CLINICAL APPS FINAL Flashcards
Chemo
N/V
muscositis
metallic taste
nutrition is important
Nurse must be certified
Large bore need is needed
Radiation
don’t remove the temporary tattoo
avoid sunlight
Thrombocytopenia
decreased platelets (150-450 normal range)
Risk for hemorrhage
Teach: soft bristle, electric razor, be careful ambulating
Neutropenic
overproduction of amature white cells.
Prevent infection: hand hygiene, avoid people, don’t eat fresh fruits and veggies, do not garden
Kids: monitor temp
Infection risk of chemotherapy
Bone marrow suppresion
Neutropenia
Anemia, thrombocytopenia risk
- bone marrow suppresion
- impaired clotting
- chemo induced n/v (CINV)
- chemo induced pheripheral neuropathy (CIPN)
- mucositis
- alopecia
- cognitive changes
Teaching for cancer
No fruits and veggies (fresh)
- hand washing education
- electric razors only
- soft bristle toothbrush
- mucositis
chemo- risk for infection, anemia, and thrombocytopenia.
pancytopenia = LOW everything
chemo brain
chemo kills all cells, not just cancer cells
Anemia
reduction in the # of RBCs, amount of HGB or HCT.
Anemia common causes
SCD, immunohemolytic anemia, iron deficiency anemia (nutrition related, vitamin B12 deficiency, folic acid deficiency, aplastic anemia, post-op anemia
in the elderly: most likely due to nutrition deficiency
Anemia s/s
lethargic
fatigue
Leukemia
loss of normal cellular regulation leading to uncontrolled production of immature WBCs (“Blast” cells) in the bone marrow.
May need a bone marrow transplant - Risk for infection
low platelet count common
avoid rectal temps
assess IV for bleeding
Multiple Myeloma
WBC cancer that involves mature B lymphocyte called a plasma cells that secrete antibodies.
Cells overgrown in the bone marrow
When they become cancerous they produce excessive antibodies called GAMMA GLOBULINS —> this leads to few RBCs, WBCs, and Platelets
Multiple Myeloma s/s
anemia
risk for infection
bleeding
Thrombocytopenia
reduced platelets
ex: Autoimmune (Idiopathic) Thrombocytopenia purpura (ITP)
Autoimmune (Idiopathic) Thrombocytopenia purpura (ITP)
circular platelets reduced even though platelet production is normal = platelets clump together leading to few platelets in circulation.
Blood will fail to clot when trauma occurs.
Tissue becomes ischemic –> can lead to RF, MI, Stroke
Blood transfusion and Hemolytic reaction
given over 3 hours
Hemolytic reaction- w/ PRBCs transfusion will cause fever, chills, red urine (hemoglobinuria), and itching
Arterial Blood Gases- Normal values
pH 7.35-7.45
pCO2 35-45
HCO3 21-28
O2 sat 94-98%
*Causes of Respiratory acidosis
COPD
Pneumonia
Atelectasis (collapsed lung)
Decreased resp stimuli (Anesthesia, Drug overdose)
*Respiratory acidosis s/s
“I can’t breath”
- hyperventilation –> Hypoxia
- rapid, shallow respirations
- headache
- hypErkalcemia
- dysrhythmias (^K+)
- dyspnea
- low BP w/ vasodilation
- muscle weakness, Hyperreflexia
Causes of Metabolic acidosis
DKA
Severe diarrhea
Renal failure
Shock
Metabolic acidosis s/s
headache decreased BP hypEkalemia muscle twitching warm, flushed skin (vasodilation) changes in LOC (confusion, ^ drowsiness) N/V diarrhea Kussmaul respirations (compensatory hyperventilation)
What goes up in acidosis
potassium levels (Hyperkalemia)
Hyperkalemia can cause
muscle weakness
potentiall life-threatning cardiac dysrhytmias
cardiac arrest
watch for oliguria (decreased urine output)
dehydration can cause hyperkalemia
causes of Respiratory alkalosis
hyperventilation (anxiety, PE, fear)
mechanical ventilation
Respiratory alkalosis s/s
seizures deep, rapid breathing lethargy & confusion light headedness hyperventilation N/V tachycardia HypOkalemia numbness & tingling of extremities low or normal BP
causes of Metabolic alkalosis
severe vomiting
excessive GI suctioning
diuretics
excessive NaHCO3
Metabolic alkalosis s/s
restlessness followed by lethargy dysrhytmias (tachycardia) compensatory hypoventilation confusion ( decreased LOC, dizzy, irritable) N/V diarrhea tremmors, muscle cramps, tingling of fingers and toes HypOkalemia
Foods high in potassium
orange juice, organ meats, fish, fresh fruits, dried fruits, beef, chicken, pork, milk, vegetables, salt substitutes
in ABGs what compensates first?
