Class 3-Pain Assessment: The 5th Vital Sign Flashcards
Pain is subjective…
-it is whatever the patient says it is, existing wherever the patient says it does
-grimacing, guarding, moaning, tachycardia, hypoventilation (hypoxia)–>alveoli can collapse
Pain assessment
-it’s not just a number!
-it’s the interpretation of the number and critically thinking about a series of other factors that may be influencing pain
-requires attention to response to pain relief methods
-requires attention to the side effects of medication administration
-requires attention to “if the patient doesn’t have relief” what else can be done
-need accurate pain assessment-better able to develop non-pharmacologic and/or pharmacologic strategies to obtain improved clinical results
**ask about if it’s new, where, intensity, radiates anywhere, limitations of ADL, duration, relief, and aggravating
Pain relief…it’s holistic in nature
-isn’t just about providing pain medication
-it is about looking at alternative methods alone or in conjunction with medication:
-music, relaxation, massage, biofeedback, acupuncture etc.
-tubes to decompress/relieve pressure
-anxiolytics
-improving breathing and oxygenation
-positioning
-heat and/or cold application (good for musculoskeletal)
Pain relief..it’s holistic in nature (cont)
-psychological support, chaplain, social work etc
-hospice support
-palliative care team
-conflict resolution
-family support
-assisting with rituals
Pain relief..it’s about understanding structure and function
-neuroanatomic pathway and nocioception
-nocioception is a term used to describe how noxious stimuli are perceived as pain
-pain originates from either the CNS or PNS or both
-where are nocioceptors located?
-how do they get stimulated?
-how does the pain stimulus travel to the CNS?
Structure and function
-nociceptors: specialized nerve endings designed to detect painful sensations
-transmit sensations to central nervous system by two primary sensory (afferent) fibers: A delta & C fibers
A delta fibers
-myelinated and larger in diameter, and they transmit pain signal rapidly to CNS: localized, short-term, and sharp sensations result from A delta fiber stimulation
C fibers
-unmyelinated and smaller, and transmit signal more slowly: sensations are diffuse and aching, and they persist after initial injury
Peripheral sensory A delta & C fibers
-enter spinal cord by posterior nerve roots within dorsal horn by tract of Lissauer
Nociceptors and the 4 concepts of…
- transduction, stimulus takes place in periphery
- transmission, pain moves from spinal cord to brain
- perception, conscious awareness of pain sensation
- modulation, inhibition of pain sensation (endogenous pain relievers)
Phase 1-transduction
-occurs in response to noxious stimuli
-release variety of chemical mediators
-substance p, histamine, prostaglandins, serotonin, and bradykinin
-neurotransmitters lead to pain propagation
- a long sensory afferent nerve fibers to spinal cord and terminate in dorsal horn
-second set of neurotransmitters carry pain signal-substance p, glutamate, and atp
Phase 2- transmission
-pain impulse moves from level of spinal cord to brain
-if pain is not stopped it moves via various ascending fibers within the spinothalamic tract to the thalamus
Phase 3-perception
-consciousness of awareness of pain signal
-cortical structures such as limbic system account for emotional response to pain
-only when “pain” has reached the cortical structures can it be perceived as pain
Phase 4- Modulation
-body has built-in mechanism to slow down and stop the process of a painful stimulus that inhibits and blocks pain
-descending pathways release third set of neurotransmitters to produce analgesic effect
-neurotransmitters include:
-serotonin; norepinephrine; neurotensin; aminobutyric acid (GABA); and our own endogenous opioids; beta endorphins; enkephalins, and dynorphins
Pain treatment approaches
-when we treat pain we take 4 basic approaches toward each of these 4 components within the neuroanatomic pathway:
1. we can modify the source of pain (moving away from source)
2. we can attempt to alter the central perception of pain
3. we can modulate the transmission of pain in the CNS (internal pain relievers)
4. we can block the transmission of pain to the CNS (nerve block)
What is neuropathic pain?
-abnormal processing of pain message
-doesn’t follow the predictable phases of nociceptive pain
-most difficult type of pain to assess and treat
-pain is perceived long after injury heals
-neurochemical level
-conditions may lead to development
-diabetes mellitus, herpes zoster (shingles), HIV/AIDS, sciatica, trigeminal neuralgia, phantom limb pain, and/or chemotherapy
What is the source of pain?
-visceral pain
-deep somatic
-cutaneous pain
-referred pain
Visceral pain
-originates from larger interior organs
-stems from direct injury to organ or from stretching of organ from tumor, ischemia, distention, or severe contraction
-presents with autonomic responses such as vomiting, nausea, pallor, and diaphoresis