Child Psychiatry, and learning disabilities Flashcards

1
Q

defining charactersitics of ADHD?

A

hyperactivity and restlessness
impulsivity
inattention-don’t pay attention, distracted
poor concentration

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2
Q

factors promoting resilience (protective factors) in the development of psychiatric illnesses in children?

A
temperament
coping strategies
problem solving
self-esteem
stability
secure relationships
friendships
achievement

all of these can be strengthened by psychosocial interventions.

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3
Q

how can problems at birth affect neurodevelopment of a child?

A

fetal hypoxia- reduced function of the frontal lobe

born prematurely- frontal lobe not fully developed.

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4
Q

importance of considering ongoing development in the presentation of psychiatric conditions in children?

A

presentation following childhood abuse may be significantly delayed due to ability to recognise this as abuse only occurring once cognitive development adequate.

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5
Q

how can parental mental health disorders affect the mental health of a child?

A

attachment issues
poor emotional response
reduced ability to parent the child-becomes insecure attachment

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6
Q

how can poverty have an effect on the development of mental illness?

A

constant stress
physical health deterioration e.g. inadequate nutrition, poor management of physical illnesses, chronic pain
lack of opportunities e.g. education, employment, social activities or sports that allow a sense of achievement and self-esteem to develop.

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7
Q

5 axes of classification in child psychiatry?

A
psychiatric disorder
specific developmental delay
global developmental delay
physical disorders
social factors
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8
Q

ADHD prevalence?

A

more common in boys than girls (3:1)
aprrox. 0.5% in all school-age children
onset in pre-school yrs

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9
Q

Methylphenidate (a stimulant) may be used in ADHD treament, what are some side-effects?

A
common or very common:
dyspepsia, diarrhoea, nausea
reduced weight gain, growth restriction
movement disorders, irritability, aggression, tics
alopecia
cough
tachycardia, arrhythmias
depression
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10
Q

monitoring required with methylphenidate treatment?

A

monitor for psychiatric disorders
pulse, BP, psychiatric symptoms, appetite, weight and height should be measured at start of therapy, after each dose adjustment and at least every 6mnths thereafter.

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11
Q

key points of ICD-10 criteria for ADHD (hyperkinetic disorder) diagnosis?

A

ADHD classified under ICD-10 as a Behavioural and emotional disorder with onset usually occurring in childhood and adolescence
core symptoms of hyperactivity, inattention and impulsivity
symptoms must have started before age of 7
symptoms present in at least 2 settings e.g. home and school
must be definite evidence of impaired function
symptoms not caused or related to another mental health disorder

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12
Q

common co-morbidities in children with ADHD?

A
depression
tic disorders
anixety
oppositional defiance disorder
substance abuse
pervasive developmental disorders e.g. autistic disorder and asperger's syndrome
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13
Q

aetiology of ADHD?

A

genetics: risk of ADHD in 1st degree relative of sufferer 5 times the risk of the general pop. condition linked to various genes mainly related to dopamine action e.g. receptors, transporters and the monoamine system.
brain structure and function abnormalities: smaller frontal lobe, cerebellum and striatum. abnormality in prefrontal cortex suggested by restlessness and difficult concentrating, and these areas rich in catecholamines.
psychological factors-early deprivation-emotional, nutritional, stimulatory
environmental-prenatal-maternal stress, substance abuse
postnatal-head injuries, another brain disease

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14
Q

prognosis in ADHD?

A

overactivity in adolescence tends to improve greatly, but in those where it doesn’t, there is high risk of conduct disorder, antisocial behaviour, juvenile delinquency.
continuation into adulthood more likely if initial symptoms more severe, FH, or co-morbid psychiatric disease.
patients more likely to attain poor qualifications, become unemployed, be involved in multiple RTAs and serve a prison sentence.
also future problems with intimate relationships, childcare and organising household tasks. and problems with substance misuse and emergence of personality disorders.

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15
Q

how is methylphenidate thought to work in the tment of ADHD?

A

centrally acting sympathomimetic, mild CNS stimulant
blocks reuptake of NA and dopamine into presynaptic neurone, increasing their release into the extraneuronal space.
stimulates the underactive frontal lobe inhibitory system in pts with ADHD.

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16
Q

what interventions can be put in place before a formal ADHD diagnosis is made but condition is suspected?

A

offer parents or carers a referral to a parent training/education programme
initial diagnosis and starting of drug treatment should be made in secondary care

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17
Q

tment of ADHD in pre-school children?

