Clinical Psychology Flashcards
Guidelines in HCPC
- Act in the best interests of the service users
- Respect every service user and includes their role in the diagnostic and therapeutic roles.
- Recognise boundaries that are appropriate and they must understand the power situation exists between themselves and their clients.
Strengths of cross-sectional studies
Cross-sectional studies are quicker to conduct than longitudinal studies because they use a variety of participants at one point in time rather than waiting to follow them through their life.
Weaknesses of cross sectional studies
One weakness is that the participants are different so they suffer from participant variables which means the conclusions made could be due to individual differences between participants.
Schizophrenia
Biological treatment
- A biological treatment is anti-psychotic drugs which aim to change the chemistry in the brain.
- They block dopamine receptors so minimising the effect of dopamine.
- A patient could take the anti-psychotics in syrup or tablet form.
- If patients forget to take them then medical practitioners could inject the anti-psychotics.
Procedure of Rosenhan (1973)
- Aimed to see if diagnosis of mental disorders was valid/reliable.
- to illustrate experimentally the problems involved in determining normality and abnormality
- All 8 pseudopatients were admitted with either a diagnosis of schizophrenia or manic depression (now bipolar)
- Rosenhan said the pseudopatients acted normally once admitted e.g. making conversations with fellow patients.
- Once admitted the pseudopatients did not claim to hear voices anymore
Evaluation of Rosenhan (1973)
- As they did not know they were part of a study the staff did not give consent to take part in the study.
- The validity of the results is higher as a variety of real hospitals were selected for the observations.
- As the pseudo patients were admitted, the staff had no reason to think they were faking it, as healthy people do not say they hear voices that are not there.
- Staff could be distressed, and may question their work which would negatively impact on real patients.
- Follow up study furthers the
- 8 ppts used in 12 hospitals, meaning the study was replicated to produce similar findings giving it reliability.
- Hospitals varied allowing for generalisation and since 12 hospitals in different states were used, this strengthens the findings further.
- Carried out 30 years ago. Methods of care and diagnosis and improved.
Unipolar depression
Biological explanation - Monoamine theory
- The monoamine hypothesis claims that low level of monoamines cause depression.
- Monoamines consist of noradrenaline/norepinephrine, dopamine and serotonin.
- Serotonin regulates the other neurotransmitters, so low levels of serotonin leads to lower levels of dopamine.
- Low levels of serotonin cause erratic brain function and thought patterns.
- Low levels of noradrenaline cause lower levels of alertness and arousal, symptoms of depression.
- Low levels of dopamine cause lack of concentration and focus.
- Low levels of monoamines could be caused because the reuptake mechanism recaptures the neurotransmitters before they reach the receptor sites.
- Depressed people could release too much monoamine oxidase so the monoamines are broken down too fast.
Unipolar depression
Evidence for biological explanation
- Drevets et al (1999) found reduced serotonin receptor-binding potential in unmedicated depressed patients.
- Versiani et al (1999) found that patients who had noradrenaline reuptake inhibitors increased their mood if changing the biology can reduce symptoms there may be a biological cause.
- Bunney et al (1970) found that urinary levels of noradrenaline decreased during episodes of depression.
- Bell et al (2001) depleted levels of tryptophan and found this caused a relapse of symptoms in depressed patients showing that changing the biology of a person can cause the symptoms of unipolar depression.
- Delgado (2000) found that monoamine depletion did not make symptoms worse in depressed patients not taking medication.
- Angoa-Perez (2014) found that mice without the gene for tryptophan did not show signs of depression.
- Delgado et al (1990) used a special diet to lower the level of tryptophan found that depressive symptoms returned for 67% of ppts.
- Anti-depressants which increase the monoamines alleviate depression showing reduced monoamines are a possible cause.
- However, the level of neurotransmitters rises very quickly once an individual is on medication.
- There are alternative theories such as the cognitive therefore it is not the only explanation.
Unipolar depression
Weaknesses of biological explanation
- Most evidence for abnormal levels of chemicals in the brain being a cause of depression is correlational so we cannot know if the change in chemicals cause unipolar depression or the other way round.
- Therefore whilst low levels of monoamines may be one cause of depression it cannot be said to be the only cause.
4 Ds
Deviance - when behaviour is considered abnormal in society. May change across time and place as social norms change.
Dysfunction - if behaviour is significantly interfering with a persons life.
Distress - the extent to which the behaviour is causing distress to the individual.
Danger - danger to themselves and danger to others.
Evaluation of the 4Ds
- Subjectivity involved in these judgments as the clinicians must interpret how the patient is being affected. Info given may incomplete or biased
- Reliability. standardising interviews is crucial to establish a reliable diagnosis.
- Clinical judgement. Person views must be kept to a minimum. A team approach to diagnosis can help.
- Validity. Interviews are unstructured or semi-structured. There is potential for bias in interpretation of interviews.
ICD
- Deals with mental disorders and diseases.
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DSM
- Looks at patterns of symptoms, including distress.
- Do not only look at physical symptoms but is linked to the distress of the person and their inability to function normally
Reliability of classification systems
- DSM and ICD undergo continuous review. Updates ensure that it is possible to make a more accurate diagnosis.
