#Clinical Flashcards
what are the 4 Ds
deviance
dysfunction
distress
danger
what is deviance
social norms= agreed ways of acting
those who break are abnormality
effected by culture; context, age and gender
eg those with sz and ocd hearing voices
what is dysfunction
intereferes with a persons everyday life is one way to disgnose mental disorders
dysfunction can affect their working life
inability to function normally, live life normally
rosenhal- failure to function
what is distress
abnormal because it upsets the individual
unhappy with symptoms - depression
can be thought to continuum ( persistant = more serious )
what is danger
danger to themselves or others
eg self harm suicide and violence
varying degrees of danger to other people and oneself
deviance strengths
effected by cultural differences
takes into account things such as gender and age making it more holistic
deviance weaknesses
subjective
Thomas szasz argues that it is a way of social control
reductionist
doesn’t consider the patients wellbeing
break in social norms which arent mental health issues
dysfunction strengths
objective through doctors tests DSM IV
definition is better as it considers life quality of the child
dysfunction weaknesses
subjective
things are dysfunctional without being a disorder- repeatedly getting drunk
distress/danger strengths
DSM IV global functioning
considers pateint life quality
distress/danger weaknesses
subjective
many behaviours are common even though they aren’t mental health issues
overal evaluation of the 4ds
DSM uses the four Ds- eg schizophrenia must have deviance and distress
not objective but used for disgnosis- issues with inter rater reliability
Timothy Davis stated that 5th D of duartion should be added
DSM description
created by APA in 1950s diagnostic criteria for mental illness ie depression 5/9 items looks at prevalance of each illness grouped by family such as psychotic illness/ affective updated every few years
ICD description
Worldwide All illnesses not just mental health Section 5- mental health issues has criteria list similar to the DSM each illness has incidence, prevalence, mortality and morbidity rates
Evaluation of DSM/ICD
G- ICD is cross cultural and worldwide so high validity
R- updates hinder the reliability of diagnosis but Rosenhan found reliability
A-entitles people to medical help
V-patients can lie lowering DSM validity (Rosenhan)
A01 Points for culture
culture=shared set of beliefs norms and values
one way in which it affects is how patients describe illness
live in a global world so we need to ensure correct diagnosis
shapes mental health of minorities- black people in UK more likely to be hospitalised
A02 Points for culture
4ds- different cultures view different things as deviant etc
DSM V ICD- later is worldwide, issues with reliabity with DSM.
Culturally BI- there are illnesses bound to specific cultures
Bias withing Cs- Black men over represented in UK mental health hospitals
A03 Points for culture- STRENGTHS
+DSM valid in korea - LEE
+high catatonia in Sri Lanka due to lack of treatment - CHANDRESA
+LIN- schizophrenia around the world shares more symptoms then what it differs in.
A03 points in culture- WEAKNESSES
- Escobar and Vega= DSM iv has strong western bias
- Burham= mexican born Americans have more auditory hallucinations
- Banyard= 5% population is black 25% of psychiatric population
Schizophrenia symptoms
two or more symptoms over a month including delusions and hallucinations
disorganised behaviour, aquditory and visual hallucinations, delusions
negative symptoms- flattened effect
features of schizophrenia
more men then women
effects men at a young age - late teens womne are mid 30s
1% chance of getting
Neuro transmitter explanation of Sz
Chemical imbalance in NT which causes excess signalling
high levels of dopamine linked
illegal drugs increase D2 receptors and produce schizophrenia like symptoms
decreased dopamine in mesocortical system causes negative symptoms
Strengths of NT theory of SZ
Anti psychotics reduce dopamine and work
Carlsson review study- linked dopamine to SZ
Amphetamines are agonist and produce SZ like symptoms
scientific and objectivce= measuring chemical imbalance
Weaknesses of NT theory of SZ
Cause and effect
only able to test through the blood or urine not brain
Amphetamines only cause pos symptoms- dopamine is reductionist
other theory such as cognitive
Carlsson found it was not just dopamine
Description of Genetic explanation of SZ
1% likelihood however this increases when related to someone with illness 48% - MZ twins
Deletion of COMT gene means D not regulated properly
deletion of section of C 22 causes digeorge syndorme in which 1/4 have SZ
c4 a specific gene. if you have it you have intensified pruning.
strengths of genetic explanation of schizophrenia
tienari-7% of adoptees had biological parent with sz
Gottesman- MZ twins 48% concordance rate
used alongside NT explanation
explains links between family members
weaknesses of genetic explanation of sz
reductionist- not just genes
concordance rate is not 100% showing environmental factors
ignores cognitive
weakness of twin studies- not always equal enviroments
what are the symptoms of depression
depressed mood decreased interest or pleasure in most activities weight change sleep pattern changes change in activity fatigue worthlessness suicidal ideation
what are the features of depression
1/5 affected each year
7-12% of population experience it in the year
7% but variation with age
more women effected 2:1 but more men commit suicide
what is the biological explanation of depression
Neurotransmitters
describe the NT explanation of depression
imbalance of NT such as serotonin and dopamine
low levels of serotonin linked to symptoms such as high anxiety and low mood
there may be too much reuptake of serotonin which has functional effect of lowering levels
low serotonin effects levels of other monoamines, like noradrenaline and dopamine
what are the strengths of the NT explantion of depression
Objective and empirical
antidepresseants increase monoamine levels and reduce depression symptoms
Drevets- found reduced serotonin receptor binding potential in depressed people
rosen et al- enzymes involved with breakdown of serotonin higher in depressed people
Weaknesses of the NT explanation of depression
cause and effect
Cognitive explanation
difficult to measure blood and urine levels reduces v
drugs dont work for everyone
Treatment aetiology drugs work but not necessarily the cause
Delgrado- monoamine depletion in depressed people didn’t make them feel worse
What is the non-biological explanation of depression
cogntiive
what does the cognitive explanation of depression state
depression is caused by faulty thinking which interprets how they view the world
catastrophising, all or nothing thoughts
maladaptive thought through learned experience
Beck’s negative triad- negative thought about oneself, future and the world
negative attributes are internal