A

parents or carers should be referred to a parent training/education programmes, these can be group programmes, or offer individual programmes if difficulty attending group e.g. transport difficulties or language barriers
if ineffective, consider referral to tertiary services

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18
Q

tment of ADHD in school age children and young people and moderate impairment?

A

group-based parent training/education programmes
group psychological tment (CBT and/or social skills training) for the younger child, and consider individual psychological tment for older child
drug tment can be tried next if therapies fail or severe symptoms or impairment. drug tment=1st line if severe ADHD, but parents should also be offered training/education programme.

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19
Q

drugs considered for ADHD tment?

A

methylphenidate for ADHD without significant comorbidity
methylphenidate for ADHD with comorbid conduct disorder
methylphenidate or atomoxetine when tics, Tourette’s syndrome, anxiety disorder, stimulant misuse or risk of stimulant diversion are present
atomoxetine if methylphenidate has been tried and has been ineffective at the maximum tolerated dose, or the child or young person is intolerant to low or moderate doses of methylphenidate.
consider dexamfetamine if ADHD unresponsive to max tolerated dose of methylphenidate or atomoxetine.

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20
Q

side effects to be particularly cautious of in pts treated with atomoxetine (NA reuptake inhibitor) for ADHD?

A

suicidal thinking and self-harming behaviour
agitation
irritability
abdo pain, nausea, malaise, jaundice or darkening or urine suggesting liver damage.

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21
Q

1st line tment for adults with ADHD?

A

drug treatment, unless person would prefer a psychological approach
methylphenidate usually considered 1st, atomoxetine or dexamfetamine considered if unsuccessful tment with methylphenidate trialled for at least 6 wks, special caution with dexamfetamine prescribing to those likely to engage in substance misuse or diversion. atomoxetine may be considered 1st line in adults where risk of diversion.
if stable on tment but remains functional impairment, or unresponsive to tment, consider group CBT.

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22
Q

triad of symptoms in autism spectrum disorder?

A

impaired social interaction
delayed and abnormal language development
rigidity of mind-may be manifest as ritualistic behaviour and difficulties in coping with change in routine.

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23
Q

ICD-10 classification of childhood autism?

A

classified under pervasive developmental disorders: characterised by qualitative abnormalities in reciprocal social interactions and in patterns of communication, and by a restricted, stereotyped, repetitive repertoire of interests and activities. These are a pervasive feature of their functioning in all situations.

childhood autism: a type of pervasive developmental disorder that is defined by:
(a) the presence of abnormal or impaired development that is manifest before the age of 3, and
(b) the characteristic type of abnormal functioning in all the three areas of psychopathology: reciprocal social interaction, communication, and restricted, stereotyped, repetitive behaviour.
In addition to these specific diagnostic features, a range of other nonspecific problems are common, such as phobias, sleeping and eating disturbances, temper tantrums, and (self-directed) aggression.

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24
Q

what is ASD and what does it describe?

A

autism spectrum disorder
this is widely used to describe a group of pervasive developmental disorders: childhood autism, asperger’s syndrome, rett’s disorder, other childhood disintegrative disorder and atypical autism, recognising that the disorders may overlap to some extent and that within any category there is a wide variation in symptom severity.

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25
Q

what is atypical autism?

A

a type of pervasive developmental disorder.this subcategory should be used when there is abnormal and impaired development that is present only after age three years, and a lack of sufficient demonstrable abnormalities in one or two of the three areas of psychopathology required for the diagnosis of autism (namely, reciprocal social interactions, communication, and restricted, stereotyped, repetitive behaviour) in spite of characteristic abnormalities in the other area(s). Atypical autism arises most often in profoundly retarded individuals and in individuals with a severe specific developmental disorder of receptive language.

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26
Q

features of impaired social interaction which may be noted in children suggesting childhood autism?

A
reduced eye contact
babies that don't like being held
those that do not value the company of their parents over complete strangers
don't respond to affection
unusual facial expressions
lack of gestures
lack of empathy
few peer relationships
27
Q

common abnormalities of speech in childhood autism?

A
echolalia
odd prosody-unusual pitch/stress/rhythm/intonation
pronoun reversal (refer to themselves as he or she)
28
Q

what are stereotypes in autism?

A

repetitive movements e.g. head rolling, hand-flapping

29
Q

pervasive developmental disorder found only in females?

A

rett’s syndrome-apparently normal early development is followed by partial or complete loss of speech and of skills in locomotion and use of hands, together with deceleration in head growth, usually with an onset between seven and 24 months of age. Loss of purposive hand movements, hand-wringing stereotypies, and hyperventilation are characteristic. Social and play development are arrested but social interest tends to be maintained. Trunk ataxia and apraxia start to develop by age four years and choreoathetoid movements frequently follow. Severe mental retardation almost invariably results

30
Q

male to female ration of childhood autism?

