Class 6 Flashcards

1
Q

Psychologically, the bottom line of trauma is a feeling of

A

utter helplessness.

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2
Q

Women prevalence vs men PTSD

A

women 2: Men 1

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3
Q

What type of trauma do men usually have

A

violent perpetrated by non-significant other; physical

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4
Q

What type of trauma do women usually have

A

violence perpetrated by significant other; sexual

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5
Q

x% of Canadian adults report some form of abuse before age 16

A

32%

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6
Q

Most prevalent form of childhood abuse

A

neglect

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7
Q

Prevalance of child abuse in mental health patients

A

70%

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8
Q

Automatic Response to Threat

A

12 msec: cortisol adrenaline =BP/ HR, blood flow, oxygenation, glucose

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9
Q

Cascade of survival responses

A

attach cry, flight, fight, freeze, feigned death

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10
Q

If the body has used an immobilizing defense,

A

, the nervous system does not get a cue to turn off
Amygdala keeps firing to signal persistence of threat
Catecholamines continue to be secreted, long after the threat has passed

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11
Q

3 main parts of brain

A

Humain brain: cortex, rational thinking
Mammal brain: limbic system, emotions
Reptile brain: brainstem, automatic response

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12
Q

Under threat, in the brain

A

limited access to prefrontal cortex, the reptile brain is running the show. The link between the amygdala and the hippocampus is broken. The memory gets stored in the implicit memory system by the amygdala where they’re encoded as sensations, smells, sounds, images, tastes, emotions, fragments of events
instead of in the explicit memory system in the hippocampus.

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13
Q

Usually brain encodes memory:

A

sensory input= thalamus= amygdala=hippocampus= prefrontal cortex

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14
Q

Amygdala has direct connection to

A

reptilian brain through hpa axis

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15
Q

After the encoded of the memory in the implicit memory system, the amygdalae acts like

A

a smoke detector, it sounds the alarm whenever it detects anything resembling the trauma

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16
Q

Truncated mobilizing defenses

A

Catecholamines
Hyper-responsive catecholamine system
↓ Cortisol
Hypo-responsive HPA axis

Hypervigilance
Increased startle
Hyperarousal

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17
Q

PTSD fMRI Findings

A
Smaller hippocampus
Reversible with treatment
Hyper-responsive amygdala
During Flashbacks:
R brain > L brain
↑ amygdala
↑ visual cortex
↓ Broca’s area
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18
Q

PTSD name all symptoms + number needed + specifiers

A
Intrusion: 1
Memories
Flashbacks
Nightmares
Distress with triggers

Avoidance: 1
Internal (memories, thoughts, feelings)
External (people, places, situations)

Mood/ cognition:2
Amnesia
Neg. beliefs (“I’m bad,” “World dangerous,” “Trust no-one”)
Self-blame
Persistent negative emotions (fear, anger, guilt, shame)
Difficulty experiencing positive emotions
Diminished interest
Detachment from others

Arousal: 2 
Hypervigilance
Startle reflex
Irritability / aggression
Reckless / self-destructive behavior
Poor concentration
Poor sleep

Specifiers
with dissociative symptoms
with delayed onset

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19
Q

Scales

A

PCL-5 - PTSD Checklist :20 items, self report, cut-off > 33
IES – Impact of Events Scale - DSM-IV : 22 items, self-report, cut-off > 24
CAPS – Clinician-Administered PTSD Scale :30 items, clinician rated, cut-off > 20

For dissociation:
DES: self-report 28 items
MDI:L self-report 30 items, need to request it

ACE Questionnaire for childhood aversity

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20
Q

Complex PTSD, what kind of trauma

A

Child abuse, neglect, combat, urban violence, concentration camps, battering relationships, forced dislocation, and enduring deprivation.

