Common Test Flashcards
Risk factor of GDM
Maternal obesity (90kg)
Maternal age older than 25 years
Previous unexplained stillbirth
Previous infant having congenital abnormalities
History of GDM in a previous pregnancy
Family history of DM
Large infant (>4kg)
Fasting glucose >126mg/dl or post glucose >200mg/dl
GDM is diagnosed via
oral glucose tolerance test
≥ 140mg/dl (positive)
< 140mg/dl (negative)
urine for glucose
Blood glucose
Fetal effects of GDM
Congenital abnormalities
Macrosomia(large size)
Intrauterine growth restriction
Birth injury
Delayed lung maturation
Neonatal hypoglycaemia
Neonatal hypocalcemia
Neonatal hyperbilirubinaemia
and jaundice
Neonatal polycythemia
(Hematocrit level>65%)
Perinatal death
Definition of uterine fibroids
Benign growth of the smooth muscle in the wall of the
uterus.
Known as myomas or leiomyomas
Well-circumscribed capsulated tumor, firm to touch
with pinkish appearance
Types of fibroids tumors
Intramural fibroids
- most common
- grow in the wall of the uterus.
Subserosal fibroids
- grow on the outside of the uterus.
- can cause pain due to their size or pressure on nearby organs
Submucosal fibroids
- least common
- grow just underneath the uterine lining and can crowd into the uterus cavity
- lead to urgency, heavy bleeding, infertility, back and pelvic pain with other more serious complications.
Pedunculated fibroids
-grow on small stalks inside or outside the
uterus.
Diagnostic Evaluations of fibroids tumors
Health history - Menstrual pattern - Fertility and child bearing desires - Pain assessment
Pelvic examination (Bimanual examination) - Detect for abdominal lumps
Diagnostic laparoscopy
- To visualize subserosal fibroids (perimetrium lining)
Ultrasound or MRI
- MRI will be able to detect fibroids which is not visible
during an ultrasound
Hysteroscopy ( submucosal fibroids)
- Inspection of uterus cavity
Blood test
- FBC to rule out anaemia
- TSH (thyroid problem)
Rationale for laparoscopy for pt with fibroids tumors
To visualize subserosal fibroids (perimetrium lining)
Rationale for hysterescopy for pt with fibroids tumors
Hysteroscopy ( submucosal fibroids)
- Inspection of uterus cavity
Definition of endometriosis
Endometriosis is the presence of functioning
endometrial glands and stroma outside the uterine
cavity, most often in the pelvic peritoneal cavity
Pathophysiology of Endometriosis
Metaplasia Theory:
• Endometrial tissue develops from embryonic epithelial cells due to hormonal or inflammatory changes: a layer of cells surrounding the ovaries and other cells within the pelvic region are able to change into endometrial cells.
Retrograde Menstruation Theory:
• Menstrual tissue backs up through the fallopian tubes during menses and implants on various pelvic structures due to contraction of smooth muscles.
Transplantation Theory:
• Endometrial implants spread via lymphatic or vascular channels.
Diagnostic evaluation of endometriosis
Health History
• Menstrual pattern
• Fertility status & child bearing desires
• Pain assessment
- Pelvic examination (Bimanual Examination)
• To detect fixed tender nodules round uterus - Full Blood Count (FBC)
• Rule out infection
• Check for anemia due to heavy menses - Diagnostic laparoscopy
• small incision made on the abdomen with camera
• guided instruments inserted to view the pelvic cavity
Definition of ovulation
Expulsion of an ovum from the ovary on
spontaneous rupture of a mature follicle as a
result of cyclic ovarian and pituitary function
It is usually occurs on or about the 11th to the
14th day before the next menstrual period.
Definition of fertilization
Occurs when a sperm penetrates an ovum and unites
with it, each with 23 chromosomes Produces a
zygote with 46 chromosomes
Sperm provides paternal chromosomes
Oocyte provides maternal chromosomes,
Occurs in upper third of uterine tube ( ampulla)
During transport through the fallopian tube, the zygote
undergoes rapid mitotic division or cleavage
Definition of implantation
It involves the attachment, penetration and embedding of the blastocyst in the lining of the uterine wall (endometrium)
Definition of embryonic stage
Development of membranes and
placenta
This stage lasts from day
15 until approximately 8
weeks after conception
End of 8 weeks, all the
organ systems and
external structures are
present
Definition of hyperemesis gavidarum
It refers to persistent and severe vomiting leading to fluid
and electrolyte disturbance, marked ketonuria, nutritional
deficiency and weight loss
Persists beyond the 20th week of pregnancy
(most common in first trimester)
Clinical manifestations of hyperemesis gavidarum
Persistent nausea and vomiting
Inability to retain food and fluids
Significant weight loss ( more than 5% prepregnant
weight)
Dehydration as evidenced by dry tongue and mucous
membranes, decreased turgor of the skin, scant and
concentrated urine
Electrolytes and acid-based imbalanced
Ketonuria
Nursing diagnosis of Hyperemesis Gavidarum
Fluid volume deficit related to severe vomiting
Impaired nutrition, less than body requirement related
to inability to tolerate fluids and foods
Anxiety related to ineffective coping of persistent and
severe vomiting
Nursing Intervention of Hyperemesis Gavidarum
Fluid and nutritional balance:
Monitor vitals signs for dehydration such as tachycardic and hypotension.