A

4:1

31
Q

aetiology of autism?

A

genetic factors: 90% heritability shown in twin studies
higher rates of social, language and learning problems in relatives, suggesting autism may represent the severe end of a general predisposition to developmental difficulties.
organic brain disorder: 20% of autism individuals experience seizures during adolescence, suggests an organic brain disorder. but no neuropathological, brain imaging or neurochemical findings consistent.
cognitive abnormalities: symbolic thinking and language. often inability to judge what others are thinking and to use this to predict their behaviour (lack of theory of mind)-*also features of personality disorders, espec. borderline PD.

32
Q

typical presenting features of asperger’s syndrome?

A

developmentally appropriate speech and language
unusual use of language e.g. prosody
motor clumsiness
unusually deep interest in 1 part. topic
difficulty in making close friends
rigid behaviour and stereotypies
social awkwardness or an eccentric social style, often they fail to read the other perosn’s feelings, and don’t understand humour or irony
child often shows enhanced ability to rote learn information about their special interests, or perform at a high level in 1 part activity.

33
Q

common comorbidities with asperger’s?

A

mood disorders
anxiety disorders- these 2 seen in 65% of those with asperger’s by adulthood.
also tics, tourette’s syndroe, ADHD, OCD and bipolar.

34
Q

physcial health problems in rett’s syndrome?

A
epilepsy (80%)
constipation
poor growth
scoliosis
cardiac and motor problems
35
Q

how should the social and physical environment be adjusted for children with autism?

A

minimise -ve impact by:
providing visual supports e.g. words, pictures or symbols that are meaningful for the child or young person
make reasonable adjustments or adaptations to amount of personal space given
consider individual sensory sensitivites to lighting, noise levels and the colour or walls and furnishings.

adjust or adapt health and social care processes e.g. appointments at start or end of day to minimise waiting time, or provide single rooms for those who need a GA in hospital e.g. dental tment.

36
Q

psychosocial interventions for the core features of autism-social impairment, language difficulties and rigid thinking?

A

Consider a specific social-communication intervention for the core features of autism in children and young people that includes play-based strategies with parents, carers and teachers to increase joint attention, engagement and reciprocal communication in the child or young person. Strategies should:
be adjusted to the child or young person’s developmental level
aim to increase the parents’, carers’, teachers’ or peers’ understanding of, and sensitivity and responsiveness to, the child or young person’s patterns of communication and interaction
include techniques of therapist modelling and video-interaction feedback
include techniques to expand the child or young person’s communication, interactive play and social routines.
The intervention should be delivered by a trained professional. For pre‑school children consider parent, carer or teacher mediation. For school‑aged children consider peer mediation.

37
Q

general tment options for pervasive developmental disorders?

A

parental training courses
psychoeducation by a specialist in PDDs
finding an appropriate educational setting
treating psychiatric and physical comorbidities

38
Q

mainstay of tment in pervasive developmental disorders?

A

intensive, focused behavioural training programmes
NICE autism guidelines: for challenging behaviour, ensure co-morbid illnesses treated, and provide interventions aimed at changing the environment e.g. advice to families and carers, adjusting or adapting physical surroundings.
offer a psychosocial intervention if no coexisting mental health or behavioural problem, physical disorder or environmental problem has been identified as triggering or maintaining the behaviour that challenges.

39
Q

when does NICE recommend pharamacological interventions in autism treatment?

A

Consider antipsychotic medication[2] for managing behaviour that challenges in children and young people with autism when psychosocial or other interventions are insufficient or could not be delivered because of the severity of the behaviour. Antipsychotic medication should be initially prescribed and monitored by a paediatrician or psychiatrist. Stop medication if no indication of a clinically important response at 6 weeks.

40
Q

when might CBT be considered in autisitic children?

A

if coexisting anxiety and have the verbal and cognitive ability to engage.

41
Q

managing sleep disturbance in autism?

A

Develop a sleep plan (this will often be a specific sleep behavioural intervention) with the parents or carers to help address the identified sleep problems and to establish a regular night-time sleep pattern. Ask the parents or carers to record the child or young person’s sleep and wakefulness throughout the day and night over a 2‑week period. Use this information to modify the sleep plan if necessary and review the plan regularly until a regular sleep pattern is established.

42
Q

ICD-10 description of asperger syndrome?

