Common causes hindlimb lameness: Stifle Flashcards

1
Q

What is always a key differential for hindlimb lameness in SA?

A

cranial cruciate ligament disease

rare in cars
2-10y most common

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2
Q

Where is the cruciate ligament, what is the origin/insertion?

A

intra-articular but extra-synovial

O: caudomedial aspect of lateral femoral condyle
I: cranial intercondylar area of the tibia

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3
Q

What are the 3 types of cranial cruciate ruptures?

A

traumatic avulsion (yank a piece of bone attached to ligament) - uncommon

traumatic rupture - very uncommon

degenerative weakening

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4
Q

What is the importance of the cranial cruciate ligament?

A

role: knee stability

craniotibial translation
internal rotation

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5
Q

What do we see in 1/3 of dogs with cranial cruciate ligament disease?

A

damage to meniscus, 50% dogs have complete rupture
medial meniscus

painful on stifle extension!
best managed by surgical resection

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6
Q

What are the menisci?

A

2 C shaped fibrocartilage pads (medial and lateral)

load bearing/shock absorbing
collagen fiber arrangement converts compression into tension

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7
Q

Where are the stifle menisci attached?

A

medial to tibia
lateral to femur

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8
Q

What makes menisci disease difficult?

A

blood supply only in outer rim = poor healing
good nervous innervation = PAINFUL

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9
Q

What do we look for when assessing if a patient is lame in a hindlimb?

A

Calcaneal dip: Sinks on Sound
Hip hike: Lifts on Lame

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10
Q

What should we assess in a stifle examination?

A

effusion
medial buttress
patellar tracking
crepitus (ROM)
+/- pain
stability tests

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11
Q

What is the most reliable stability stifle test and how is it performed?

A

cranial draw

grip tightly on to bone patella-back femur and tibial crest - back tibia

standing or lateral

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12
Q

How do we perform the tibial thrust?

A

less reliable, well tolerated in conscious animals, easier in bigger dogs, simulates walking

hand over femur, finger over patella, push up paw
should feel finger being pushed forward: when jumps forward its abnormal

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13
Q

What is the conservative management/ treatment for cruciate ligament disease?

A

None really
maybe for dogs less than 15kg
no response in 8 weeks = surgical stabilisation
if meniscal injury it requires surgical tx

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14
Q

What is the role of cranial cruciate ligament surgery?

A

to stabilise the joint
stop/reduce tibial thrust

will not stop the progression of osteoarthritis

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15
Q

What are the 3 types of CCL surgeries?

A

intra-articular
extra-articular
osteotomy techniques

whatever is chosen: MENISCAL INSPECTION IS MANDATORY since over 50% of CCL ruptures have meniscal damage

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16
Q

What is the goal of extra-articular CCL surgery?

A

stops instability whilst fibrosis develops which is what will permanently stabilise the joint since the suture will always snap

17
Q

What is the principle of osteotomy techniques for CCL surgery?

A

Femoral condyle is a ball rolling down the slope of the tibial plateau = unstable
flatten slope = stable knee

neutralises tibial thrust but the cranial draw test would stay positive

18
Q

What are te 2 most common osteotomy techniques for CCL surgery?

A

TPLO: tibial plateau levelling osteotomy
TTA: tibial tuberosity advancement

19
Q

What is the prognosis for dogs with CCL tears?

A

around 50% get bilateral rupture in 2 years
90% dogs get to 90% of pre-rupture activity post surgery

OA will progress irrespective but doesn’t always mean lame
generally good outcomes

20
Q

What surgical technique is superior for CCL tears?

A

TPLO > TTA = Extrascapular > Intrascapular

21
Q

How does the patella function?

A
22
Q

How do patellar luxations usually arise?

A

developmental not congenital
usually medial

23
Q

What can cause patellar luxations?

A

problems with correct quadriceps mechanism development (possibly from hip/femoral development)

uneven pressure on physes (alteration growth dynamics)

boney deformation follows (lateral bow distal stifle, compensatory deviation tibia, skeletal torsion/angular deformities)

groove development needs pressure: affects depth and alignment during development

24
Q

What is the signalment for patellar luxation?

A

common cause of intermittent lameness in small dogs usually quite young

occurs in cats but often not clinical

25
Q

What is the clinical presentation for patellar luxations?

A

classical: intermittent non-weight bearing significant periods of normal, the “skipping” lamness

collapse episodes
abnormal gait/holding limb rotated in severe
chronic lameness

26
Q

What should the orthopaedic examination on a patellar luxation look like?

A

standing
- no neuro deficits
- comfortable hips
- tarsi ok, comformtable tarsus no instability
- stifle: alignment of patellar feels ok or abnormal
- no tibial thrust/cranial draw
- minimal effusion
- +/- muscle atrophy/ joint effusion

27
Q

How do we examine the patella?

A

Palpate limb standing
determine alignment: tibial crest-patella ligament - patella

work out where the patella is at rest: IN vs OUT

medial luxation: may have genu vara (bow legged) +/- stifle hyperflexed
lateral luxation: may have genu valga (knock kneed) +/- stifle hyperflexed

walk a few steps and repeat
lateral recumbency, stifle extended, patella manipulated medially and laterally for laxity
flex and extend stifle with rotation and manipulate patella

28
Q

How can we assess pain on luxation/retropatellar pain?

A

push patellar deep into groove
may help surgical decision making
indicate severity of change/damage

29
Q

What are the 4 grades of patellar luxation?

A

1: patella IN, returns in automatically (incidental)
2: patella IN, stays out (spontaneous)
3: patella OUT, can be returned into groove (abnormal stifle function)
4: patella OUT, can’t be returned into groove (lameness with crouched stance/gait)

30
Q

How helpful are radiographs for patellar luxation?

A

they could be normal
clinical palpation is most important diagnostic

could document bone deformities, OA, effusion, etc
help with surgical planning

31
Q

What are the criteria ofr patellar luxation surgery?

A

not simply grade but grade influences prognosis
make global view on clinical impact, may not influence progression of oA, talk to the owner
is there a clinical problem? lameness? frequency? pain? OA?
20% complication rate with patellar luxation surgery

32
Q

What patellar luxation cases are NON-surgical?

A

incidentally identified low grade (1 or 2) with no lamness, no discomfort on patella manipulation/retropatellar pressure, happy life

highly infrequent skipping lameness (once a month) may benefit from physiotherapy: improving quadriceps mechanism = patellar stability

33
Q

What patellar luxation cases are surgical?

A

+/- all grade 4 and 3s

any mild patella luxation 2 with pain on manipulation, retropatellar pressure pain, frequent intermittent lameness (weekly), consistent lameness

34
Q

What are the 4 procedures that can deepen the patellar groove?

A

trochleoplasty
chondroplasty
wedge recession sulcoplasty
block recession sulcoplasty

35
Q

What is a tibial tuberosity transposition and when is it used?

A

intra-operative assessment - post trochleoplasty: straighten femur and tibia, flex/extend stifle

a deviated patellar tendon suggests needs transposition
re-aligned bone stabilised with pin and tension band wire

36
Q

How do we manage severe patellar luxations in skeletally immature animals?

A

ideally wait over 10months
or 2 stage: trochleoplasty + soft tissue augmentation and later TTT

severe luxation could cause more skeletal abnormalities but tibial tuberosity transposition in young could lead to abnormal distal positioning of the insertion of the patellar tendon

37
Q
A