
- Block b/w SA and AV Node
2nd Degree Heart Block Type II
Mr. Morbitz!
- Mr Morbitz plays it safe by being on time multiple nights then one night DOES NOT COME HOME
- Mr Morbitz is On time, On time, On time, then DOESN’T COME HOME (DROPPED QRS)
- **AV blocks @ lvl of Bundle of HIS OR @ B/L bundle branches of trifascicular
2nd Degree Heart Block Type II
Mr Morbitz
RISKS
- Risk of going into more severe heart cond.
- Dx AFTER acute MI
3rd Degree Heart Block
- *Atria and Vents fire completely isolated of ea. other ==> NO COMMUNICATION
- VARYING PR intervals AND SO MANY P’S!!!
- LIFE-THREATENING ARRHYTHMIA
1st Degree Heart Block vs.
2nd Degree Heart Block Type I (Mr Weckenbach) vs.
2nd Degree Heart Block Type II (Mr Morbitz) vs.
3rd Degree Heart Bloc k
see pics
Pacemaker
What are the precautions?
NO LIFTING w/ UE w/ PM
*up to 2wks
The SA Node is the Pacemaker for
Atria
The AV Node is the Pacemaker for
Ventricles
Pacemaker
What is it?
electronic device used to generate an artificial action pot in the Atrium (SA PM) and/ or Ventricles (AV PM)
Pacemaker may be used temporary or permanent
If permanent: placed where?
Implanted under skin just below L. Clavicle
*may be adjusted EXTERNALLY
Pacemaker
Extra details
- Used for Brady or Dysrhythmias (arrhythmias)
- Pulse from PM firing results in a distinctive VERTICAL DEFLECTION called a Spike
- Pacer spike followed by a P for Atrial pacing and by a QRS for Ventricle pacing
Dual chamber Pacemaker
- 2 Leads
- One connected to Rt. Atrium
- for SA
- Other connected to Rt. Ventricle
- for AV
- One connected to Rt. Atrium
12 Lead EKG
V1-V6
physically put on
Picks up (+) deflections
Bundle Branch Blocks or…
The ones w/ TWO R waves
- Must be dx’d on 12 lead ECG
- Causes one ventricle to depolarize and contract LATER than the other due to the delay/block of the impulse w/in the bundle branch (after the AV node)
- 2 R waves–> one W/IN QRS
-
QRS will be .12s or WIDER
- Wider==any issue w/ vents
Bundle Branch Blocks–RIGHT
- Rt. Vents fire late
- R/R’ in leads V1 or V2
Bundle Branch Blocks LEFT
- L. vent fires late
- R/R’ in leads V5 & V6
12-Lead EKG: Hypertrophy
Right Vent Hypertrophy
vs.
Left Vent Hypertrophy
- Shift of the axis so you will pick up TALLER R-wave in:
- V1–Right
- V5–Left
- Loc. of side the R wave is GREATER (higher) on==side of hypertrophy
12 Lead EKG: Hypertrophy==
*INC in thickness of cardiac muscle OR chamber size
12-Lead EKG: Hypertrophy
Rt. Vent Hypertrophy vs. Left Vent Hypertrophy
-
R. Vent
- V1: LG R Wave bc mm BIGGER so takes LONGER
- R wave becomes progressively smaller in V2-V5
-
L. Vent–more common
- Lg. R wave in V5
*V1==Right
*V5==Left
12 Lead EKG: Ischemia
- ==> reduced blood flow to myocardium due to occlusion of CA’s from:
- Vasospasm
- atherosclerotic occlusion and/or Thrombus
12 Lead EKG
How is Ischemia demo’d?
3 Options:
- T wave INVERSION
2. ST segment ELEVATION
3. ST segment DEPRESSION
12 Lead EKG: Infarction
- ==> Cell DEATH resulting from complete occlusion of Coronary Artery
12 Lead EKG: Ischemia
Transmural Infarction
vs. NON-Transmural Infarction
-
Transmural (Completely covered)
- Cell necrosis or death COMPLETELY COVERS entire Myocardial wall of heart
-
NON-Transmural (Only one part)
- just occurs in one part—> still @ risk for FULL
NOTE: ST Segment depression in absence of ischemia or angina may be due to digitalis toxicity
12-Lead EKG: Ischemia
Leads that demo presence of T wave inversion, ST segment changes, or Q waves identify Location of ischemia, injury, or infarction
see pics
Significant Q wave and/or ST elevation in leads V1, V2, V3, V4
indicates—–
ANTERIOR INFARCTION
ANTERIOR INFARCTION
Significant Q wave and/or ST elevation in leads V1, V2, V3, V4
Significant Q wave and/or ST elevation in Leads II, III, and aVF
indicates
INFERIOR INFARCTION
*notice the aVF (Feet==inferior)
INFERIOR INFARCTION
*notice the aVF (Feet==inferior)
Sig. Q wave and/or ST elevation leads II, III, aVF
Sig. Q wave and/or ST elevation in chest leads I or aVL indicates…
LATERAL INFARCTION
*notice aVL (L means Lateral)
LATERAL INFARCTION
*notice aVL (L means Lateral)
Sig Q wave and/or ST elevation in chest leads I or aV_L_
DIRECT OPPOSITE tracing of Anterior Infarction in V1 an V2
indicates….
POSTERIOR INFARCTION
V1/V2 FLIPPED CONCEPT
POSTERIOR INFARCTION
V1/V2 FLIPPED CONCEPT
DIRECT OPPOSITE tracing of ANTERIOR INFARCTION in V1/V2
***From NPTE
What condition could explain why the HR of an exercising pt abruptly drops to half the value that was pre-recorded?
2nd Degree Heart Block
(Nrml–>block–>Nrml–>block)
Cuts CO by 50% bc of DROPPED QRS’s