Cognitive theories of memory: L13 Flashcards

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1
Q
  1. Matlin definition of memory

2. Ashcroft definition

A
  1. maintaining information over time

2. the mental processes of acquiring and retaining information for later retrieval

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2
Q

define procedural memory

A
  • storage of skills and procedures
  • important in motor performance
  • supported by memory systems that are independent of hippocampal formation
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3
Q

define declarative memory

A
  • accumulation of facts/data from learning experiences
  • processed by various systems, feeding into hippocampal formation
  • its relational (new information about e.g. cats combines with previously acquired information)
  • activation of declarative memory = activation of related memories
  • memory activation can be independent of environment
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4
Q

whats the job of the hippocampal formation

A

create relationships between 2 pieces of information e.g. 2 concepts, an emotion + event experienced

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5
Q

serial memory model:
1. Atkinson-shiffrin model :
3 components

A
  • sequential order over time
    1. sensory memory
    2. working memory
    3. long term memory
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6
Q

Atkinson-shiffrin model, whats needed for each step?

A
  1. attention needed for sensory
  2. attention + rehearsal for working
  3. first 2 steps-> for long term
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7
Q

memories that exist in long term represent?

A

individuals perception of events, affected by interpretation and mental state

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8
Q

Serial model: levels of processing -

Craik and Lockhart

A
  • information retained according to level of processing it has undergone:
    1. shallow-deep
    2. maintenance (regurgitation) vs elaborative (imbedded in semantic framework) rehearsal
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9
Q

Serial model:

Tulving’s model of memory

A

Long term memory:

  1. procedural
  2. episodic (declarative = recall events)
  3. semantic (declarative = knowledge/facts)
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10
Q

Parallel model:

Parallel distributed processing

A
  • memory is activation of connections in different areas
  • strength between different nodes represents the memory itself
  • learning = strength of connections between relevant sites is changed
  • emphasis on the connection between the elements & strength of these connections
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11
Q

comparison of models of memory

A
  • not one model accounts for all data that exists

- serial models are most useful for studying amnesia

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12
Q

Episodic memory definition

A
  • “time travel”
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13
Q

Tulving’s conceptualisation of episodic memory

- episodic memory works by…

A

-allowing an individual to re-experience previous experiences

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14
Q

define autonoetic awareness

A

reexperiencing oneself in the autobiographical past

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15
Q

episodic memory: what do operations depend on?

A
  • semantic and other forms of memory
  • shares neural mechanisms & cognitive processes with other systems
  • also subserved by specific mechanisms not involved in other systems
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16
Q

Define semantic memory

A
  • ‘knowledge memory’
  • system that makes acquisition, retention and use of factual information possible
  • not purely concerned with language
  • no autonoetic awareness of the personal past
17
Q

how independent are episodic and semantic memory systems?

A
  • parallel with declarative memory (Squire & Zola)

- not parallel (Tulving)

18
Q
Squire & Zola:
declarative memory 
- dependent on
-damage =
-> evidence
A
  • dependent on the hippocampal system
  • damage results in equal impairment to episodic and semantic memories
  • > Amnesic patients have equal difficulties with ‘event’ and ‘fact’ memory
19
Q

Tulving:

declarative memory - serial parallel independent (SPI) hypothesis

A
  • encoding into episodic memory relies on semantic system (& the other way too)
  • episodic memory is a ‘unique’ extension of semantic memory
  • retrieval is independent, can be supported by either system or both
20
Q

Tulving SPI model flow chart

A

In -> PRS -> semantic -> episodic

PRS -> identify objects -> out

semantic -> know the present -> out

episodic -> remember the past -> out

21
Q

Dissociations:

1. no dissociation

A
  1. damage X = affects A & B equally. Damage Y =affects A & B equally
22
Q
  1. single dissociation

- > example

A
  1. damage X = affects A, not B. Damage Y = affects A & B equally
23
Q
  1. double dissociation
A
  1. damage X = affects A, not B. Damage Y = affects B not A
24
Q

Vargha-Khadem study

- what did it examine

A
  • seminal study, group of children

- 3 patients suffered early bilateral medial temporal lobe injury

25
Q

Vargha-Khadem study

-Neuropathology (2)

A
  1. assessed with MRI and spectroscopy
  2. all 3 cases had:
    - abnormally small bilateral hippocampi
    - intact extra-hippocampal temporal lobes
26
Q

Vargha-Khadem study

- memory

A
  • 3 cases: impaired memory function relative to intellectual capacity
  • memory assessed with standard clinical measures of memory (tests of episodic memory)
27
Q

Vargha-Khadem study

-memory deficits included

A
  • spatial memory
  • temporal memory
  • episodic memory
28
Q

Vargha-Khadem study

- what remained “normal”

A
  • academic achievements

- factual knowledge = normal range -> extensive semantic information

29
Q

Vargha-Khadem study

- conclusions

A
  • intact semantic memory
    -impaired episodic memory
    = episodic memory relies on hippocampal circuit
30
Q

Vargha-Khadem’s study & Tulving’s model…

A
  • fit together
  • i.e. episodic memory function relies on semantic memory
  • semantic memory function does NOT rely on intact episodic memory
31
Q

Episodic - semantic dissociation + Vargha-Khadem’s study & Tulving’s model…

A

evidence exists suggesting these results to not necessarily be true -> double dissociation also exists in the literature
= interaction between these two systems isn’t clearly understood

32
Q

Episodic memory relies on what?

A

the hippocampal circuit