- Use of structured interviews increase reliability as clinicians use the same questions.
- Improve reliability when clinicians collaborate with other clinicians when making diagnosis. This increases inter-rater reliability.
- Gold Stein looked at 199 patients along with other clinicians, re-diagnosed them using single-blind technique and found reliability in diagnosis.
- Wilson (1993) suggests that DSM III was developed precisely to tackle the unreliability of the previous manual.
Unreliability of classifications systems
- Have distinct differences. Therefore reliability is reduced.
- Andrews et al (1999) found there is only 68% agreement between the ICD and DSM
- Rosenhan (1973) found that diagnosis were flawed, as staff were unable to tell mentally disordered patients apart from the mentally healthy.
- Reliability of diagnosis varies for different disorders. It is goof for depression and worse for PTSD, reliability is lowered.
- Beck found 54% concordance in agreement between psychologists who re-diagnosed 153 patients with schizophrenia, so depends on the mental disorder.
Symptoms of schizophrenia
Positive
- Hallucinations
- Delusions of grandeur is when they think they are in a position of power.
- Thought disorders make someones speech hard to follow. They lose concentration and can be disorganised.
Negative
- Avolition: lack of energy and apathy
- Social withdrawal.
- Not looking after appearance of self.
Schizophrenia
Neurotransmitter theory
- Increased number of dopamine, excess dopamine is believed to cause schizophrenia with more dopamine activity and the synapse.
- This over activity at synapse causes positive symptoms such as hallucinations and delusions.
- Lower dopamine activity due to less dopamine receptors in the mesocortical pathway which connects midbrain to frontal lobes is associated with negative symptoms such as lack of motivation and concentration.
- Changes in dopamine sensitivity in the brain may arise due to genetic inheritance or brain lesioning.
Schizophrenia
Supporting evidence for neurotransmitter theory
Randrup and Munkvad (1966)
- Injected rats with amphetamine to raise dopamine levels.
- Behaviour changed, became more aggressive and isolated showing that changing dopamine levels resulted in psychotic type behaviour.
- Found that amphetamine acts on the brain by increasing levels of dopamine.
Lieberman et al (1987)
- 75% of patients with schizophrenia show new symptoms or an increase in psychosis after using drugs such as amphetamines.
- Only a small proportion of people who regularly use these drugs suffer from psychotic symptoms, which suggests some people’s brains react differently to the presence of dopamine.
Supported by post-mortem examination of schizophrenic brains who show a higher density of dopamine receptors in certain parts of the brain compared to non-schizophrenics.
Schizophrenia
Evaluation of genetic theory
Gottesman (1991)
- Increased concordance rate in MZ twins suggests the significance of genes in schizophrenia. However as it is not 100%, genetic factor is not the only affecting factor.
- Family studies establish that there is a inherited component of schizophrenia. However research such as Tamminga and Sehulz (1991) have failed to isolate a single gene responsible for the disease. Family studies are criticised for not considering that schizophrenia may be the result of environmental influences.
Treatments for schizophrenia
Typical anti-psychotic drugs
- Combat positive symptoms of schizophrenia.
- Bind to dopamine receptors, thus blocking their action and not stimulating them
- By reducing stimulation of dopamine system in the brain, drugs eliminate the positive symptoms experienced by patients.
Treatments for schizophrenia
Evaluation of typical anti-psychotic drugs
Davis (1990)
- Analysed results of 29 studies, using 3519 people. Found that relapse in 55% of patients whose drugs were replaced by a placebo compared to only 19% of those who remained on anti-psychotics.
Barlow and Durand (1995)
- Found that chloropromazine was effective in reducing symptoms in 60% of cases but mainly reduced positive symptoms.
Lieberman et al (2005)
- Found that many patients stop taking medicine because of the side effects.
- Side effects include: weight gain, diabetes, insomnia
Treatments for schizophrenia
Atypical anti-psychotic drugs
- Bind to D2 receptors rather than permanently blocking the dopamine action.
- They temporarily bind to the receptors and rapidly dissociate to allow normal dopamine transmission.
- They also block serotonin receptors.
Treatments for schizophrenia
Evaluation of atypical anti-psychotic drugs
- They seem to have a positive effect on positive and negative symptoms. This may be due to the effect on serotonin.
- Picker et al (1992) found that clozapine was most effective in reducing symptoms when compared to other neuroleptics and a placebo in patients who resisted treatments.
- Melzer et al (2004) studied effectiveness of drug treatment for schizophrenia symptoms.
Compared to a placebo, Haloperidol and 4 other atypical drugs.
Found that Haloperidol gave significan improvement in all aspects of functioning compared to placebo, yet showed greater side effects.
2 of the atypical drugs compared seemed to alleviate both positive and negative and with fewer side affects, the other two did not.
Side effects include: weight gain, sleeping problem..
Schizophrenia
Strengths and weaknesses of drug therapy
- Have strong biological evidence about causes of schizophrenia.
- If the drugs have passed clinical trials, this must show that they were effective.