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21
Q

Complex PTSD symptoms

A
Core symptoms of PTSD and: 
Emotion regulation issues: Self-harm
Substance abuse
Eating disorders
Shoplifting, hoarding, gambling
Picking, trichotillomania
Outbursts
Relational difficulties: Reenactments
Isolation / mistrust
Abusive relationships / revictimization
Therapeutic ruptures
Parenting difficulties

Alteration in consciousness:DID

Adverse belief system:Negative core beliefs (“I’m bad/damaged/worthless”)

Somatic distress: Functional syndromes
Fibromyalgia, chronic pain
Non-epileptic seizures, headaches

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22
Q

Most important risk factors PTSD

A

Severity
Duration
Proximity

23
Q

Pre trauma risk factors

A
Childhood trauma
History of trauma
Low SES
Low education
Low IQ
Ethnic minority
Past psych issues
Past SUD
Family psych history
Low cortisol: Low cortisol: cortisol is what’s responsible for shutting off feedback loop of catecholamines, hpa axis is less active. high levels of catecholamines in the brain = the connection between amygdala and hippocampus gets blocked.
24
Q

During trauma risk factors

A
Nature of trauma:
Violent
Sexual
Death
Personal
Degrading
External locus control: external locus of control: when we face events do we feel like we have power over them or feel like things just happen to us. internal: i have power and agency over the world
Dissociation during trauma
25
Q

Post trauma risk factors

A

ASD (50% progress)
Little support or services
Shame/guilt/doubt
Ongoing stressors

26
Q

Adverse childhood experience mental health outcomes

A
Suicide (12 x more)
  Depression (5x)
  Addictions (5x)
  Alcohol use disorder (7x)
  Injecting drugs (10x)
  Linked to most psych illnesses:
 ALL anxiety disorders
 BAD
 Eating disorders
 ADHD & LD
27
Q

Adverse childhood experience physical health outcomes

A
Heart disease
COPD
Cancer
Liver disease
Sexually transmitted diseases
Obesity
Autoimmune diseases
Pelvic pain
Migraines, headaches
Unexplained symptoms
28
Q

Course - PTSD

A
Onset mid-late 20’s 
Onset can be delayed by years
Chronic, waxing & waning 
60% respond to treatment
40% have residual symptoms
 Suicide (6x attempts), violence
Associated with poor quality of life
29
Q

Poor Prognostic Factors

A
Delayed onset of sympt.
Delayed intervention
Poor premorbid function
Poor social support
Severe sympt.
Comorbidities
Veterans 
Elderly / children
30
Q

Comorbidity

A
60% of patients have > 3
Major depression in 50%
Anxiety disorders
Substance use disorders
Personality disorders
Somatization
General Medical Conditions: pain, TBI, sexual dysfcn, IBS
Children: ODD, ADHD, separation anxiety
31
Q

Non pharmacological treatment

A
Therapy is the treatment of choice!
 Combo  Therapy > Meds
 Combo not routinely recommended
 Individual & group are effective
 Positive literature for:
 VRE and I-CBT
 rTMS (L1)
32
Q

Pharmacology

A

60% response rate
Poorer response in veterans
May take up to 12 wks
Maintenance 1-2 yrs

33
Q

1st line

A

Sertraline
Fluoxetine
Venlafaxine
Paroxetine

34
Q

2nd line

A

Fluvoxamine
Mirtazapine
Phenelzine

35
Q

Adj 2nd line

A

Eszopiclone
Olanzapine
Risperidone

36
Q

Nightmares

A

Prazosin for nightmares

Nabilone off-label

37
Q

3 stages of stage based model

A

1)Safety & Stabilization
Building the foundation below your feet

2)Trauma Processing
Taking the sting out of the memories

3) Integration & Reconnection
Moving beyond

38
Q

Stage 1 , 3 types of safety,

A

Physical safety: Substance use, self-injury, eating disorders
Environmental safety: Stable living situation, non-abusive relationships
Emotional safety: Ability to calm the body, modulate intense emotions, set boundaries