Assess for signs and symptoms for dehydration such as dry lips and mucosa, decreased skin turgor and sunken eyes to determine the severity of dehydration.
Maintain Nil By Mouth (NBM) status to allow GI tract to rest.
Administer antiemetic drugs as ordered e.g. Maxolon, IV Zofran.
Administer IV Dextrose 5% in lactated ringer
Monitor intake and output chart to assess fluid balance and determine fluid replacement.
Daily weight to assess fluid balance.
Introduce small amounts of oral fluid and food gradually when patient’s nausea and vomiting subsided for better toleration.
Comfort:
- Ensure comfort in oral cavity by providing oral hygiene, lips therapy and oral gel.
- Grouping nursing care to avoid unnecessary disturbances to provide maximum rest.
3.Emotional support
Listen to her concerns and feelings by answering all her questions
Teach the patient about therapeutic lifestyle changes like avoid stressors
and fatigue
Health teaching
Take frequent, small amounts of food and fluid, to prevent the
stomach becoming too full
Eat easily digested carbohydrates, e.g. crackers to avoid empty
stomach
Sitting upright after meals at least 2 hours after eating, to reduce
gastric reflux into the esophagus
Avoid noxious stimuli to prevent triggers that cause nausea and
vomiting
Nursing Intervention of Hyperemesis Gavidarum ( Fluid and nutritional balance )
Fluid and nutritional balance
Monitor vitals signs for dehydration such as tachycardic and hypotension.
Assess for signs and symptoms for dehydration such as dry lips and mucosa,
decreased skin turgor and sunken eyes to determine the severity of dehydration.
Maintain Nil By Mouth (NBM) status to allow GI tract to rest.
Administer antiemetic drugs as ordered e.g. Maxolon, IV Zofran.
Administer IV Dextrose 5% in lactated ringer
Monitor intake and output chart to assess fluid balance and determine fluid
replacement.
Daily weight to assess fluid balance.
Introduce small amounts of oral fluid and food gradually when patient’s nausea
and vomiting subsided for better toleration.
Nursing Intervention of Hyperemesis Gavidarum ( Health teaching)
Take frequent, small amounts of food and fluid, to prevent the
stomach becoming too full
Eat easily digested carbohydrates, e.g. crackers to avoid empty
stomach
Sitting upright after meals at least 2 hours after eating, to reduce
gastric reflux into the esophagus
Avoid noxious stimuli to prevent triggers that cause nausea and
vomiting
Nursing Intervention of Hyperemesis Gavidarum ( Comfort )
Ensure comfort in oral cavity by providing oral hygiene, lips
therapy and oral gel.
Grouping nursing care to avoid unnecessary disturbances to
provide maximum rest.
Nursing Intervention of Hyperemesis Gavidarum (Emotional support)
Listen to her concerns and feelings by answering all her questions
Teach the patient about therapeutic lifestyle changes like avoid stressors
and fatigue
Mild pre-eclampsia
BP: ≥ 140/90 mmHg but less than
160mmHg; diastolic greater
than 90mmHg but less than
<110mmHg
Proteinuria: Trace to 1+
Oedema: Trace to 1+ pedal, if present
Headache: Absent
Epigastric pain: Absent
Visual problems: Absent
Reflexes: Maybe normal
Serum creatinine: Normal
Platelets: Normal
Liver enzymes: Normal or minimal
Severe pre-eclampsia
BP: ≥ 160/110 mmHg and greater
Proteinuria: ≥ 2+ to 3+
Oedema: Oedema ±; oedema of face or
hand is significant
Headache: Severe
Epigastric pain: Present
Visual problems :Blurred, photophobia
Reflexes: Hyperreflexia >3+ possible
ankle clonus
Serum creatinine: elevated
Platelets : Decreased
Liver enzymes: Markedly elevated
Clinical Manifestation of severe pre-eclampsia
Central Nervous System:
Severe headache (brain edema and small cerebral haemorrhages)
Hyperactive deep tendon reflexes (CNS irritability)
Eyes:
Visual disturbances (blurred or double vision or ‘spots before the eyes’) due to arterial spasm and edema surrounding the retina
Urinary tract:
Reduces urine production and worsens hypertension due to decrease
blood flow to the kidneys
Respiratory system:
▪Dyspnoea due to pulmonary edema (accumulation of fluid in the lungs)
Gastrointestinal system and liver:
▪Epigastric pain or nausea due to liver edema, ischaemia and necrosis
Blood clotting:
▪HELLP syndrome
▪Involves haemolysis, elevated liver enzymes and low platelets