- Drugs have thought to be better that treatment from 1950s such as ECT and insulin shock therapy, because of drug therapy, it is regarded as more humane.
- The side effects can be severe and permanent such as tardive dyskinesia which 30% of patients develop and of which only 30% recover.
- Individual differences can affect the effectiveness of treatments.
- Ethical point of view, drugs have been called a ‘chemical straight jacket’ as it is form of social control by society.
Schizophrenia
Evaluation of biological treatment
- Anti-psychotics allow patients to stay in society rather than become institutionalised.
- Emsley (2008) found 84% of patients on anti-psychotics had at least 50% reduction in symptoms if they were given early enough.
- Anti-psychotics have serious side effects such as a decrease in motivation so schizophrenics may prefer not to take them.
- Rosa et al (2005) found only 50% of patients complied with taking their anti-psychotics.
Schizophrenia
Cognitive theory
Symptoms are the cause rather the consequence of the disorder
- Cognitive deficits: these impairments in thought processes such as perception, memory and attention. The word deficit means something is missing or lacking.
- Cognitive biases: these effect the way people see and interpret the world.
Schizophrenia
Research evidence for cognitive theory
Pickering (1981)
- Suggested that catatonic schizophrenics lack interaction with the outside world occurs because it is the only way in which the amount of sensory information can be kept at a manageable level.
Maher (1968)
- Studied the bizarre language used by schizophrenics. He identified vulnerable words that cause the sufferers to respond in a personal way to the word association. Bizarre language was conducted to be a result of disordered language processing, which is a cognitive factor.
Schizophrenia
Neuro-physiological theories
Frith (1992)
- Proposed that cognitive impairments results in sufferers being unable to distinguish between action brought by external forces and those generated internally.
- He believes that the mechanism which allows us to do this is controlled by a connection between the frontal areas of the brain that control action and the posterior areas that control perception.
- These claims are supported by using brain scanning techniques to monitor blood flow in brain of schizophrenics when a given specific cognitive tasks.
Helmsey (1993
- Suggests that the central deficit in schizophrenia is breakdown in the relationship between information that has been stored and new coming information.
- In schizophrenia these schema’s are not activated so that sufferers are subjected to sensory overload and so do not know which aspects of the situation to tend to and which to ignore.
- Helmsey also suggested that internal thoughts are not recognised as arising from memory and so are attributed to an external source and experienced as auditory hallucinations.
Schizophrenia
Cognitive bahvioural therapy
- Encourages patients to engage in activities they avoid.
- Break the cycle of maladaptive thinking, feelings and behaviour.
- Focus on current problems and difficulties, instead of focusing on the cause of distress and past symptoms.
Schizophrenia
Research evidence in cognitive behvioural therapy
Chadwick (2000)
- Studied 22 schizophrenics who heard voices. They each had 8 hours of CBT and all had reduced negative beliefs about how powerful the voices were, and how much the voice controlled them, thus allowing them to live with the auditory hallucinations better.
Schizophrenia
Cultural differences
- Bhugra et al (1999) found differences between Asian and whites.
Asians were more likely to neglect activities, lose appetite and commit suicide.
More whites reported auditory hallucination - McCabe and Priebe (2004) compared different explanations models of illness among people with schizophrenia from 4 different cultural backgrounds: African-Caribbeans, West Africans, Bangledeshi and Whites.
Whites showed biological cause more than non-whites group who were more likely to give supernatural or religious reasons.
Schizophrenia
Developmental differences
- David Lewis (1996) claims children who later develop schizophrenia have ‘premorbid’ behaviour, such as learning difficulties and behavioural problems.
By age 2 they are less likely to be walking and talking than other children. - Lindermer et al (2001) found that patients with late onset schizophrenia are more likely to be female, with less negative symptoms.
Schizophrenia
Gender differences
- Schizophrenia occurs equally in men and women. Kaplan et al (1994) describes the peak ages of onset among men being 15-25 years. The peak for women is 25-35 years but they also peak again in their 40s and 60s.
- Szymanstei et al (1995) found women were diagnosed more frequently with disorganised subtypes of schizophrenia than in men.
Schizophrenia
Personality differences
- High N (neuroticism) are prone to mood swings and stress and easily feel anxious. This is linked with schizophrenia.
- High E (extrovertism) are outgoing and crave excitement. This is linked to absence of schizophrenia .
Schizophrenia
Longitudinal study
Harvey et al (1999)
- Study looked at 326 geriatric, chronic, long-stay patients. The patients showed cognitive and functional impairments that were in decline, post-illness onset.
- Lasted 30 months and 2 separate assessments of the patients were conducted.
- Cognitive and functional was measured using the clinical dementia rating.
- Participants who had less severe scores at the start of the study, 30% had declined further by second assessment.
- 7% of those appeared to improve in their functioning,
- Several characteristics at baseline assessment predicted increased rate for cognitive and functional decline, including lower levels of education, older age and more severe positive symptoms.
Longitudinal studies
- Asses the same group of ppts repeatedly over an extended period of time.
- This allows the researcher to look at how behaviour changes over time and developmental trends can be established.