A

A disorder of uncertain nosological validity, characterized by the same type of qualitative abnormalities of reciprocal social interaction that typify autism, together with a restricted, stereotyped, repetitive repertoire of interests and activities. It differs from autism primarily in the fact that there is no general delay or retardation in language or in cognitive development. This disorder is often associated with marked clumsiness. There is a strong tendency for the abnormalities to persist into adolescence and adult life. Psychotic episodes occasionally occur in early adult life.

43
Q

if manging sleep with a sleep plan in autism is ineffective, what pharmacological options might be considered?

A

melatonin-can bee useful for severe sleep-wake cycle disturbances

44
Q

5 principles of the MCA?

A

1: presumption of capacity- all presumed to have capacity until proven otherwise, musn’t assume someone with a mental illness or disability e.g. learning disability, can’t make a decision for themselves.
2: support individuals in making their decisions- every effort to help person make their own decision.
3: unwise decisions- making an unwise decision does not mean that a person lacks capacity.
4: best interests- act in best interests of person if assessed not to have capacity.
5: least restrictive option- when acting in pt’s best interests, should also act in a way that interferes less with person’s rights and freedoms of action.

45
Q

the 2 stage functional test to assess capacity?

A

Stage 1. Is there an impairment of, or disturbance in the functioning of a person’s mind or brain? If so,
Stage 2. Is the impairment or disturbance sufficient that the person lacks the capacity to make a particular decision?
all capacity assessments must be time and decision specific

46
Q

definition of a learning disability?

A

significantly impaired intelligence (IQ of 70 or below) and social functioning, with onset before 18 yrs of age.

47
Q

ICD-10 components for a MILD learning disability?

A

IQ 69-50
expressive language: delayed, but everyday speech usual
comprehension: reasonable
non verbal communication: good
self care: fully independent in washing, dressing, eating, normal continence
independent living: possible
academic work: difficulties but should learn to read, write and do simple maths
adult work: capable of work demanding practical rather than academic sills.
mobility: normal
social development: some immaturity but otherwise normal
associated deficits: as normal population

48
Q

% of population with an IQ score of 70 or below?

A

2%

49
Q

aspects of adaptive functioning in a learning disability?

A

self-direction- can they plan ahead and structure their day, and health and safety
self-care
language and communication
independent living skills and community use

50
Q

physical health problems in those with a learning disability?

A
sensory impairment
constipation
dental problems
epilepsy
GORD
infections-H pylori
mobility problems
obesity
swallowing problems
51
Q

prevalence of epilepsy in patients with a learning disability?

A

40%

52
Q

prevalence of depression, mood disorder and anxiety disorders in those with a learning disability?

A

depression=20-25%
mood disorder=6%
anxiety disorder=16%

53
Q

why might other illnesses be underdiagnosed in those with a learning disability?

A

diagnostic overshadowing- their symptoms are attributed to their learning disability
or difficulty of the pt to get their symptoms across e.g. if they have an otitis media, may bang their head against a wall rather than saying they are in pain.

54
Q

caution with drug prescribing in learning disability?

A

be more cautious with increasing dose as pt may be unable to report ADRs

55
Q

prevalence of autism in learning disability pts?

A

20-30%

56
Q

autism prevalence?

A

2-3%

57
Q

IQ for moderate learning disability under ICD-10?

A

35-49

58
Q

IQ for severe learning disability under ICD-10?

A

0-34

59
Q

relationship between autism and learning disability?

A

autism more likely the more severe the learning disability

60
Q

most common identifiable cause of learning disability?

A

Down syndrome

61
Q

most common mental and behavioural problems in children and young people?

A

conduct disorders, and associated antisocial behaviour

62
Q

what are conduct disorders?

A

disorders characterized by a repetitive and persistent pattern of dissocial, aggressive, or defiant conduct. Such behaviour should amount to major violations of age-appropriate social expectations; it should therefore be more severe than ordinary childish mischief or adolescent rebelliousness and should imply an enduring pattern of behaviour (six months or longer). Features can also be symptomatic of other psychiatric conditions, in which case the underlying diagnosis should be preferred.

Examples include excessive levels of fighting or bullying, cruelty to other people or animals, severe destructiveness to property, fire-setting, stealing, repeated lying, truancy from school and running away from home, unusually frequent and severe temper tantrums, and disobedience. Any one of these behaviours, if marked, is sufficient for the diagnosis, but isolated dissocial acts are not.

63
Q

psychiatric comorbidities in those with conduct disorder?

A

ADHD

50% go on to develop antisocial PD in later life