39
Q

freeze gateway to

A

dissociation

40
Q

Why do we need to assure safety in phase 1

A

While launching a survival response, we cannot integrate new information
Our first priority is to help the nervous system find its way back to safety

41
Q

Contraindications to Trauma Therapy

A

Unstable housing
Recent psychiatric admission (within 3-6 months)
Active acute suicidality or homicidality
Severe unstable substance use, eating disorder, medical condition
Current ongoing abusive relationships

42
Q

Goal of stage 1

A

Help people rediscover the resources & resilience they already have inside through
Psychoeducation
Skills
No trauma details

43
Q

Stage 2 – Trauma Processing, goal

A

Exposure to the narrative & desensitization

Goal: Remember the trauma without getting dysregulated; integrate it into explicit memory

44
Q

Why avoid benzos

A

right after trauma can increase the risk of PTSD because block capacity to consolidate memories = interfering with natural process

45
Q

Evidence-Based Therapies

A

Eye-Movement Desensitization Reprocessing (L1): Processing with BLS: bls: bilateral stimulation: stay in the present, innately soothing, in dreams where in REM, recreating processing memories
Resourcing

Trauma-Focused Cognitive Behaviour Therapy (L1): In vivo exposure
Imaginal exposure
Cognitive restructuring

Narrative Exposure Therapy (L1): Lifeline
Exposure
Transcript

Prolonged Exposure
Cognitive Processing Therapy

46
Q

Therapies that aren’t recommended

A

Supportive, dynamic, hypnotherapy

47
Q

Stage 3 – Integration & Reconnection

A
Addressing grief, attachment wounds
Re-investing in community, purpose, meaning
Transition out of therapy
Relapse-prevention plan
Goal: Thriving
48
Q

Kids below age 6:

A

Repetitive play in which themes of trauma are expressed
Less flashbacks, more reenactments in play
Nightmares not specific to trauma – frightening dreams
Withdrawn
Irritability is expressed with tantrums

49
Q

Acute stress disorder criteria

A

Intrusion: Memories
Flashbacks
Nightmares
Distress with triggers

Avoidance:Internal (memories, thoughts, feelings)
External (people, places, situations)

Mood : Difficulty experiencing positive emotions

Arousal: Hypervigilance
Startle reflex
Irritability / aggression
Poor concentration
Poor sleep

Dissociation: Amnesia
Derealization / depersonalization

> 9 criteria
3 days – 1 month

50
Q

Differences between PTSD and GAD

A

Dissociation is not a specifier
No negative cognitions
Timeline

51
Q

Treatment ASD

A

Mass screening & debriefing is not recommended
Psychological First Aid Model
Ensure basic needs & maintain daily routine
Recruit social support
Psychoeducation about normal reactions to trauma
Monitor & identify vulnerable individuals
No pharmacological treatment is indicated
Early use of BZD can increase risk of PTSD
Some evidence for EMDR prior to 1 month

52
Q

Adjustment disorder

A

“the presence of emotional or behavioral symptoms in response to an identifiable stressor/s, which occurred within three months of the beginning of the stressor/s. In addition, one or both of the following criteria must exist:
Distress that’s out of proportion with the expected reactions to the stressor.
Symptoms must be clinically significant. They cause severe distress and impairment in functioning.
In addition, the following criteria must be present:
The distress and impairment are related to the stressor and not because of an intensification of existing mental health disorders.
The reaction isn’t part of normal bereavement.
When the stressor is removed or the individual has begun to adjust and cope, the symptoms subside within six months.

53
Q

Specifiers adjustment disorder

A

with depressed mood: low mood, tearfulness, hopelessness
with anxiety: nervousness, worry, jitteriness, separation anxiety
with mixed anxiety and depressed mood
with disturbance of conduct: violation of rights of others/ age appropriate societal norms, rules
with mixed disturbance of emotions and conduct
unspecified: physical complaints, withdrawal form relationships, impaired work, academic performance