lungs
Echocardiography
non-invasive (like an ultrasound)
Cardiac Magnetic Resonance Imaging (MRI)
may use contrast or iodine (look for shellfish allergies) also look for kidney function before test.
Infective Endocarditis- infection priority and management
*Priority = dyspnea
Prophylactic antibiotics before procedures such as dentist
Can be cause from IV drug use
Rheumatic fever
caused by untreated strep infections –> leads to damaging of Mitral and Aorta valves.
treat strep quickly and take all of the antibiotics
affects: joints, skin, brain, seroud surfaces, and heart
Rheumatic heart disease
caused by rheumatic fever.
swishing heart sounds
echo typically ordered
permanent valve damage (Mitral and Aorta)
Nephrotic syndrome
kidney disorder that causes the body to excrete too much protein in the urine
Nephrotic syndrome clinical state
Proteinuria Hypoalbuminemia Hyperlipidemia Edema Massive urinary protein loss
Nephrotic syndrome lab finds
Massive proteinuria on dipstick GFR normal or high Total serum protein low, albumin low Serum sodium low Platelets may be elevated Hemogloin and Hematocrit normal or elevated
Complications of nephrotic syndrome include
infection
circulatory insufficiency
thromboembolism
(episodes often happen with viral or bacterial infection)
What is important to monitor in nephrotic syndrome
Strict I&O are essential, may be difficult to obtain in young children.
Infection is a constant source of danger to edematous children, particularly for those receiving corticosteroid therapy
what vaccine is recommended for children with nephrotic syndrome
pneumonia vaccine
VEAL CHOP
Variable decelerations = Cord compression (not okay)
Early decelerations = Head compression (okay)
Accelerations = Okay to have
Late decelerations = Placental insufficiency
Leopold maneuvers
palpate uterus through abdomen to determine fetal lie, fetal attidue, and the point of maximal impulse (PMI)
Point of maximal impulse (PMI)
where the FHR can be heard the loudest, where we place the external transducer to monitor FHR
Whats expected for FHR
HR or 110-160 bpm
moderate variability
excelerations may be present or absent
early decelerations may be present or absent
We DO NOT want to see Late decelerations or Variable decelerations in FHR
Excelerations
temorarily increase in FHR above baseline
its reassuring and no interventions are needed
Bradycardic FHR excelerations
due to:
interventions:
FHR below 110 bpm for at least 10 minutes
Due to: utero placenal insufficiency, umbilical cord prolapse, maternal hypotension, analgesic medications
Action: place pt in side lying position, administer oxygen, and notify provider
Tachycardic FHR excelerations
due to:
Inerventions:
FHR above 160 for longer than 10 minutes
Due to: infection, cocaine use, dehydration
Action: administer antipyretics, administer oxygen, possible IV fluid bolus
Early decelerations
Due to:
Intervention:
slowing of baby heart rate during contraction.
Due to compression of head when mother is having a contraction, this is an expected finding no interventions are needed.